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Rhodopsin

Rhodopsin is a light-sensitive G protein-coupled receptor found in the retinal photoreceptor cells of the eye.
It plays a crucial role in the initial steps of vision, converting light energy into electrical signals that are processed by the brain.
Rhodopsin is composed of the protein opsin and the chromophore retinal, which undergoes a conformational change upon absorbing light.
This change activates the G protein transducin, initiating a signaling cascade that ultimately leads to the generation of a neural impulse.
Rhodopsin is essential for both scotopic (low-light) and photopic (daylight) vision, and its dysfunction can result in various vision disorders, such as retinits pigmentosa and Oguchi disease.
Reasearchers studying Rhodopsin can leverage PubCompare.ai, an AI-driven platform that streamlines Rhodopsin research by locating protocols from literature, preprints, and patents, and providing AI-driven comparisons to identify the most effective protocols and products.
This can enhance reproducibity and accuracy in Rhodopsin studies.

Most cited protocols related to «Rhodopsin»

Detailed materials and methods are provided in the SI Appendix. This includes information on field work, flow sorting, whole-genome amplification, amplicon library construction and sequencing, data processing, assembly and analysis, including gene predictions, as well as phylogenetic, crystallization and phylogenomic analyses. The final ChoanoV1 genome assembly was assembled from 13,802,665 quality-filtered reads, and viral contigs were differentiated from the cellular assembly by tetranucleotide frequency and GC content. The ChoanoV1 assembly consisted of 11 contigs with average coverage of 215 ± 157×. For eastern North Pacific Ocean gene expression analyses, reads from metatranscriptomes were mapped to ChoanoVirus genomes at >95% nucleotide identity with bbmap. For 18S V4 amplicon sequencing, we had on average 131,385 ± 121,027 amplicons well−1 (the lowest number being 1,037) clustered at 99% and classified via the Protistan Ribosomal Reference database. Rhodopsin functionality of viral VirRDTS was determined via heterologous expression in E. coli. The VirRDTS crystallization samples were produced by a cell-free system, and crystals were grown using the in meso approach. Accession numbers and DOI for alignment and tree files are available in Dataset S2.
Publication 2019
Cell-Free System Cells Crystallization DNA Library Escherichia coli Gene Expression Profiling Genes Genome Nucleotides Rhodopsin Ribosomes Trees Virus Physiological Phenomena

