Log In Sign up
The largest database of trusted experimental protocols
> Disorders > Pathologic Function > Acidosis

Acidosis

Acidosis is a medical condition characterized by an abnormal increase in the acidity of the body's fluids, often resulting from an imbalance in the production or excretion of acids.
This condition can have various causes, including respiratory disorders, metabolic disturbances, and dietary factors.
Acidosis can lead to a range of symptoms, such as fatigue, confusion, and even life-threatening complications if left untreated.
Accurate diagnosis and effective management of acidosis are crucial for maintaining overall health and well-being.
Resaerchers can utilize PubCompare.ai's AI-driven platform to enhance the reproducibility and accuracy of their acidosis research, locating the best protocols from literature, pre-prints, and patents using intelligent comparisons to identify the most effective approaches and optimize their studies for improved results.

Most cited protocols related to «Acidosis»

1
Maternal Age and Adverse Perinatal Outcomes
Observational studies using case control and cohort designs that reported one or more of the pre-specified primary and/or secondary outcomes for maternal age <35 (control) and ≥35 years (AMA) populations were included. Primary outcomes were stillbirth (according to individual study gestational age cut off) and fetal growth restriction (FGR) defined as birthweight below 5th centile adjusted for gestational age [17 (link)] Secondary outcomes were neonatal death (NND), small for gestation age (SGA; defined as a birthweight below 10th centile adjusted for gestational age or related definitions specified by authors), neonatal intensive care unit (NICU) admissions and neonatal acidosis (umbilical artery pH <7.0–7.2), preeclampsia (blood pressure ≥140/90 with significant proteinuria or as classified by authors where definition was not provided), placental abruption (classified by authors), preterm birth (PTB) <37 weeks gestation and gestational diabetes mellitus (GDM). Where authors stated different definitions of outcomes, data were re-classified in line with definitions stated (e.g. if authors defined FGR as <10th Centile this was re-classified as SGA in these analyses). Where definitions of classifications were not stated, authors’ classifications were accepted. Where possible, extracted data was sub-divided by parity (primiparous and multiparous mothers). Where reported, data regarding the frequency of maternal co-morbidities (obesity, hypertension and diabetes) and use of ART were extracted.
Duplicate studies were removed and the papers were excluded if they: were case reports, were restricted to multiple pregnancies or did not separate data from multiple pregnancies from singletons, primarily reported the success rates of assisted reproductive technologies or pre-existing medical conditions as a primary outcome, focused on chromosomal abnormalities or substance abuse, relevant data could not be extracted or were review papers. Reports on rising Cesarean section rates with AMA were not included as a systematic review was recently conducted on this topic [12 (link)] The initial search was conducted by one investigator (SL) and validated by a secondary conductor (HD) to ensure accuracy of search and application of exclusion criteria.
Lean S.C., Derricott H., Jones R.L, & Heazell A.E. (2017). Advanced maternal age and adverse pregnancy outcomes: A systematic review and meta-analysis. PLoS ONE, 12(10), e0186287.
Full text: Click here
Publication 2017
Abruptio Placentae Acidosis Assisted Reproductive Technologies Birth Weight Blood Pressure Cesarean Section Chromosome Aberrations Diabetes Mellitus Fetal Growth Retardation Gestational Age Gestational Diabetes High Blood Pressures Infant, Newborn Obesity Population Group Pre-Eclampsia Pregnancy Premature Birth Substance Abuse Umbilical Arteries
2
Hypothermia for Neonatal Encephalopathy
Participants were recruited between July 2000 and May 2003. Infants were eligible if they had either moderate or severe encephalopathy within 6 hours after birth, with severe acidosis or resuscitation at birth after an acute perinatal event. Infants were randomly assigned either to undergo whole-body hypothermia at 33.5°C for 72 hours or to usual care. All surviving children were evaluated at 6 to 7 years of age; the families of those who did not return for follow-up were contacted by telephone to obtain information about the primary outcome. The protocol, available at NEJM.org, was approved by the institutional review board at each site, and written informed consent was obtained from the parents of all trial participants.
