The study sample included all patients who had thiopurine metabolite analysis, complete blood count, and a comprehensive chemistry panel drawn within a 24 hour period at the University of Michigan between 1 May 2004 and 31 August 2006 and is described in greater detail in the manuscript.1 (link) This study was approved by the University of Michigan Medical Institutional Review Board with a waiver of explicit consent from the participants. The patient sample included 774 cases in a total of 346 individuals. For the analysis of the outcome of clinical response to thiopurines, 5 exclusion criteria were applied: exclusion of patients who did not have IBD, exclusion of patients who had not started on thiopurines at the time when the metabolites were measured, exclusion of patients on biological antitumour necrosis factor therapy, exclusion of patients without documentation of their clinical status at the time of laboratory measurement, and exclusion of patients who had an infection that confounded assessment of clinical response. This data set was used as the basis of a predictive model to identify patients with clinical response to thiopurine immune suppressant medication with a c statistic of 0.86.1 (link)
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Comprehensive Metabolic Panel
Comprehensive Metabolic Panel
The Comprehensive Metabolic Panel (CMP) is a common laboratory test that provides a comprehensive assessment of a person's metabolic status.
It measures a variety of serum chemicals and electrolytes, including glucose, kidney and liver function markers, and other key metabolic indicators.
The CMP is often used as a screening tool to evaluate overall health and detect potential imbalances or underlying medical conditions.
Clinicians may order a CMP as part of a routine checkup or to investigate specific symptoms or concerns.
The test results can provide valuable insights into a patient's metabolic profile and help guide further diagnostic evaluation and treatment planning.
Optimizing CMP research through techniques like PubCompare.ai can enhance the reproducibility and accuracy of these important clinical measurements.
It measures a variety of serum chemicals and electrolytes, including glucose, kidney and liver function markers, and other key metabolic indicators.
The CMP is often used as a screening tool to evaluate overall health and detect potential imbalances or underlying medical conditions.
Clinicians may order a CMP as part of a routine checkup or to investigate specific symptoms or concerns.
The test results can provide valuable insights into a patient's metabolic profile and help guide further diagnostic evaluation and treatment planning.
Optimizing CMP research through techniques like PubCompare.ai can enhance the reproducibility and accuracy of these important clinical measurements.
Most cited protocols related to «Comprehensive Metabolic Panel»
Complete Blood Count
Comprehensive Metabolic Panel
Ethics Committees, Research
Immunosuppressive Agents
Infection
Necrosis
Patients
Pharmaceutical Preparations
Therapies, Biological
Adult
Blood Pressure
Bone Density
Child
Chronic Condition
Complete Blood Count
Comprehensive Metabolic Panel
Diagnosis
Echocardiography
Electrocardiography, 12-Lead
Eligibility Determination
Ethnicity
Gonads
Health Risk Assessment
Hemoglobin A, Glycosylated
Insulin
Lipids
Malignant Neoplasms
Measure, Body
Neoplasms
Operative Surgical Procedures
Parent
Physical Examination
Rate, Heart
Recurrence
Survivors
Thyroid Gland
Urinalysis
KEEPS was designed as a randomized, placebo-controlled, double-blinded, prospective trial (KEEPS; NCT000154180) to evaluate effects of MHT on progression of atherosclerosis as defined by carotid intima–media thickness (CIMT) [44 (link)] and coronary arterial calcification (CAC) [8 (link), 73 (link)] in women who more closely match the age of initiation of MHT reported by prior observational studies. Women meeting inclusion criteria subsequently were randomized to daily placebo, oral CEE, or transdermal 17β-estradiol with placebo or pulsed progesterone for 12 days/month. The detailed inclusion and exclusion criteria for KEEPS have been published elsewhere [36 (link)]. In brief, women between the ages of 42 and 58 years of age who were at least 6 months and no more than 36 months from their last menses with plasma follicle-stimulating hormone (FSH) level ≥35 ng/mL and/or E2 levels <40 pg/mL were eligible. A history of clinical CVD including myocardial infarction, angina, congestive heart failure, or thromboembolic disease excluded women from KEEPS. Other major cardiovascular risk factors excluding participation were current heavy smoking (more than ten cigarettes/day by self-report), morbid obesity [body mass index (BMI) >35 mm2/kg], dyslipidemia (LDL cholesterol >190 mg/dL), hypertriglyceridemia (triglycerides >400 mg/dL), and uncontrolled hypertension (systolic blood pressure >150 mm Hg and/or diastolic blood pressure >95 mm Hg) and glucose >126 mg/dL. Complete blood count and chemistry panel, estradiol, and FSH were performed at the clinical laboratories at each recruiting center. Lipid profiles and thyroid-stimulating hormone (TSH) were performed at the Kronos Science Laboratories (Phoenix, AZ, USA). At screening, women were asked to rank their menopausal symptoms (hotflashes, night sweats, vaginal dryness, dyspareunia, palpitations, insomnia, depression, mood swings, and irritability) as either none, mild, moderate, or severe. Finally, all subjects were screened for CAC and women with Agatston score ≥50 U, indicating significant subclinical coronary artery disease, were excluded. All women meeting inclusion criteria underwent baseline measurements of CIMT by B-mode ultrasound [44 (link)]. All imaging results are read centrally by individuals blinded to participant demographics (CAC at the Los Angeles Biomedical Research Institute at Harbor-UCLA, Torrance, CA, USA under the direction of Dr. M. Budoff and CIMT at the Atherosclerosis Research Unit Core Imaging and Reading Center, University of Southern California, Los Angeles, CA, USA under the direction of Dr. H. Hodis).
