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Locked nucleic acid

Locked nucleic acid (LNA) is a modified RNA/DNA analogue that exhibits increased thermal stability and binding affinity when paired with complementary DNA or RNA sequences.
LNA molecules contain a methylene bridge connecting the 2'-oxygen and 4'-carbon of the ribose ring, which locks the ribose in the N-type (C3'-endo) conformation.
This structural modification enhances the hybridization properties of LNA, making it a powerful tool for a variety of applications in molecular biology, genetics, and therapeutics, including antisense oligonucleotides, microRNA detection and inhibition, and diagnostic probe desgin.
Reserach on LNA continues to unlock new potentials for this versatile nucleic acid analog.

Most cited protocols related to «Locked nucleic acid»

In single-cell RNA-Seq, small amounts of sample loss during a number of steps can lead to significant decreases in transcript detection sensitivity. A decrease in assay sensitivity results in data that is only accurate and reproducible for highly expressed genes, limiting the scope and confidence of gene expression analyses. Further complications in assay sensitivity arise from an uneven distribution of sequencing reads along a transcript; usually, in SMARTer, there is a bias towards more reads at the 3′ end of the transcript. Even coverage along a transcript improves the accuracy of analytical tools used to quantify gene expression and transcript isoform abundance. A method published by Picelli et al (Nature Methods, 2013) modified the traditional SMARTer protocol to address this by improving transcript detection, coverage, accuracy, yield, and cost. Following the same strategy as SMARTer library construction, Smart-seq2 uses several alternative reagents to generate whole-transcriptome full-length cDNA libraries.
Avoiding small-volume, bead-based SPRI cleanups of each sample is an effective way of reducing loss and increasing assay sensitivity. Lysing single cells in a guanidine thiocyanate buffer necessitates SPRI cleanup due to the protein denaturing effects of the compound, which will affect downstream reactions, like reverse transcription. Multiple alternative lysis buffers exist that address this. The Ambion Single Cell Lysis buffer (Life technologies, #4458235), often used for single-cell RT-PCR, only requires the addition of a stop solution to inactivate its lytic activity before subsequent reactions. A hypotonic lysis buffer with small amounts of RNase-inhibitor and surfactant, as described in Smart-seq2, is the preferred buffer due to the lack of a need for a post-lysis cleanup or the addition of a stop solution prior to reverse transcription. However, the optimal lysis strategy will depend on the experimental system being analyzed.
Smart-seq2 takes additional steps to minimize sample loss during library construction. The reverse transcription is improved by the addition of betaine and additional magnesium chloride to the reaction mix and by the use of a template-switch oligonucleotide with one locked nucleic acid (LNA) riboguanosine base. These improvements assist in the hybridization between the template-switch oligonucleotide and the cDNA product, thereby increasing the probability of successfully introducing a second PCR adapter onto the cDNA product (see Figure 1). A second key improvement was made in the preamplification PCR step, which can be heavily biased against either long transcripts or those containing regions with high G/C content. Picelli et al found that the preamplification PCR is improved by using the KAPA HiFi HotStart ReadyMix, which dramatically improved coverage and sensitivity, particularly for GC-rich transcripts.
Publication 2014
Betaine Biological Assay Buffers cDNA Library Cells Crossbreeding DNA, Complementary Endoribonucleases Gene Expression Gene Expression Profiling Genes guanidine thiocyanate Hypersensitivity locked nucleic acid Magnesium Chloride Oligonucleotides Protein Isoforms Proteins Reverse Transcriptase Polymerase Chain Reaction Reverse Transcription Single-Cell RNA-Seq Surface-Active Agents Transcriptome
ASOs 1, 3 and 4 (sequence 5′-GCTCATACTCGTAGGCCA-3′, position 791–808) and 2 (sequence 5′-CTCATACTCGTAGGCC-3′, position 792–807) are complementary to Mus musculus TNFRSF1A-associated via death domain (TRADD) mRNA (Genbank accession no. NM_001033161). The ASO lead 1a is the murine homolog (a G to A base change at position 5) of the human TRADD lead reported previously (28 (link)). Control oligonucleotides 5 (5′-GCCCAATCTCGTTAGCGA-3′) were designed with six mismatches to 4, such that they contained ≥4 mismatches to all known mouse sequence. ASOs 6 and 7 (sequence TCTGGTACATGGAAGTCTGG, position 8232–8251) and 8 (sequence AAGTTGCCACCCACATTCAG, position 5586–5605) are complementary to Mus musculus apolipoprotein B (ApoB) mRNA (Genbank accession no. XM_137955.5). The sequences were identified by a screen of 5-10-5 MOE 20mer ASOs as described previously (29 (link)–31 (link)). ASOs 9, 10 and 11 (sequence 5′-CTGCTAGCCTCTGGATTTGA-3′, position 1931–1950) are complementary to M.musculus phosphatase and tensin homolog (PTEN), mRNA (Genbank accession no. NM_008960). ASO 9 (18 (link)) and control oligonucleotide 12 (19 (link)) have been described previously.
MOE phosphoramidites were prepared as described previously (7 ,32 ,33 (link)). LNA and 2′-deoxyribonucleoside phosphoramidites were purchased from commercial suppliers. Oligonucleotides were prepared similar to that described previously (34 (link)) on either an Amersham AKTA 10 or AKTA 100 oligonucleotide synthesizer. Modifications from the reported procedure include: a decrease in the detritylation time to ∼1 min, as this step was closely monitored by UV analysis for complete release of the trityl group; phosphoramidite concentration was 0.1 M; 4,5-dicyanoimidazole catalyst was used at 0.7 M in the coupling step; 3-picoline was used instead of pyridine for the sulfurization step, and the time decreased from 3 to 2 min. The oligonucleotides were then purified by ion-exchange chromatography on an AKTA Explorer and desalted by reverse phase HPLC to yield modified oligonucleotides in 30–40% isolated yield, based on the loading of the 3′-base onto the solid support. Oligonucleotides were characterized by ion-pair-HPLC-MS analysis (IP-HPLC-MS) with an Agilent 1100 MSD system. The purity of the oligonucleotides was ≥90% (Supplementary Table S1).
Publication 2006
Apolipoproteins B Death Domain Deoxyribonucleosides High-Performance Liquid Chromatographies Homo sapiens Ion-Exchange Chromatographies Mice, House Mus Muscle Tissue Oligonucleotides phosphoramidite Phosphoric Monoester Hydrolases Picoline PTEN protein, human pyridine RNA, Messenger Tensin TNFRSF1A protein, human