Shankaran S., Pappas A., McDonald S.A., Vohr B.R., Hintz S.R., Yolton K., Gustafson K.E., Leach T.M., Green C., Bara R., Huitema C.M., Ehrenkranz R.A., Tyson J.E., Das A., Hammond J., Peralta-Carcelen M., Evans P.W., Heyne R.J., Wilson-Costello D.E., Vaucher Y.E., Bauer C.R., Dusick A.M., Adams-Chapman I., Goldstein R.F., Guillet R., Papile L.A, & Higgins R.D. (2012). Childhood Outcomes after Hypothermia for Neonatal Encephalopathy. The New England journal of medicine, 366(22), 2085-2092.
Publication 2012
Acidosis Child Childbirth Encephalopathies Ethics Committees, Research Human Body Infant Parent Resuscitation
3
Umbilical Cord Lactate for Neonatal Outcomes
This was a prospective cohort study of consecutive deliveries at Washington University in St. Louis Medical Center from 2009 to 2014. The study was approved by the Washington University School of Medicine Human Research Protection Office.
Inclusion criteria were singleton pregnancies at term, vertex presentation, and labor prior to delivery. Multiple gestations and pregnancies with fetal anomalies were excluded. Term pregnancy was defined as gestational age ≥37 weeks. Pregnancies were dated by a woman’s last menstrual period and confirmed with first or second trimester ultrasound [14 (link)]. Demographic information, medical and surgical history, obstetric and gynecologic history, prenatal history and detailed labor and delivery information were abstracted from patients’ charts by trained research nurses.
Umbilical cord blood was collected immediately after infant delivery, prior to knowledge of neonatal outcomes as previously described [7 (link)]. Briefly, a policy of universal umbilical cord gas and lactate measurement was instituted prior to the study. Both arterial and venous blood samples are obtained from a clamped segment of cord immediately after delivery. Umbilical blood lactate is measured from whole blood using an automated benchtop analyzer (DXC-800 Automated Chemistry Analyser, Beckman Coulter). As previously reported the coefficient of variation of the lactate assay in our laboratory is 2.9% [7 (link)]. Umbilical arterial blood samples were validated to be arterial or venous by ensuring that pH was at least 0.02 lower in the artery than the vein [15 (link)].
The outcome measures were arterial lactic acidemia and a composite neonatal outcome. Umbilical arterial lactic acidemia was defined as arterial lactate >3.9 mmol/L based on a prior study in our institution [7 (link)]. The composite neonatal outcome was made up of neonatal death and any of a number of neonatal morbidities including endotracheal intubation, mechanical ventilation, meconium aspiration syndrome, hypoxic-ischemic encephalopathy, and therapeutic hypothermia as previously reported [7 (link)]. Components of the composite were diagnosed by the attending neonatologist, without knowledge of the umbilical cord lactate levels. Meconium aspiration syndrome was diagnosed based on the presence of meconium stained amniotic fluid, neonatal respiratory distress, and characteristic radiographic abnormalities [16 (link)]. Hypoxic-ischemic encephalopathy was diagnosed based on the National Institute of Child Health and Human Development (NICHD) criteria [17 (link)]. Administration of therapeutic hypothermia was indicated for neonates meeting the following criteria per institutional protocol: ≥36 weeks gestational age at birth, moderate to severe hypoxic-ischemic encephalopathy with or without seizures, and any one of 10-minute Apgar score <5, prolonged resuscitation at birth, severe acidosis (pH < 7.1) on cord or neonate blood gas analysis within 60 minutes of birth, or base deficit (>12 mmol/L) on cord or neonate blood gas analysis within 60 minutes of birth [18 (link)]. Only one morbidity was counted per patient for the composite.