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Publication 2010
11-cis-Retinal Biological Assay Bos taurus Cells Chromatography, Affinity Dithionite Fatty Acids Fluorescence Glycolipids Homo sapiens Lipids Mannose Membrane Proteins Monoclonal Antibodies Phospholipids proteoliposomes Rhodopsin Rod Cell Outer Segment Rod Opsins SDS-PAGE Serum Albumin Triton X-100
Human rhodopsin and medium-wave opsin sequences were fused to YFP to create transgenes of 4xGrm6-SV40.RHO-http://YFP.WPRE.pA and 4xGrm6-SV40.MWC-http://YFP.WPRE.pA. The transgenes were packaged in either AAV2 (4YF), AAV2 (7m8), or AAV8 (BP2) capsids using the standard triple transfection method and prepared in PBS [35 (link)]. Titres were achieved by qPCR targeting the ITRs common to all vectors.
Publication 2021
Capsid Cloning Vectors Homo sapiens Rhodopsin Rod Opsins Simian virus 40 Transfection Transgenes
If the LRT indicates that a particular site ( ) is subject to episodic diversifying selection, it may be of interest to explore which branches at that site have undergone diversification. The empirical Bayes (EB) procedure originally used to identify individual sites subject to diversifying selection in random effects models [28] (link), can be readily adapted here. To compute the empirical posterior probability at branch that , we apply Bayes' theorem, using to denote the data at site and to denote all the maximum likelihood parameter estimates from the alternative MEME model fitted to site : To compute the two likelihood terms and , we use and , respectively, for the model assigned to branch in equation 2. The rest of the branches employ the matrices fitted under the alternative model of MEME. Having computed for each branch , we evaluate the empirical Bayes factor for the event of observing positive selection at each branch: When , sequence data increase the prior odds of observing selection at the branch. We do not recommend using this type of inference other than for the purposes of data exploration, even for large values of (e.g. 100). Intuitively, all the information contributing to the estimate of is derived from observing the evolution along a single branch at a single site (i.e. from a sample with size ). To quantify this supposition, we simulated sequence data using the vertebrate rhodopsin phylogeny and branch lengths, applied positive selection of varying strength to five branches in the tree selected a priori (see Text S1), and applied the EB procedure to infer the identity of selected branches.
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Publication 2012
Biological Evolution Rhodopsin Trees Vertebrates
At 6-8 weeks postinjection, the eyes were harvested and fixed in 4% paraformaldehyde for 1 hour at room temperature. Whole mounts were prepared by making a circular incision around the ora serrata, removing the cornea and gently manipulating the eye cup to remove the neural retina. For retinal cross-sections, whole mounts were embedded in agarose and sectioned transversely using a vibratome (Leica Microsystems) at medium speed, maximum vibration, and 100 μm thickness. Whole mounts and sections were mounted on slides using VECTASHIELD antifade mounting medium (Vector Laboratories) containing DAPI to stain cell nuclei and examined under confocal microscopy to confirm expression of the opsin-YFP fusion proteins. For staining of rhodopsin, mounted sections were permeabilised with 0.2% Triton-X-100 PBS for 20 minutes at room temperature then blocked with 10% donkey serum (D9663, Sigma UK) in 0.2% Triton-X-100 PBS for one hour at room temperature. Primary antibody solution was then applied for 3 hours at room temperature (1 : 200 rabbit anti-human rhodopsin, Abcam ab112576 in blocking buffer). Sections were rinsed with 0.05% Tween20 PBS four times for 10 minutes each then incubated with secondary antibody solution for 2 hours at room temperature (1 : 200 AlexaFluor donkey anti-rabbit 488 in 2.5% donkey serum, 0.2% Triton-X-100 PBS). Slides were rinsed four times in 0.05% Tween20 PBS then once with water before being DAPI mounted as described above.
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Publication 2021
Buffers Cardiac Arrest Cell Nucleus Cloning Vectors Cornea DAPI Equus asinus Homo sapiens Immunoglobulins Microscopy, Confocal Nervousness Ora Serrata paraform Proteins Rabbits Retina Rhodopsin Rod Opsins Sepharose Serum Stains Triton X-100 Tween 20 Vibration

Most recents protocols related to «Rhodopsin»

