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> Disorders > Disease or Syndrome > Endometritis

Endometritis

Endometritis is an inflammation of the endometrium, the lining of the uterus.
It can be caused by bacterial, viral, or fungal infections, and is often associated with conditions like pelvic inflammatory disease, postpartum complications, or intrauterine device (IUD) use.
Symptoms may include pelvic pain, abnormal bleeding, and fever.
Diagnosis typically involves pelvic examiniation and lab tests.
Treeatment usually consists of antibiotics, and in severe cases, hospitalization may be required.
Proper management is essential to prevent complications like infertility or chronic pelvic pain.

Most cited protocols related to «Endometritis»

1
Obstetric Outcomes Determination Protocol

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Publication 2013
Antibiotics Apgar Score Blood Transfusion Cellulitis Chorioamnionitis Clavicle Committee Members Deep Vein Thrombosis Endometritis Forceps Fracture, Bone Hemorrhage Hospitalization Hypoxic-Ischemic Encephalopathy Hysterectomy Infant Infant, Newborn Infection Laceration Mothers Obstetric Delivery Paralysis, Facial Perineum Placenta Accreta Placenta Previa Plexus, Brachial Postpartum Hemorrhage Pregnancy Pulmonary Embolism Shoulder Dystocia Skeleton Vacuum Vagina Venous Thromboembolism Ventilation-Perfusion Scan Woman Wounds X-Ray Computed Tomography
2
Explant Cytokine and PG Response to Oxytocin, LPS, and Polymyxin B
After 24 h culture, tissue explants were challenged with different concentrations of oxytocin (OT3, 3-300 nM, Bachem), LPS (0.03-1 μg/ml, Sigma, E. coli serotype 055.B5) and polymyxin B (2 μg/ml, Sigma) individually or in combination as indicated. Once confluence had been reached, stromal and epithelial cells were challenged with different concentrations of arachadonic acid (AA3, 10-300 μM, Sigma), OT (100 nM), LPS (0.1 −3 μg/ml), heat-killed E. coli (102 −105 CFU/ml, isolated from a case of clinical bovine endometritis associated with pyrexia (14 (link))), polymyxin B (2 μg/ml), 17-β estradiol (3 pg/ml, Sigma) or progesterone (5 ng/ml, Sigma) individually or in combination and for the period of time indicated. Culture supernatants were harvested and frozen prior to cytokine and PG determination.
Herath S., Fischer D.P., Werling D., Williams E.J., Lilly S.T., Dobson H., Bryant C.E, & Sheldon I.M. (2005). Expression and Function of Toll-like Receptor 4 in bovine endometrium. Endocrinology, 147(1), 562-570.
Publication 2005
Acids Bos taurus Cytokine Endometritis Epithelial Cells Escherichia coli Estradiol Fever Freezing Oxytocin Polymyxin B Progesterone Tissues
3
Estrus Synchronization Protocols for High-Producing Dairy Cows
All procedures were approved by the Ethics Committee on Animal Experimentation of the University of Lleida (license numbers CEEA.09–01/12 and CEEA.09–01/13).
During the weekly reproductive visit, open cows with more of 50 days in milk and with no reproductive disorders such as ovarian cysts and endometritis detected by ultrasound were randomly assigned to one of the following groups: 2PGG, 2PGGe, 2PGe and PGe (Fig. 1

Treatment protocols used to synchronize estrus for fixed-time AI (FTAI) in high-producing dairy cows. All cows (n=232) were fitted with a progesterone releasing intravaginal device (PRID-DELTA, containing 1.55 g of progesterone; CEVA Salud Animal, Barcelona, Spain) for 5 days (PRID-5 days).