Baseline characteristics were calculated for the entire cohort and compared between women with and without the composite neonatal outcome. Continuous variables were compared using the Student’s t test while categorical variables were compared using the chi-square or Fisher’s exact test as appropriate. Normality of distribution of the continuous variables was evaluated using the Kolmogorov-Smirnov test.
We used linear regression analysis to examine the relationship between umbilical cord arterial and venous lactate. We constructed receiver-operating characteristics (ROC) curves to assess the predictive ability of umbilical venous lactate for arterial lactic acidemia, and to compare the predictive ability of venous and arterial lactate for the composite neonatal outcome. The ‘optimal’ cut-point of venous lactate for predicting arterial lactic acidemia was estimated based on the maximal Youden index [19 (link)]. The cut-point corresponding to the maximal Youden index maximizes the correct classification of subjects [20 (link)]. The area under the ROC curves for venous and arterial lactate were compared using the method described by Delong et al. [21 (link)]. We calculated and compared predictive characteristics (sensitivity, specificity, positive and negative predictive values, and positive and negative likelihod ratios) of arterial and venous lactate for neonatal morbidity based on the ‘optimal’ cut-points. Sensitivities and specificities were compared using an extension of the McNemar test [22 (link)]. To explore the effects of using different cut-points of venous lactate on prediction of the composite neonatal outcome, we calculate predictive characteristics for different venous lactate cut-points. These included the 95th and 99th percentile thresholds from the current cohort as well as venous lactate thresholds estimated from arterial lactate thresholds in the literature [2 (link), 23 (link)].
We did not estimate the sample size a priori; all consecutive patients meeting the inclusion criteria during the study period were included. All statistical tests were 2-tailed and P<0.05 was considered significant. Analyses were conducted using STATA software package, version 12, Special Edition (College Station, TX).
Tuuli M.G., Stout M.J., Macones G.A, & Cahill A.G. (2016). Umbilical Cord Venous Lactate for Predicting Arterial Lactic Acidemia and Neonatal Morbidity at Term. Obstetrics and gynecology, 127(4), 674-680.
Publication 2016
Acidosis Amniotic Fluid Apgar Score Arteries Biological Assay Birth BLOOD Blood Gas Analysis Cone-Rod Dystrophy 2 Congenital Abnormality Fetal Anomalies Gestational Age Homo sapiens Hypersensitivity Hypothermia, Induced Hypoxic-Ischemic Encephalopathy Infant Infant, Newborn Intubation, Intratracheal Lactates Mechanical Ventilation Meconium Meconium Aspiration Syndrome Menstruation Neonatologists Nurses Obstetric Delivery Obstetric Labor Operative Surgical Procedures Patients Pharmaceutical Preparations Pregnancy Prognosis Respiratory Rate Resuscitation Seizures Student Ultrasonography Umbilical Cord Umbilical Cord Blood Umbilical Vein Umbilicus Veins Woman X-Rays, Diagnostic
4
Neonatal Encephalopathy Screening Protocol
The infant will be assessed sequentially by criteria A, B and C listed below:
A. Infants ≥ 36 completed weeks gestation admitted to the NICU with at least one of the following:
• Apgar score of ≤ 5 at 10 minutes after birth
• Continued need for resuscitation, including endotracheal or mask ventilation, at 10 minutes after birth
• Acidosis within 60 minutes of birth (defined as any occurrence of umbilical cord, arterial or capillary pH < 7.00)
• Base Deficit ≥ 16 mmol/L in umbilical cord or any blood sample (arterial, venous or capillary) within 60 minutes of birth
Infants that meet criteria A will be assessed for whether they meet the neurological abnormality entry criteria (B) by trained personnel:
B. Moderate to severe encephalopathy, consisting of altered state of consciousness (lethargy, stupor or coma) AND at least one of the following:
• hypotonia
• abnormal reflexes including oculomotor or pupillary abnormalities
• absent or weak suck
• clinical seizures
Infants that meet criteria A & B will be assessed by aEEG (read by trained personnel):
C. At least 30 minutes duration of aEEG recording that shows abnormal background aEEG activity or seizures. There must be one of the following:
• normal background with some seizure activity
• moderately abnormal activity
• suppressed activity
• continuous seizure activity
Azzopardi D., Brocklehurst P., Edwards D., Halliday H., Levene M., Thoresen M, & Whitelaw A. (2008). The TOBY Study. Whole body hypothermia for the treatment of perinatal asphyxial encephalopathy: A randomised controlled trial. BMC Pediatrics, 8, 17.