The AON-treated ROs were fixed in 2% paraformaldehyde (Thermo Fisher Scientific) and 5% sucrose (Thermo Fisher Scientific) for 15 min at 4°C, followed by a 30-min incubation in 7.5% sucrose, 30 min in 15% sucrose, and 2 h incubation in 30% sucrose. The organoids were transferred to a cryomold and embedded in 7.5% gelatin (porcine skin; Merck KGaA) and 10% sucrose. The sample blocks were then frozen at −80°C. Sections of 10 μm thickness were sliced on a Cryotome FSE (Thermo Fisher Scientific), rehydrated in PBS, and stained following the protocol described by Cowan et al.46 (link) ABCA4 was detected using the anti-ABCA4 3F4 clone (1:100; Abcam, Cambridge, UK), rhodopsin was stained using the anti-rhodopsin 4D2 clone (1:300, Invitrogen), mitochondria were detected with an anti-MTCO2 antibody (1:150; Abcam), and nuclei were stained with Hoechst 33,342 (1:1000; Thermo Fisher Scientific). Images were collected on an LSM 800 confocal microscope (Carl Zeiss, Oberkochen, Germany) using a 60× objective and analyzed with ZEN Blue edition (Carl Zeiss) using the maximum intensity projection.
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Publication 2023
Antibodies, Anti-Idiotypic Cell Nucleus Clone Cells Freezing Gelatins Microscopy, Confocal Mitochondria Organoids paraform Pigs Rhodopsin Skin Sucrose
Mouse eyes were enucleated in darkness under dim red light. Each eye was flash-frozen on dry ice immediately after enucleation. Rhodopsin was extracted with 20 mm HEPES, pH 7.4, containing 10 mm n-dodecyl-β-maltoside and 5 mm freshly neutralized NH2OH·HCl, as described previously (Palczewska et al., 2018 (link)). Briefly, the tissue was homogenized with 0.9 ml of buffer in a 2-ml Dounce tissue homogenizer (Kontes Glass Co) and shaken for 5 min at 4°C. The sample was then centrifuged at 17,200 × g for 5 min at 4°C. The supernatant was collected, the pellet was extracted a second time with 0.9 ml of buffer, and the combined supernatants were filtered through a 0.22-μm polyethersulfone membrane. Absorbance spectra were recorded using a Varian Cary 50-Scan UV-Vis spectrophotometer (Varian Australian Pty Ltd.); the sample was used as blank, then it was bleached for 5 min with a white-light, 875-Lumens bulb, and finally the difference absorbance spectrum was recorded immediately following a bleach. The concentration of rhodopsin was determined by the decrease in absorbance at 500 nm using the molar extinction coefficient ε500nm = 42,000 M−1 ·cm−1.
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Publication 2023
Buffers Darkness dodecyl maltoside Dry Ice Extinction, Psychological Freezing HEPES Light Mice, House Molar Plant Bulb polyether sulfone Radionuclide Imaging Rhodopsin Tissue, Membrane Tissues

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Publication 2023
Alexa594 alexa fluor 488 Antibodies Arrestin Clone Cells Cryoultramicrotomy DAPI Eye Goat Mice, House Microscopy, Confocal paraform Rabbits Retinal Cone Rhodopsin Serum Sucrose Triton X-100
Our study followed the NIH Guide for the Care and Use of Laboratory Animals and the ARVO Statement for the Use of Animals in Ophthalmic and Vision Research. The Institutional Animal Care and Use Committee (IACUC) at the University of Oklahoma Health Sciences Center approved all protocols. The floxed LDHA (Stock No: 030112), Chx10-EGFP/Cre (Stock No: 005105), transgenic glial high-affinity glutamate transporter (GLAST-CreER), Rax-CreER (Stock No: 025521), tdTomato reporter (Stock No: 007909), and RiboTag (Stock No: 011029) mice were purchased from the Jackson Laboratory (Bar Harbor, ME). The rhodopsin-Cre (i75-Cre) mice have been described earlier (47 (link)) and were provided by Dr. Ching-Kang Jason Chen (Baylor College of Medicine, Houston, TX). Tetracycline-inducible RPE-specific VMD2-Cre mice and cone opsin-Cre (human red/green pigment gene promoter) mice were provided by Dr. Yun Le (University of Oklahoma). All mice were screened for rd1 and rd8 mutations and were negative for these mutations. The eyes or retinas were harvested after CO2 asphyxiation. For metabolic experiments, mouse retinas were harvested under deep anesthesia, or retinas were removed after decapitation. These tissues were subjected to biochemistry or immunohistochemistry. To eliminate bias, mice of the same sex, age, and genetic strain were randomly assigned to each experimental group. Litters were mixed to prevent litter bias. Once mice were genotyped and provided with unique ear tag identifiers, cohorts were selected randomly by the principal investigator (Dr. Rajala), such that research personnel doing experiments were blinded and only knew the ear tag number.
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Publication 2023
Anesthesia Animals Animals, Laboratory Animals, Transgenic Asphyxia Cone Opsins Decapitation Eye Glutamate Transporter Homo sapiens Immunohistochemistry Institutional Animal Care and Use Committees LDH 5 Mice, Laboratory Mutation Neuroglia Patient Holding Stretchers Pharmaceutical Preparations Pigmentation Promoter, Genetic Reproduction Retina Rhodopsin Strains tdTomato Tetracycline Tissues Vision Vitelliform Macular Dystrophy
Immunohistochemistry and immunoblot analysis were performed as previously described (48 (link)). In the current study, blots were incubated with LDHA (1:1,000), LDHB (1:1,000), hexokinase 1 (1:1,000), hexokinase 2 (1:1,000), Aldolase C (1:1,000), cone arrestin (1:1,000), pPKM2 (1:1,000), PKM2 (1:1,000), PKM1 (1:1,000), Pde6β (1:1,000), rhodopsin (1:1,000), rod arrestin (1:1,000), M-opsin (1:1,000), PDH (1:1,000) and actin (1:1,000) antibodies (Table S5) overnight at 4° C. The blots were then washed and incubated with HRP-coupled anti-mouse or anti-rabbit secondary antibodies (as appropriate) for 60 min at room temperature. After washing, blots were developed with enhanced SuperSignal West Dura Extended Duration Substrate (Thermo Fisher Scientific, Waltham, MA) and visualized using a Kodak Imager with chemiluminescence capability.
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Publication 2023
Actins Aldolase C Anti-Antibodies Antibodies Arrestin Chemiluminescence Dura Mater Hexokinase Hexokinase II Immunoblotting Immunohistochemistry LDH 5 Mus Rabbits Retinal Cone Rhodopsin Rod Opsins