). Cows in the 2PGG group were treated with a progesterone-releasing intravaginal device (PRID) (PRID-DELTA, containing 1.55 g of progesterone; CEVA Salud Animal, Barcelona, Spain) plus GnRH (100 μg i.m.; Cystoreline, CEVA Santé Animale, Libourne, France) at PRID insertion. The PRID was left for 5 days, and these animals were also given PGF2α (25 mg dinoprost i.m.; Enzaprost, CEVA Santé Animale, Libourne, France) at PRID removal. Twenty-four hours later, the cows received a second PGF2α dose, and they were inseminated and received a second GnRH dose 36 hours after receiving the second PGF2α dose. The remaining groups were treated with the same P4-based protocol but with the following differences: cows in the 2PGGe group received 500 IU of eCG i.m. (Syncostim, CEVA Santé Animale, Libourne, France) at PRID removal; cows in the 2PGe received eCG at PRID removal and no GnRH was given at PRID insertion; and cows in the PGe group received eCG at PRID removal
and no GnRH at PRID insertion nor the second dose of PGF2α. In this latter group, cows were fixed-time inseminated 60 h after PRID removal. Only healthy cows with no signs of mastitis, lameness or digestive disorders were included in the study. Two experiments were performed to investigate effects of treatments on follicular/luteal dynamics (Experiment I) and fertility (Experiment II).
Cows diagnosed as not pregnant received no further treatment related to the study. This meant that a cow receiving a five-day P4-based protocol was included only once in both experiments. All gynecological exams and pregnancy diagnoses were performed by the second author.
GARCIA-ISPIERTO I, & LÓPEZ-GATIUS F. (2014). Effects of different five-day progesterone-based fixed-time AI protocols on follicular/luteal dynamics and fertility in dairy cows. The Journal of Reproduction and Development, 60(6), 426-432.
Publication 2014
Animals Corpus Luteum Dairy Cow Device Removal Diagnosis Digestive System Disorders Dinoprost Endometritis Estrus Ethics Committees Fertility Gonadorelin Gynecological Examination Mastitis Medical Devices Milk, Cow's Ovarian Cysts Pregnancy Progesterone Reproduction Treatment Protocols Ultrasonography
4
Ovarian Follicular LPS Measurement
Follicular fluid was aspirated using a sterile needle guided by transrectal ultrasonography, from postpartum (40–60 days) dairy cows (n=58) in which the uterine disease had been evaluated as part of an independent study (Moussavi et al. 2007 (link)). Briefly, the uterine disease cytology evaluates the level of inflammation from 0 (normal; no inflammation), 1 (subclinical; mild inflammation), 2 (subclinical; moderate inflammation) to 3 (clinical endometritis). Samples were stored in endotoxin-free glass or polystyrene tubes (Lonza, Basel, Switzerland) at −20 °C until analysed. Concentrations of bacterial LPS were measured in samples using the QCL-1000 Chromogenic Limulus Amebocyte Lysate (LAL) Endpoint Assay Kit (Lonza) following the manufacturer's guidelines. Samples were thawed, diluted in endotoxin-free 0.05 M Tris and tested for non-specific LAL inhibition by comparing samples spiked with a known concentration of LPS with unspiked samples. Samples with evidence of LAL inhibition were heated in a water bath at 75 °C for 30 min using temperatures and times validated in our laboratory to remove non-specific inhibitors of the LAL reaction (Williams et al. 2007 (link)). Samples were then mixed with the LAL substrate reagent and assayed in duplicate in 96-well endotoxin-free microplates (Corning, Lowell, MA, USA) alongside standard curve LPS concentrations of 0.01, 0.25, 0.5, 1.0 and 5 endotoxin units/ml (10 eu=1 ng LPS) in serum. Serial dilutions were made in 50 mM Tris until concentrations were measurable in the linear part of the standard curve. Internal recovery as determined using positively spiked serum samples was >80% and the intra- and inter-assay coefficients of variation were 4.0 and 7.2% respectively and the limit of detection was 0.01 ng/ml. To establish further that LPS crosses the ovarian follicle basement membrane, bovine ovaries were obtained from a slaughterhouse and eight medium (4–8 mm diameter) and large follicles (>8 mm diameter) dissected and maintained in 6 ml Dulbecco's modified Eagle's medium (DMEM)/F12 (Sigma) containing 10 μg/ml LPS (Sigma: E. coli serotype 055:B5) for 18 h at 37 °C. Ovaries were washed six times in water and endotoxin-free Tris, and follicular fluid aspirated using a sterile needle (25 G) and syringe. Concentrations of LPS were measured as previously described.
Herath S., Williams E.J., Lilly S.T., Gilbert R.O., Dobson H., Bryant C.E, & Sheldon I.M. (2007). Ovarian follicular cells have innate immune capabilities that modulate their endocrine function. Reproduction (Cambridge, England), 134(5), 683-693.
Publication 2007
azo rubin S Bacteria Bath Biological Assay Cattle Cytological Techniques Dairy Cow Endometritis endotoxin binding proteins Endotoxins Escherichia coli Follicular Fluid Hair Follicle Inflammation inhibitors Limulus Membrane, Basement Needles Ovarian Follicle Ovary Polystyrenes Psychological Inhibition Serum Sterility, Reproductive Syringes Technique, Dilution Tromethamine Ultrasonography Uterine Diseases