Analysis of variance was used to determine statistical significance except where an alternative test is specified. Statistical significance was accepted at P < 0.05.
Analysis of variance was used to determine statistical significance except where an alternative test is specified. Statistical significance was accepted at P < 0.05.
Angina Pectoris
Artery, Coronary
Atherosclerosis
Calcinosis
Carotid Intima-Media Thickness
Cholesterol, beta-Lipoprotein
Clinical Laboratory Services
Complete Blood Count
Comprehensive Metabolic Panel
Congestive Heart Failure
Coronary Arteriosclerosis
Desiccation
Disease Progression
Dyslipidemias
Estradiol
Glucose
High Blood Pressures
Human Follicle Stimulating Hormone
Hypertriglyceridemia
Index, Body Mass
Lipids
Menopause
Menstruation
Mood
Myocardial Infarction
Obesity, Morbid
Placebos
Plasma
Pressure, Diastolic
Progesterone
Sleeplessness
Sweat
Systolic Pressure
Thromboembolism
Thyrotropin
Triglycerides
Ultrasonography
Vagina
Woman
Adult
Blood Pressure
Body Composition
Child
Complete Blood Count
Comprehensive Metabolic Panel
Electrocardiography, 12-Lead
Exercise, Aerobic
Malignant Neoplasms
Muscle Strength
Neoplasms
Physical Examination
Range of Motion, Articular
Rate, Heart
Survivors of Childhood Cancer
Urinalysis
We extracted the following information from the VA databases: rehabilitation visits (physical and occupational therapy), rheumatology visits, plain radiographs (hand, wrist, foot, ankle and cervical spine), extra-articular manifestations (pulmonary, soft tissue nodules, Felty's syndrome and Sjogren's syndrome), number of inflammatory marker (CRP and ESR) tests, number of platelet counts and chemistry panels ordered, rheumatoid factor testing, joint surgery (hand, wrist, knee, foot, ankle, elbow, cervical spine and shoulder) and DMARD use. The administrative study data period included both the one-year (1 July 1999 to 29 June 2000) and two-year (1 July 1 1998 to 29 June 2000) period before the one-year chart review study period.
Each physical therapy and occupational therapy visit was counted as a rehabilitation visit. Tests for CRP and ESR were aggregated into one category. Tests performed on the same day counted as separate tests. The number of hand, wrist, foot, ankle and cervical spine radiographs were also added together into one category. Three methods were used to count the number of prescriptions in a given year. First, we counted the total number of prescriptions (including repeat prescriptions) for the following 10 medications: auranofin, aurothioglucose, azathioprine, cyclosporine, etanercept (Enbrel, Amgen), hydroxychloroquine, infliximab (Remicade, Centocor), leflunomide, methotrexate and sulfasalazine (adalimumab, abatacept and rituximab were not yet available for RA). For the second method, prescriptions for each DMARD were counted once and added to obtain the total number of different DMARDs. For the third method, synthetic DMARDs and biological DMARDs were counted separately. Prescription for each type of DMARD was counted only once and then added together to obtain the total number of different synthetic DMARDs and biological DMARDs.
Each physical therapy and occupational therapy visit was counted as a rehabilitation visit. Tests for CRP and ESR were aggregated into one category. Tests performed on the same day counted as separate tests. The number of hand, wrist, foot, ankle and cervical spine radiographs were also added together into one category. Three methods were used to count the number of prescriptions in a given year. First, we counted the total number of prescriptions (including repeat prescriptions) for the following 10 medications: auranofin, aurothioglucose, azathioprine, cyclosporine, etanercept (Enbrel, Amgen), hydroxychloroquine, infliximab (Remicade, Centocor), leflunomide, methotrexate and sulfasalazine (adalimumab, abatacept and rituximab were not yet available for RA). For the second method, prescriptions for each DMARD were counted once and added to obtain the total number of different DMARDs. For the third method, synthetic DMARDs and biological DMARDs were counted separately. Prescription for each type of DMARD was counted only once and then added together to obtain the total number of different synthetic DMARDs and biological DMARDs.