Full text: Click here
Publication 2008
Acidosis Apgar Score Arteries BLOOD Capillaries Childbirth Comatose Consciousness Debility Encephalopathies Infant Lethargy Nervous System Abnormality Pregnancy Pupil Malformations Resuscitation Seizures Stupor Umbilical Cord Veins
5
Maximal Cardiorespiratory Fitness Assessment
Participants performed all the experimental trials on the same cycle ergometer (Ergoselect 200, Ergoline, Germany). Immediately following a standardized warm-up of 10 min at 50 W, all participants performed a ramp protocol with increments of 25 W·min-1 until exhaustion. During GXT participants were monitored by standard 12 lead ECG (Quark T12, Cosmed, Italy), Oxygen consumption ( V˙O2 ) and carbon dioxide production ( V˙CO2 ) were recorded using breath-by-breath indirect calorimetry (Quark B2, Cosmed, Italy). Familiarization GXT fulfilled three objectives: a) discard cardiac defects or diseases in any of the participants, b) to minimize the bias of progressive learning on test reliability and c) to discard any participant V˙O2max lower than 55.0 ml·kg-1·min-1.
Both experimental maximal GXT with 15 min warm-up divided in three 5-min steady state stages at 45%, 55%, and 65% of the peak power output (PPO), being the three intensities below the second ventilatory threshold (VT2). After 10 minutes of passive recovery in which each participant ingested 200–250 ml of water to ensure adequate hydration status, a sample of capillary blood from the finger was obtained to assess CBL (Lactate Pro, Arkray, Japan). Following, participants performed the GXT according to a modification of the protocol described by [4 (link)]. Initial workload was set at 50 W, with increments of 25 W·min-1, requiring at all times a cadence between 80–85 rpm.
Heart rate was continuously monitored (RS400, Polar, Finland), gas exchange was recorded breath by breath using indirect calorimetry and capillary blood samples were obtained and analyzed every 2 min (i.e., each 50 W increments). Each participant indicated their rate of perceived exertion every two minutes using the Borg Scale 6–20, where 6 is defined as an effort "very very light" and one 20 "Maximum, strenuous" effort [18 ]. Capillary blood lactate analyzer and indirect calorimetry devices were calibrated before each test. In order to avoid the local acidosis that could impair the attainment of maximum cardiorespiratory performance, and according to the subjects’ maximal PPO in the GXTPRE (i.e., 375-425W), starting at 50 W, the workload was progressively increased by 25 W·min-1 that ensure that testing duration was not excessively long (i.e., 13.5–15.0 min). This protocol also allowed collecting between 7 to 9 capillary blood samples before exhaustion to be used in the CBL data analysis.
Pallarés J.G., Morán-Navarro R., Ortega J.F., Fernández-Elías V.E, & Mora-Rodriguez R. (2016). Validity and Reliability of Ventilatory and Blood Lactate Thresholds in Well-Trained Cyclists. PLoS ONE, 11(9), e0163389.