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DAPI is a fluorescent dye that binds strongly to adenine-thymine (A-T) rich regions in DNA. It is commonly used as a nuclear counterstain in fluorescence microscopy to visualize and locate cell nuclei.
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Rhodopsin is a photoreceptor protein found in the retinal cells of the eye. It is responsible for initiating the visual transduction process, which converts light energy into electrical signals that the brain can interpret. Rhodopsin is a member of the G protein-coupled receptor family and is essential for the detection of light in low-light conditions.
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Rhodopsin is a light-sensitive G-protein-coupled receptor found in the retinal photoreceptor cells of the eye. It plays a central role in the visual transduction process, converting light energy into electrical signals that are transmitted to the brain.
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Anti-rhodopsin is a laboratory reagent used for the detection and analysis of rhodopsin, a light-sensitive protein found in the retinal cells of the eye. It provides a specific and sensitive tool for researchers studying visual perception and related biological processes.
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DAPI is a fluorescent dye used in microscopy and flow cytometry to stain cell nuclei. It binds strongly to the minor groove of double-stranded DNA, emitting blue fluorescence when excited by ultraviolet light.

More about "Rhodopsin"

Rhodopsin, a light-sensitive G protein-coupled receptor, plays a crucial role in the initial stages of vision.
Located in the retinal photoreceptor cells, rhodopsin converts light energy into electrical signals that are processed by the brain.
This essential visual protein is composed of the opsin protein and the chromophore retinal, which undergoes a conformational change when exposed to light.
This light-induced activation of rhodopsin triggers a signaling cascade that ultimately leads to the generation of a neural impulse.
Rhodopsin is vital for both scotopic (low-light) and photopic (daylight) vision, and its dysfunction can result in various vision disorders, such as retinitis pigmentosa and Oguchi disease.
Researchers studying rhodopsin can leverage PubCompare.ai, an AI-driven platform that streamlines the research process.
PubCompare.ai helps locate protocols from literature, preprints, and patents, and provides AI-driven comparisons to identify the most effective protocols and products.
This can enhance reproducibility and accuracy in rhodopsin studies.
When exploring rhodopsin, researchers may also encounter related terms and techniques, such as DAPI (4',6-diamidino-2-phenylindole), Alexa Fluor 488, MAB5356, Ab5417, AB15282, Anti-rhodopsin, and AB5405.
These tools and reagents can be utilized to visualize, detect, and analyze rhodopsin in various experimental settings.
By utilizing the insights gained from PubCompare.ai and incorporating relevant terms and techniques, researchers can enhance their understanding and investigation of this crucial visual protein, ultimately advancing the field of vision research and addressing related health concerns.