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Publication 2019
Adolescent Adult Amygdaloid Body Antisense Oligonucleotides Anxiety Ethanol KDM6B protein, human locked nucleic acid Males Nucleus, Central Amygdaloid Rats, Sprague-Dawley Rattus RNA, Small Interfering Saline Solution

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Publication 2014
1H NMR Anti-Antibodies Biotin Brain Cells DNA, Complementary Fibroblasts Fishes HEK293 Cells Homo sapiens Immunoassay Immunofluorescence neuro-oncological ventral antigen 2, human Nucleic Acid Probes Patients Poly A RNA, Messenger Skin Spectroscopy, Nuclear Magnetic Resonance Spectrum Analysis Streptavidin Tromethamine Western Blot
Zebrafish and their embryos were handled according to standard protocols23 and in accordance with University of Massachusetts Medical School IACUC guidelines. For laser-assisted microsurgery, embryos at 46 hpf were anesthetized and immobilized in 0.5% of low-melt agarose (Biorad). The connection between AA5 and AA6 and the ventral aorta was ablated using a Micropoint laser (Photonic Instrument, Inc) mounted on a Zeiss AX10 Imager M1. SU5416 (Calbiochem) was prepared and used as described previously11 (link). Control embryos were treated with 0.1% dimethyl sulfoxide (DMSO). To arrest heartbeat, embryos were treated with 15 mM of 2,3-butanedione 2-monoxime (BDM; Sigma-Aldrich) or with buffered Tricaine methanesulfonate (Sigma-Aldrich) at 0.66 mg/ml in egg water for the indicated times. Two-photon time-lapse imaging, confocal microscopy and microangiography was performed as previously13 (link), 24 (link), with additional modifications as noted in Supplementary Methods. Antisense riboprobes against dll4, vegfa, kdrl, fli1a, and cdh5 were generated and used for whole mount in situ hybridization as described elsewhere25 (link). A klf2a fragment was PCR amplified and cloned by Gateway recombination. The resulting clone was linearized with BglII and a DIG-labeled riboprobe was synthesized using T7 polymerase. Digoxigenin (DIG)-labeled locked nucleic acid (LNA) probes (Exiqon, Copenhagen) were used to detect mature miR-126 and let-7 using in situ hybridization or Northern analysis as described elsewhere18 (link). Morpholinos, mRNA and Tol2-based plasmids were prepared and injected as previously11 (link),21 (link). In cases of co-injection with Morpholinos, Tol2-plasmids and transposase, a DNA/transposase mRNA mixture was initially injected, followed by Morpholino. Plasmid construction details are provided in the full methods section. Morpholinos against vegfa, tnnt2 and gata1 have been described elsewhere15 (link), 26 (link), 25 (link); all other Morpholino and oligonucleotide sequences are provided in the full methods section.
Publication 2010
Aorta Cardiac Arrest CDH5 protein, human Clone Cells diacetylmonoxime Digoxigenin DNA, A-Form Embryo GATA1 protein, human In Situ Hybridization Institutional Animal Care and Use Committees locked nucleic acid methanesulfonate Microscopy, Confocal Microsurgery Morpholinos Nucleic Acid Probes Oligonucleotides Plasmids Pulse Rate Recombination, Genetic RNA, Messenger Sepharose SU 5416 Sulfoxide, Dimethyl Transposase tricaine Zebrafish