Full text: Click here
Publication 2016
Acidosis BLOOD Calorimetry, Indirect Capillaries Carbon dioxide Electrocardiography, 12-Lead Ergoline Fingers Heart Lactates Light Medical Devices Oxygen Consumption Rate, Heart

Most recents protocols related to «Acidosis»

1
Respiratory Responses to Acidosis
Data were analyzed with GraphPad (GraphPad Prism9, San Diego, California, United-States). Normality of data distribution for fR, VT and VE, for cells immunolabelled c-FOS, 5-HT, c-FOS/5-HT, PHOX2B, c-FOS/PHOX2B, for concentrations of 5-HT, 5-HTP and 5-HIAA, and for 5-HT/5-HTP and 5-HIAA/5-HT ratios were assessed using the d’Agostino and Pearson omnibus normality test. Depending on whether the distribution was normal or not, data were expressed as mean ± standard deviation or median and interquartile range [Q1; Q3] and parametric or non-parametric tests were performed: within a group of preparation, in paired conditions, paired t-test, Wilcoxon signed rank test, one-way analysis of variance, or Friedman test followed by Dunn’s or Benjamini, Krieger and Yekutieli’s multiple comparison test; between two groups, in unpaired conditions, unpaired t test, Mann & Whitney test and two-way analysis of variance followed by Benjamini, Krieger and Yekutieli’s multiple comparison test in unpaired conditions; between more than two groups, one way analysis of variance or Friedman test followed by Benjamini, Krieger and Yekutieli’s multiple comparison test. Percentages of Phox2b mutant mice acidosis-responder or acidosis-non-responder with or without etonogestrel were compared by Fisher’s exact test. Differences were considered to significant if p < 0.05.
Casciato A., Bianchi L., Reverdy M., Joubert F., Delucenay-Clarke R., Parrot S., Ramanantsoa N., Sizun E., Matrot B., Straus C., Similowski T., Cayetanot F, & Bodineau L. (2023). Serotonin and the ventilatory effects of etonogestrel, a gonane progestin, in a murine model of congenital central hypoventilation syndrome. Frontiers in Endocrinology, 14, 1077798.
Full text: Click here
Publication 2023
5-Hydroxytryptophan Acidosis c-fos Genes Cells etonogestrel Hydroxyindoleacetic Acid Mice, House
2
Premature Neonatal Intensive Care Protocol
Extremely premature-born animals were cared for in humidified, heated incubators (Drägerwerk) as outlined in Fig. 1A. In brief, PC-CMV was continuously adapted to maintain paO2 between 7.3 and 9.3 kPa (55-70 mmHg) and paCO2 between 6.0 and 7.3 kPa (45-55 mmHg). Ketamine and midazolam (Akorn) were provided as required. Heparinized, sodium- and pH-adapted maintenance fluids (normal saline, half-normal saline, or sodium acetate with 1 U/mL unfractionated heparin—all from Hospira) and an individually prepared, amino acid-rich, electrolyte, and glucose-adapted total parenteral nutrition were infused alongside intravenous lipids. Maintenance fluid rates were adapted to maintain mean arterial pressures> >25 mmHg. If hypotensive, a maximum of 2 boluses (10 mL/kg) normal saline was given and escalated to inotropic therapy if required with dopamine ± dobutamine (both from Hospira) up to doses of 20 µg/kg/min, respectively. If hypotension persisted, hydrocortisone at 1 mg/kg every 12 hours was initiated and tapered as soon as possible. Acidosis was buffered with sodium bicarbonate (Hospira) at 1 mmol/kg. Animals were transfused with stored placental or maternal packed blood cells at 10 mL/kg if hematocrits persisted below 30%. To prevent sepsis, preemptive ampicillin (50 mg/kg), gentamicin (2.5 mg/kg) and vancomycin (15 mg/kg, all from Hospira) were given (Fig. 1A). Whole-body anteroposterior X-ray films were taken every 24 hours to assess the lung, heart, intestines and evaluate the position of the endotracheal tube and the intravascular catheters. Echocardiographic studies were performed daily by a pediatric cardiologist; an open ductus arteriosus was neither treated medically, nor surgically.