5
Identifying Prolonged Postpartum Stay After Cesarean
The study cohort was identified using a dataset (the Cesarean registry) sourced from a previous multicenter study by the National Institute of Child Health and Human Development Maternal-Fetal Medicine Units (MFMU) Network.9 (link) Briefly, between 1999 and 2002, data were collected in women who underwent primary cesarean delivery, repeat cesarean delivery, or trial of labor after cesarean, and who delivered infants ≥ 20 weeks or ≥ 500 g at 19 academic centers. The final 2 years of the study included only women who underwent repeat cesarean delivery or vaginal birth after cesarean. Data were collected through detailed chart review at delivery, and information regarding perioperative morbidity was collected from discharge summaries. Patients and hospitals were deidentified by the MFMU.
For the current study, we included only women from the Cesarean registry who had undergone a primary or repeat cesarean delivery. Based on a definition described by Kuklina et al,2 (link) we defined a prolonged postpartum LOS as a postpartum hospitalization (number of hospital days between cesarean delivery to hospital discharge) with a postpartum LOS ≥ 90th centile. In the Cesarean registry dataset, all dates were expressed as day numbers. Maternal demographic, antepartum, perioperative, and neonatal variables were compared between women with and without prolonged LOS. Maternal characteristics included: age, race/ethnicity, body mass index (BMI) at delivery, diabetes, chronic hypertension, and number of prior cesarean deliveries. Obstetric variables included: gestational age at delivery, pregnancy-associated hypertension, labor, or induction before the cesarean delivery, and chorioamnionitis. Perioperative variables included: type of uterine incision, mode of anesthesia, intraoperative red blood cell transfusion, hysterectomy, and postpartum complications, such as endometritis and wound complications. We also compared birth weights of neonates of women with versus without prolonged LOS.
We performed bivariate analyses to compare maternal, obstetric, and neonatal characteristics between women with and without prolonged LOS after cesarean delivery. Categorical variables were compared using the chi-square test; bivariate analyses did not account for missing data. Based on bivariate analyses, unadjusted odds ratios (ORs) and accompanying 95% confidence intervals (CIs) were calculated. Candidate variables that were associated with prolonged LOS on bivariate analyses (p ≤ 0.1) were included as covariates in an unconditional multivariable logistic regression model. The final model was determined using a traditional backward elimination, with all variables initially included and then selectively removed if not significant (p < 0.05). To determine the presence of collinearity between independent variables, variance inflation factor (VIF) testing was performed. Collinearity was determined to be insignificant as VIF scores ranged from 1.01 to 1.49 with a mean VIF score of 1.18. Population attributable fractions (PAFs) were used to calculate the proportional reduction in risk of prolonged LOS that would occur by eliminating the exposure of interest from the population while the distribution of other risk factors remained unchanged. PAFs were calculated for selected risk factors that were considered modifiable by using adjusted ORs (aORs) from the final multivariate model.10 (link) We calculated the area under the receiver-operating characteristic curve (AUROC) using standard methodology to assess the predictive performance of the final model.
Based on data for the date of hospital admission and discharge, we performed a secondary analysis to assess risk factors for prolonged total length of hospital stay, defined as the interval from admission to discharge. For the total period of hospital stay we defined a prolonged delivery hospitalization as a total hospital LOS ≥ 90th centile. We did not count the day of admission in the calculation for the total hospital LOS.
Data analyses were performed using SAS 9.2 (SAS Inc., Cary, NC) and STATA version 12 (Statacorp, College Station, TX). As the Cesarean registry contains deidentified data, our study was deemed institutional review board exempt by the Stanford Institutional Review Board.
Blumenfeld Y.J., El-Sayed Y.Y., Lyell D.J., Nelson L.M, & Butwick A.J. (2015). Risk Factors for Prolonged Postpartum Length of Stay Following Cesarean Delivery. American journal of perinatology, 32(9), 825-832.
Publication 2015
Anesthesia Cesarean Section Chorioamnionitis Diabetes Mellitus Endometritis Ethics Committees, Research Ethnicity factor A Gestational Age High Blood Pressures Hospitalization Hypertension, Gestational Hysterectomy Hysterotomy Index, Body Mass Infant Infant, Newborn Mothers Obstetric Delivery Obstetric Labor Patient Discharge Patients Red Blood Cell Transfusion Trial of Labor Vaginal Birth after Cesarean Woman Wounds