Most recents protocols related to «Locked nucleic acid»

Mice underwent PAB surgery to induce RVF. PAB gradient was confirmed by echocardiography. At the time of surgery, animals were randomly assigned to receive locked nucleic acid (LNA)‐anti‐miR‐34a (25 mg/kg) intraperitoneally (n=9) or scramble control (n=8). Clinical and echocardiographic assessment was performed weekly, and animals were euthanized at the onset of overt heart failure. Capillary assessment and miR and protein expression studies were performed as described above in a subset of animals based on sample availability.
Publication 2024
A total of 48 IDC samples consisting of 8 biological replicates from each of the biological groups were taken for both array analyses (Grade 2 and 3 (24 from each grade) consisting of 8 samples each from stages I, II III of every grade) and were used for normalizing with pooled 10 adjacent normal samples. TLDA (ver2.0), which contains 667 human miRNAs covering Sanger miRBase (ver10.0), was performed as per the manufacturer’s protocols. The experiments were repeated with LNA arrays (ver11.0) containing 1372 miRNAs from hmr-miRBase 14.0 + miRPlus from Exiqon, Denmark, following the manufacturer’s instruction manual. Rodent array (Applied Biosystems version v 3.0) consisting of 641 mouse and 373 rat unique miRNAs along with appropriate controls were carried out in 5 biological replicates of Brca2/p53 double knock out mammary models along with 6 wild type controls to find out the common miRNAs between human and rodent systems.
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Publication 2024

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Publication 2024

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Publication 2024
A direct sequencing method was applied for detecting EGFR mutation without routine tumor enrichment. Retrieved Formalin-fixed, paraffin embedded (FFPE) tumor samples were used for genomic DNA extraction by the QIAmp DNA FFPE Tissue Kit (Qiagen, Hilden, Germany). Polymerase chain reaction (PCR) amplification of EGFR exons 18 to 21, using intron-based primers was followed. Sequencing was performed in both the forward and reverse directions. Since September 2014, the peptide nucleic acid-locked nucleic acid (PNA-LNA) PCR clamp method has been applied in almost all cases. Genomic DNA of EGFR mutation hot-spots were amplified by PCR with a PNA clamp primer synthesized from a PNA with a wild-type sequence and detected by a fluorescent primer that incorporates locked nucleic acids. This method for preferential amplification of the mutant sequence can detect EGFR mutation in specimens containing 100 to 1000 excess copies of wild-type EGFR sequence [39 ].
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Publication 2024

Top products related to «Locked nucleic acid»