Möbius M.A., Seidner S.R., McCurnin D.C., Menschner L., Fürböter-Behnert I., Schönfeld J., Marzahn J., Freund D., Münch N., Hering S., Mustafa S.B., Anzueto D.G., Winter L.A., Blanco C.L., Hanes M.A., Rüdiger M, & Thébaud B. (2023). Prophylactic Administration of Mesenchymal Stromal Cells Does Not Prevent Arrested Lung Development in Extremely Premature-Born Non-Human Primates. Stem Cells Translational Medicine, 12(2), 97-111.
Publication 2023
Acetate Acidosis Amino Acids Ampicillin Animals Bicarbonate, Sodium BLOOD Cardiologists Catheters Childbirth Dobutamine Dopamine Ductus Arteriosus Echocardiography Electrolytes Gentamicin Glucose Heart Heparin Sodium Human Body Hydrocortisone Intestines Ketamine Lipids Lung Midazolam Normal Saline Operative Surgical Procedures Parenteral Nutrition, Total Placenta Premature Birth Septicemia Sodium Stem Cells Therapeutics Vancomycin Volumes, Packed Erythrocyte X-Ray Film
3
Acidosis-Induced Pain Sensitivity in Females
Considering the gender differences in pain caused by acidosis in our previous reports, only female rats were used to study in the current experiment (57 (link)). Rats were first habituated for 30 min in a Plexiglas chamber during the nociceptive behavioral experiment. The rats received two intraplantar injections, each with a volume of 50 μl. For the first time, rats in five different groups (n = 10/group) injected with either 50 μl of 10 μM AMG 9810 + vehicle, a 50 μl of cocktail containing10 μM AMG 9810 + different doses (0.1, 1, and 10 ng) of LY354740, or 50 μl of cocktail containing AMG 9810 + 50 ng LY341495 + 10 ng LY354740. After 10 min, another experimenter injected 50 μl of acetic acid solution (1% v/v, pH value was adjusted to 6.0 with NaOH and external solution containing 10 mM MES) into the ipsilateral hind paws and tested the nociceptive behavior. The assessor of the behavioral measures was blinded to the prior treatment conditions. Nociceptive behavior (i.e., number of flinching, shaking, and licking) was counted within 5 min after injection. In one group, acetic acid was injected into one hind paw and 10 ng LY354740 was injected into the contralateral hind paw. Nociceptive behaviors were expressed as the number of flinches and counted immediately within 5 min after injection (29 (link), 35 (link)).
Li Q., Liu T.T., Qiao W.L., Hao J.W., Qin Q.R., Wei S., Li X.M., Qiu C.Y, & Hu W.P. (2023). Group II metabotropic glutamate receptor activation attenuates acid-sensing ion channel currents in rat primary sensory neurons. The Journal of Biological Chemistry, 299(3), 102953.
Full text: Click here
Publication 2023
(E)-3-(4-t-butylphenyl)-N-(2,3-dihydrobenzo(b)(1,4)dioxin-6-yl)acrylamide Acetic Acid Acidosis Females LY 341495 LY 354740 Pain Plexiglas Rattus norvegicus
4
Comprehensive ICU Respiratory Monitoring
For all included patients, age, gender, body weight, comorbidity, smoking history, and HIV-related data were collected. We also recorded the clinical and laboratory data just before the enrollment and clinical-related data during the treatment and follow-up. The severity of illness was evaluated by the Acute Physiological and Chronic Health Status Score II (APACHE II) within 24 h of ICU admission and the sequential organ failure assessment (SOFA) at inclusion [20 (link),21 (link)]. The vital signs (heart rate, systolic blood pressure (SBP), diastolic blood pressure (DBP), respiratory rate), the arterial blood gases (arterial pH, PaCO2, PaO2/FiO2), and calculated the HACOR score and ROX index at baseline, 2 and 24 h after randomization, were recorded. The HACOR score is based on clinical and laboratory parameters, including heart rate, acidosis (assessed by pH), consciousness (evaluated by Glasgow Coma Scale), oxygenation (assessed by PaO2/FiO2 ratio), and respiratory rate. The ROX index was calculated by dividing SpO2/FiO2 to respiratory rate. A visual analogue scale (VAS, 0–10 scoring), on which 0 indicated absence and 10 indicated the highest possible levels, was used to express feelings of dyspnea and device discomfort and completed at the same three points. The cause of ARF was determined by the consensus of three senior intensivists (J.L., L.P., and C.L.). Initial settings and ventilation characteristics were collected at 2 h after randomization during NIV or HFNC treatment. Airway care interventions were defined as times to correct unplanned device displacement or assist in the removal or fixation of the device. Adverse events were nasofacial skin breakdown, intolerance, nasal prongs or masks broken, and air leaks. Intolerance was defined as patient-reported complaints due to airway dryness, claustrophobia, gastric distension, ocular irritation, headache, breathlessness, or device-related discomforts like airflow, pressure, temperature, and noise. The complaints of intolerance for patients who need immediate intubation were not included in this adverse event. All data were collected on a dedicated case report form.