Most recents protocols related to «Endometritis»

1
Invasive Group A Streptococcus Infections
Invasive disease was defined by the isolation of GAS from a normally sterile site (blood, bone, pleural fluid, synovial fluid, peritoneal fluid, or cerebrospinal fluid (CSF)).
Probable invasive disease was defined as an unwell patient with GAS isolated from a non-sterile site (deep skin or retropharyngeal aspiration) who required one or more of the following: hospitalization for intravenous antibiotics, surgery, or admission to the intensive care unit (ICU).
Clinical syndromes of piGAS and iGAS disease were categorized as isolated bacteremia, pneumonia/empyema, skin and soft tissue infection (SSTI) with or without bacteremia, NF, STSS, septic arthritis, osteitis, bursitis, abdominal/peritoneal infection, meningitis, pharyngeal abscess with and without bacteremia, and endometritis (pregnancy or not pregnancy related).
Clinical syndromes are presented in Supplementary Information 1. In case of concurrent diagnosis in the same patient, the most severe diagnosis was retained.
IGAS infections were considered healthcare related if they occurred at least 48 h after the time of admission or if the patient underwent surgery within the 7 days preceding the onset of iGAS. IGAS or piGAS infections were considered associated to chickenpox if it occurred within a maximum time span of 7 days from the onset of the varicella infection. The pediatric population was limited to children between 0 and 16 years. An adult was defined as a patient at least 17 years old.
The 2010 case definition [15 ] based on two major criteria (hypotension and the involvement of at least two organs/systems) was applied for the diagnosis of STSS.
Zangarini L., Martiny D., Miendje Deyi V.Y., Hites M., Maillart E., Hainaut M., Delforge M., Botteaux A., Matheeussen V., Goossens H., Hallin M., Smeesters P, & Dauby N. (2023). Incidence and clinical and microbiological features of invasive and probable invasive streptococcal group A infections in children and adults in the Brussels-Capital Region, 2005–2020. European Journal of Clinical Microbiology & Infectious Diseases, 42(5), 555-567.
Publication 2023
Abscess Adult Antibiotics Arthritis, Infectious Bacteremia Blood Bones Bursitis Cerebrospinal Fluid Chickenpox Child Diagnosis Empyema, Pleural Endometritis Hospitalization Infection Intraabdominal Infections Meningitis Operative Surgical Procedures Osteitis Patients Peritoneal Fluid Peritoneum Pharynx Pleura Pregnancy Skin Soft Tissue Infection Sterility, Reproductive Syndrome Synovial Fluid Tetradecyl Sulfate, Sodium
2
Letrozole Exposure Effects on Rat Offspring
Pregnant rats were housed in standard plastic cages individually. They were randomly divided into 4 experimental groups (n = 3/each) to orally receive letrozole at 4 doses (0.25, 0.75, 1.00, and 1.25 mg/kg BW) and a control group. Considering the reports on the embryotoxic effects of letrozole on pregnant rats and rabbits (16), a pilot study was performed to identify the doses above the physiological levels that were not lethal to the mother or the offspring. Accordingly, letrozole administration between 1.5 and 3.0 mg/kg BW caused fetal mortality, adsorption or death in early life, and uterine infection.
Letrozole (L6545, Sigma-Aldrich, St. Louis, USA) was dissolved in 1% carboxymethylcellulose (C5013, Sigma-Aldrich, St. Louis, USA) and was orally administered on days 16-18 of gestation (13). Testicular testosterone surge on 16-18 GDs is necessary for the brain masculinization and normal development of male rats (9, 10). The control rats were administrated with 1% carboxymethylcellulose on same days. The offspring's number, birth weight, and sex were recorded. The offspring remained with their mothers until weaning. At postnatal day (PND) 21, they were weaned, sexed, and weighed, and their AGD was measured. The anogenital distance index (AGDI) was calculated as AGD/BW × 100 (17). Male offspring (n = 4 per group) were kept in separate standard cages and were weighed weekly until the end of the study.
Shaaban Z., Derakhshanfar A., Reza Jafarzadeh Shirazi M., Javad Zamiri M., Moayedi J., Vahedi M, & Valizadeh A. (2023). The effects of letrozole-induced maternal hyperandrogenism on sexual behaviors, testicular histology, and serum biochemical traits in male offspring rats: An experimental study. International Journal of Reproductive Biomedicine, 21(1), 71-82.
Publication 2023
Adsorption Birth Weight Brain Carboxymethylcellulose Endometritis Letrozole Males Mothers Oryctolagus cuniculus physiology Pregnancy Rattus norvegicus Testis Testosterone Virilism
3
Fetal Membrane Collection and Analysis
Fetal membranes were collected (between 14 March 2021 to 30 January 2022) from the Obstetrics Department, Estaing University Hospital, Clermont-Ferrand, France. All patients presented no underlying chronic or specific diseases of pregnancy (preeclampsia, pregnancy-induced hypertension, gestational diabetes, symptoms of preterm birth), no acute or chronic treatments (for hypertension, for example) as confirmed by (i) normal clinical following-up and (ii) macroscopic and microscopic placenta analyses and histological examinations that excluded chorioamnionitis. Furthermore, study patients (mother and baby) displayed no signs of sepsis ((French criteria: chorioamnionitis, urinary infections, isolated fever, elevated CRP, endometritis, early neonatal onset sepsis) during and after parturition; infection participation was rejected. Moreover, French clinical recommendations opted for the absence of amniotic fluid analyses for asymptomatic patients.