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Lipofectamine 2000 is a cationic lipid-based transfection reagent designed for efficient and reliable delivery of nucleic acids, such as plasmid DNA and small interfering RNA (siRNA), into a wide range of eukaryotic cell types. It facilitates the formation of complexes between the nucleic acid and the lipid components, which can then be introduced into cells to enable gene expression or gene silencing studies.
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Lipofectamine RNAiMAX is a transfection reagent designed for efficient delivery of small interfering RNA (siRNA) and short hairpin RNA (shRNA) into a wide range of cell types. It is a cationic lipid-based formulation that facilitates the uptake of these nucleic acids into the target cells.
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TRIzol reagent is a monophasic solution of phenol, guanidine isothiocyanate, and other proprietary components designed for the isolation of total RNA, DNA, and proteins from a variety of biological samples. The reagent maintains the integrity of the RNA while disrupting cells and dissolving cell components.
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LNA probes are synthetic nucleic acid analogues that can be used in various molecular biology applications. They have increased thermal stability and binding affinity compared to standard DNA or RNA probes, allowing for improved specificity and sensitivity in detection and quantification of target sequences.
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The Nikon A1Si Laser Scanning Confocal Microscope is a high-performance imaging system that utilizes laser technology to capture detailed, high-resolution images of samples. It is designed to provide researchers with a powerful tool for exploring the structures and dynamics of biological specimens at the cellular and subcellular levels.
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LNA GapmeRs are single-stranded, modified oligonucleotides designed for targeted knockdown of gene expression. They contain locked nucleic acid (LNA) modifications that enhance their stability and binding affinity.
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Lipofectamine 3000 is a transfection reagent used for the efficient delivery of nucleic acids, such as plasmid DNA, siRNA, and mRNA, into a variety of mammalian cell types. It facilitates the entry of these molecules into the cells, enabling their expression or silencing.
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The MirVana miRNA Isolation Kit is a product designed for the isolation and purification of microRNA (miRNA) from various biological samples, including cells, tissues, and body fluids. The kit utilizes a specialized protocol and reagents to selectively extract and concentrate miRNA from the sample, while removing other RNA species and cellular components.
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The Fluorescent In Situ Hybridization (FISH) Kit is a laboratory tool used for the detection and localization of specific DNA or RNA sequences within cells or tissue samples. The kit provides the necessary reagents and protocols to perform the FISH technique, which involves the hybridization of fluorescently labeled DNA or RNA probes to their complementary target sequences in the sample.

More about "Locked nucleic acid"

Locked Nucleic Acid (LNA) is a revolutionary nucleic acid analog that has transformed the fields of molecular biology, genetics, and therapeutics.
This modified RNA/DNA molecule exhibits enhanced thermal stability and binding affinity when paired with complementary DNA or RNA sequences.
The key to LNA's power lies in its structural modification - a methylene bridge that connects the 2'-oxygen and 4'-carbon of the ribose ring, locking the ribose in the N-type (C3'-endo) conformation.
This structural 'lock' enhances the hybridization properties of LNA, making it a versatile tool for a variety of applications.
One of the primary applications of LNA is in the design of antisense oligonucleotides, which can be used to selectively target and silence specific genes.
LNA-based antisense probes demonstrate improved target specificity and stability, making them a powerful tool for gene expression analysis and therapeutic development.
Additionally, LNA has proven valuable in the detection and inhibition of microRNAs, small non-coding RNAs that play crucial roles in gene regulation.
LNA-based microRNA detection and inhibition methods, such as those utilizing the Lipofectamine RNAiMAX transfection reagent, have become essential for understanding and manipulating these important regulatory molecules.
Beyond these applications, LNA has also found utility in the design of diagnostic probes, including those used in Fluorescent In Situ Hybridization (FISH) techniques.
These LNA-based probes, often used in conjunction with the Nikon A1Si Laser Scanning Confocal Microscope, allow for highly specific and sensitive detection of target sequences, enabling advanced genetic and genomic analyses.
Researchers continue to unlock the full potential of LNA, exploring new frontiers in fields like gene editing, where LNA-based 'GapmeRs' are used to selectively silence target genes.
The versatility of LNA is further enhanced by its compatibility with various transfection reagents, such as Lipofectamine 2000, Lipofectamine 3000, and Opti-MEM, which facilitate the efficient delivery of LNA-based molecules into cells.
To maximize the impact of your LNA research, tools like PubCompare.ai can help you identify the most effective LNA protocols and products from the ever-expanding body of literature, preprints, and patents.
By leveraging the power of AI-driven optimization, you can take your LNA-based investigations to new heights, unlocking novel discoveries and driving progress in areas ranging from gene therapy to diagnostic development.
Whether you're working with LNA antisense oligonucleotides, LNA-based microRNA detection and inhibition, or exploring the vast potential of this remarkable nucleic acid analog, the insights and resources available can help you push the boundaries of what's possible.
Embark on your LNA journey with confidence, and let the 'locked' power of this versatile molecule unlock new frontiers in your research.