Hao J., Liu J., Pu L., Li C., Zhang M., Tan J., Wang H., Yin N., Sun Y., Liu Y., Guo H, & Li A. (2023). High-Flow Nasal Cannula Oxygen Therapy versus Non-Invasive Ventilation in AIDS Patients with Acute Respiratory Failure: A Randomized Controlled Trial. Journal of Clinical Medicine, 12(4), 1679.
Full text: Click here
Publication 2023
Acidosis Arteries Blood Gas Analysis Body Weight Catabolism Cell Respiration Claustrophobia Consciousness Dyspnea Feelings Gastric Dilatation Gender Headache Intubation Medical Devices Nose Patients physiology Pressure Pressure, Diastolic Rate, Heart Respiratory Rate Saturation of Peripheral Oxygen Signs, Vital Skin Systolic Pressure Vision Visual Analog Pain Scale
5
Exclusion Criteria for Noninvasive Ventilation
Patients with hemodynamic instability (cardiogenic shock, myocardial infarction), coma and severe hypoxemia (SpO2 < 60%) or acidosis (pH < 7.1) were excluded. Patients with contraindications to NIV (Glasgow score < 12, swallowing disorder, severe bronchial obstruction, massive retention of secretions despite bronchoscopy, vomiting, those with anatomical or functional upper airway obstruction or ongoing upper gastrointestinal bleeding or ileus), non-cooperative patients as well as those who refused to give consent were also excluded.
Bel Haj Ali K., Sekma A., Chamtouri I., Beltaief K., Msolli M.A., Mezgar Z., Bouida W., Boukef R., Boubaker H., Grissa M.H, & Nouira S. (2023). Pulse amplitude ratio under noninvasive ventilation as a new method in the diagnosis of left heart failure in patients with acute exacerbation of chronic obstructive pulmonary disease. BMC Cardiovascular Disorders, 23, 105.
Full text: Click here
Publication 2023
Acidosis Airway Obstruction Bronchi Bronchoscopy Comatose Deglutition Disorders Hemodynamics Ileus Myocardial Infarction Patients Retention (Psychology) Saturation of Peripheral Oxygen Secretions, Bodily Shock, Cardiogenic

Top products related to «Acidosis»

1
L lactate assay kit by Cayman Chemical
Sourced in United States
The L-Lactate Assay Kit is a colorimetric assay designed to quantify L-lactate levels in biological samples. The kit provides a simple and reliable method for the detection and measurement of L-lactate concentrations.
2
Gem premier 3000 by Werfen
Sourced in United States, Italy, United Kingdom
The GEM Premier 3000 is a blood gas and electrolyte analyzer designed for use in clinical laboratories. It provides automated analysis of various parameters, including pH, blood gases, and electrolytes, in patient samples.
3
Ain 76a rodent chow by Research Diets
Sourced in Canada
AIN-76A is a purified, nutritionally complete rodent chow formulated to meet the dietary needs of laboratory rodents. It provides a balanced source of proteins, carbohydrates, fats, vitamins, and minerals essential for rodent health and growth.