Samples of first-trimester (T1) membranes were obtained by aspiration after voluntary termination of pregnancy (10 to 12 weeks of gestation (WG)). Second-trimester (T2) membranes were collected after premature termination of pregnancy for severe fetal abnormalities (mainly severe cardiac malformations) without clinical repercussion for the mother’s health (22–25 WG). Macroscopic and microscopic analyzes were still normal. Third-trimester (T3) membranes were collected from pregnancies after spontaneous delivery (31–36 WG). Moreover, term FMs were collected from the women after spontaneous labor followed by vaginal deliveries (herein referred to as TIL, 37–41 WG) and after scheduled cesarean deliveries without labor due to scarred uterus and non-cephalic fetal position (herein referred to as TNL, 37–41 WG). After the collection of FM samples, they were immediately stored at −80 °C before quantitative RT-PCR, Western blot assay, or freezing sections. All membranes were separated into the amnion and choriodecidua (except for those collected during the first trimester), as previously described [45 (link)].
Coste K., Bruet S., Chollat-Namy C., Filhol O., Cochet C., Gallot D., Marceau G., Blanchon L., Sapin V, & Belville C. (2023). Characterization of RAGE and CK2 Expressions in Human Fetal Membranes. International Journal of Molecular Sciences, 24(4), 4074.
Publication 2023
Amnion Amniotic Fluid Birth Cesarean Section Chorioamnionitis Congenital Abnormality Congenital Heart Defects Endometritis Fetal Membranes Fetus Fever Gestational Diabetes High Blood Pressures Hypertension, Gestational Induced Abortions Infant Infection Microscopy Mothers Obstetric Delivery Obstetric Labor Patients Physical Examination Placenta Pre-Eclampsia Pregnancy Premature Birth Reverse Transcriptase Polymerase Chain Reaction Sepses, Neonatal Septicemia Specimen Collection Tissue, Membrane Urinary Tract Infection Uterus Vagina Western Blot Woman
4
Equine Uterus Collection Protocol
Mare uteri were collected within 5 min after slaughtering at a local abattoir (Rawicz, Poland), according to the European legislation (EFSA, AHAW/04–027), and under the veterinary official inspection. Only healthy mares were considered for this study. A blood jugular sample was also collected at the abattoir into ethylenediaminetetraacetic acid (EDTA) tubes. Estrous cycle determination was based on ovarian feature observation, and confirmed by progesterone (P4) plasma determinations. The follicular phase (FP) criteria were: plasma P4 concentration < 1 ng/mL and a follicle > 35 mm diameter. The conditions to be considered a mid-luteal phase (MLP) sample were: plasma P4 concentration > 6 ng/mL, follicles between 15 and 20 mm diameter and the presence of a well-developed corps luteum. The uteri that showed the presence of endometritis (increased uterine mucus, altered surface endometrium color and the occurrence of bacteria/neutrophils) were discarded [9 (link),64 (link)]. Sample limitation only allowed the use of category IIA and IIB of Kenney and Doig [67 ], that corresponds to mild to moderate alterations of endometrosis [67 ]. After collection, uteri from the follicular phase (FP; n = 7) and the mid-luteal phase (MLP; n = 6) were transported on ice to the laboratory, and immersed in cold Dulbecco’s modified Eagle’s medium (DMEM) F-12 Ham medium (D/F medium; 1:1 (v/v); D-2960; Sigma-Aldrich, St Louis, MO, USA), supplemented with 100 IU/mL penicillin (P3032; Sigma-Aldrich, St Louis, MO, USA), 100 µg/mL streptomycin (S9137; Sigma-Aldrich, St Louis, MO, USA), and 2 g/mL amphotericin (A2942; Sigma-Aldrich, Burlington, MA, USA). Approximately 80 equids are slaughtered per day, for meat consumption. To perform the several in vitro studies carried out in our laboratory, approximately 10 FP and 10 MLP uteri were collected in each visit to the slaughterhouse. An average of 4 uteri were discarded in each assay due to the presence of signs of endometritis described above, or secondary contamination.
Amaral A., Cebola N., Szóstek-Mioduchowska A., Rebordão M.R., Kordowitzki P., Skarzynski D, & Ferreira-Dias G. (2023). Inhibition of Myeloperoxidase Pro-Fibrotic Effect by Noscapine in Equine Endometrium. International Journal of Molecular Sciences, 24(4), 3593.
Publication 2023
Amphotericin Bacteria Biological Assay BLOOD Cold Temperature Eagle Edetic Acid Endometritis Endometrium Estrous Cycle Europeans Hair Follicle Luteal Phase Meat Menstrual Cycle, Proliferative Phase Mucus Neutrophil Ovarian Follicle Ovary Penicillins Plasma Progesterone Streptomycin Uterus
5
Equine Endometritis Diagnosis and Pathogen Identification
We conducted clinical examination of mares that had been infertile for a long time on the farm and laboratory examination of mares suspected of having endometritis (fluid or swelling in the uterus upon examination) after rectal and ultrasound examinations. Briefly, the mare was bound, and the mare's vulva was disinfected using benzalkonium bromide (0.1% concentration). After disinfection, a double-layer sampling tube was inserted into the mare's vagina, and the inner tube was slowly inserted into the mare's uterus when the outer tube reached the cervix. Saline was injected into the uterus, and then the uterine flush was collected, followed by bacteria isolation and identification (see Supplementary material for results). The purified pathogens (E. coli, S. equi subsp zooepidemicus, and S. aureus) were inoculated into nutrient broth liquid medium containing sheep serum (Servicebio, Wuhan, China) and incubated for 12 h at 37°C on a shaker (23 (link)).
Ding X., Cui X., Shi J., Cheng X., Yao D., Gao Y, & Zhang Y. (2023). Construction of a model of endometritis in domestic rabbits using equine-derived pathogens and evaluation of therapeutic effect of sensitive drugs. Frontiers in Veterinary Science, 10, 1064522.
Publication 2023
Bacteria Benzalkonium Bromides Cervix Uteri Disinfection Endometritis Escherichia coli Flushing isolation Nutrients Pathogenicity Physical Examination Rectum Saline Solution Serum Sheep Staphylococcus aureus Ultrasonography Uterus Vagina Vulva

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Fetal Bovine Serum (FBS) is a cell culture supplement derived from the blood of bovine fetuses. FBS provides a source of proteins, growth factors, and other components that support the growth and maintenance of various cell types in in vitro cell culture applications.
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More about "Endometritis"

Endometritis, also known as uterine inflammation, is a condition characterized by the inflammation of the endometrium, the innermost lining of the uterus.
This condition can be caused by a variety of factors, including bacterial, viral, or fungal infections, and is often associated with conditions like pelvic inflammatory disease (PID), postpartum complications, or intrauterine device (IUD) use.
Symptoms of endometritis may include pelvic pain, abnormal vaginal bleeding, and fever.
Diagnosis typically involves a pelvic examination and laboratory tests, such as FBS (Fasting Blood Sugar) and Diff-Quik staining, which can help identify the underlying cause of the inflammation.
Treatment for endometritis usually consists of antibiotics, and in severe cases, hospitalization may be required.
In some cases, DMSO (Dimethyl Sulfoxide) or TransIT reagents may be used to enhance the delivery of antibiotics or other medications.
Additionally, GraphPad Prism 7 may be used to analyze and visualize data related to endometritis research.
It is important to note that proper management of endometritis is essential to prevent complications, such as infertility or chronic pelvic pain.
In some cases, Amphotericin B or Mueller Hinton agar plates may be used to treat fungal infections associated with endometritis.
If you're researching endometritis, you may find the Ab97046 antibody or the PrimeScript RT reagent kit useful for your investigations.
By leveraging the power of AI-driven protocol optimization, you can streamline your research process and make informed decisions to ensure accurate and reproducible outcomes.