4
Ultraflux av1000s hollow fiber hemofilter by Fresenius
Sourced in Germany
The Ultraflux® AV1000S is a hollow-fiber hemofilter used in renal replacement therapy. It features a semi-permeable membrane that allows for the removal of waste products and excess fluids from the blood.
5
Multifiltrate by Fresenius
Sourced in Germany
The MultiFiltrate is a blood purification device used in renal replacement therapy. It is designed to filter and remove waste, electrolytes, and excess fluid from the blood. The MultiFiltrate utilizes a semi-permeable membrane to selectively filter and control the balance of substances in the bloodstream.
6
Statistica by StatSoft
Sourced in United States, Poland, France, Germany, Czechia, Italy, Sweden, Austria, Canada, Belgium, United Kingdom
Statistica is a comprehensive data analysis and visualization software suite. It provides a wide range of statistical and analytical tools for researchers, scientists, and professionals across various industries. The core function of Statistica is to enable users to efficiently manage, analyze, and interpret complex data sets, supporting informed decision-making processes.
7
Matlab by MathWorks
Sourced in United States, United Kingdom, Germany, Canada, Japan, Sweden, Austria, Morocco, Switzerland, Australia, Belgium, Italy, Netherlands, China, France, Denmark, Norway, Hungary, Malaysia, Israel, Finland, Spain
MATLAB is a high-performance programming language and numerical computing environment used for scientific and engineering calculations, data analysis, and visualization. It provides a comprehensive set of tools for solving complex mathematical and computational problems.
8
Facscanto 2 by BD
Sourced in United States, Germany, United Kingdom, Belgium, China, Australia, France, Japan, Italy, Spain, Switzerland, Canada, Uruguay, Netherlands, Czechia, Jersey, Brazil, Denmark, Singapore, Austria, India, Panama
The FACSCanto II is a flow cytometer instrument designed for multi-parameter analysis of single cells. It features a solid-state diode laser and up to four fluorescence detectors for simultaneous measurement of multiple cellular parameters.
9
Ammonia assay kit by Merck Group
Sourced in United States, Germany
The Ammonia Assay Kit is a laboratory tool designed to quantify the concentration of ammonia in various sample types. It provides a colorimetric method for the determination of ammonia levels. The kit includes all the necessary reagents and instructions for performing the assay.

More about "Acidosis"

Acidosis is a medical condition characterized by an abnormal increase in the acidity of the body's fluids, often resulting from an imbalance in the production or excretion of acids.
This condition, also known as acidaemia, can have various causes, including respiratory disorders, metabolic disturbances, and dietary factors.
Researchers can utilize PubCompare.ai's AI-driven platform to enhance the reproducibility and accuracy of their acidosis studies, locating the best protocols from literature, pre-prints, and patents using intelligent comparisons to identify the most effective approaches and optimize their research for improved results.
Acidosis can lead to a range of symptoms, such as fatigue, confusion, and even life-threatening complications if left untreated.
Accurate diagnosis and effective management of acidosis are crucial for maintaining overall health and well-being.
Clinicians may use L-Lactate Assay Kits or GEM Premier 3000 analyzers to assess lactic acid levels, while researchers may utilize AIN-76A rodent chow or Ultraflux® AV1000S hollow-fiber hemofilters to study acidosis in animal models.
Additionally, tools like MultiFiltrate, Statistica, MATLAB, and FACSCanto II can aid in data analysis and interpretation.
Abbreviations related to acidosis include pH (potential of hydrogen), pCO2 (partial pressure of carbon dioxide), and HCO3- (bicarbonate).
Subtopics within the field of acidosis research include respiratory acidosis, metabolic acidosis, diabetic ketoacidosis, lactic acidosis, and renal tubular acidosis.
By incorporating these synonyms, related terms, and key subtopics, researchers can enhance the discoverability and relevance of their acidosis-related content, ultimately improving the reproducibility and accuracy of their studies.