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Abatacept
Adalimumab
Ankle
Antirheumatic Drugs, Disease-Modifying
Auranofin
Aurothioglucose
Azathioprine
Biopharmaceuticals
Cervical Vertebrae
Comprehensive Metabolic Panel
Cyclosporine
Elbow
Enbrel
Etanercept
Felty Syndrome
Foot
Hydroxychloroquine
Inflammation
Infliximab
Joints
Knee Joint
Leflunomide
Methotrexate
Operative Surgical Procedures
Pharmaceutical Preparations
Physical Examination
Platelet Counts, Blood
Rehabilitation
Remicade
Rheumatoid Factor
Rituximab
Shoulder
Sjogren's Syndrome
Sulfasalazine
Therapies, Occupational
Therapy, Physical
Tissues
Wrist Joint
X-Rays, Diagnostic
Most recents protocols related to «Comprehensive Metabolic Panel»
Protocol full text hidden due to copyright restrictions
Open the protocol to access the free full text link
Albumins
Aspartate
Blood Platelets
Comprehensive Metabolic Panel
D-Alanine Transaminase
Diabetes Mellitus
Diagnosis
Fibrosis
Fibrosis, Liver
Hypersensitivity
Injuries
Lavandula angustifolia
Liver
Liver Cirrhosis
Serum
Transferase
At baseline, CVRFs were classified during the HABS‐HD consensus review based on lab values, objective measures, self‐report, and current medication use.16 We defined hypertension as at least two readings of systolic blood pressure ≥140 mm Hg, diastolic blood pressure ≥90 mm Hg, or with a medical history and use of medications. We defined diabetes as hemoglobin A1c (HbA1c) ≥ 6.5% or with a medical history and use of medications. We defined dyslipidemia as low‐density lipoprotein (LDL) cholesterol ≥120 mg/dL, total cholesterol ≥240 mg/dL, triglycerides ≥200 mg/dL, or with a medical history and use of medications. Cigarette smoking was based on self‐reported current smoking. We used height and weight from the baseline exam to calculate body mass index (BMI) and defined obesity as a BMI ≥30 kg/m2. Participants underwent a collection of fasting blood at baseline, which was then assayed for a comprehensive metabolic panel, lipid panel, and HbA1c. Information on systolic and diastolic blood pressure, HbA1c, height and weight, LDL cholesterol, total cholesterol, high‐density lipoprotein (HDL) cholesterol, and triglycerides were collected at the baseline exam.
We assessed aggregated CVRF burden using the Framingham Risk Score (FRS),25 a sex‐specific algorithm based on age, total cholesterol, HDL cholesterol, systolic and diastolic blood pressures, smoking, and diabetes. FRS was initially developed to assess risk of coronary heart disease (CHD),25 , 26 and has been shown to be associated with cognitive decline and brain pathology.27 , 28 , 29 We defined low, intermediate, and high FRS as having 10‐year CHD risk of <10% (female FRS <10; male FRS <6), 10% to 19% (female FRS 10‐14; male FRS 6‐8), and ≥ 20% (female FRS ≥15 male FRS ≥9), respectively.25
We assessed aggregated CVRF burden using the Framingham Risk Score (FRS),
BLOOD
Brain
Cholesterol
Cholesterol, beta-Lipoprotein
Comprehensive Metabolic Panel
Diabetes Mellitus
Disorders, Cognitive
Dyslipidemias
Heart Disease, Coronary
Hemoglobin A, Glycosylated
High Blood Pressures
High Density Lipoprotein Cholesterol
Index, Body Mass
Lipids
Low-Density Lipoproteins
Males
Obesity
Pharmaceutical Preparations
Pressure, Diastolic
Systole
Systolic Pressure
Triglycerides
Woman
This prospective study included a cohort of adults with newly diagnosed HCC with and without HIV enrolled in an NCI-funded study examining epigenomic biomarkers associated with HIV-associated HCC (Award number U54CA221205). Subjects with HCC were recruited between August 2018 and November 2021 from the Hepatology clinics and inpatients wards of both the Jos University Teaching Hospital (JUTH) and Lagos University Teaching Hospitals (LUTH), and their affiliated sites in Nigeria.
Inclusion criteria were: a) ≥18yrs; b) confirmed to have HCC diagnosed by ultrasound and computed tomography (CT) scan. Exclusion criteria were: a) other known primary or secondary malignancy either currently or within the past 5 years. HCC was diagnosed based on radiologic criteria using triple phase CT according to the AASLD 2018 guidelines [15 (link)], which was offered to all patients who had a ≥1cm nodule on ultrasound and/or risk factors for HCC (i.e. HIV, HBV, HCV infection or cirrhosis of any etiology), and/or other symptoms and signs suggestive of HCC. CT findings of arterial phase hyperenhancement and washout during the portal venous phase were considered diagnostic. A liver biopsy was not required for diagnosis, and was not performed in any of the study subjects.
All study subjects confirmed with HCC by CT were provided with information about the study and asked to sign an informed consent. At their enrollment visit, participants underwent a physical exam by the study physician and laboratory testing for HIV (ELISA and Western Blot); complete blood count (CBC), comprehensive chemistry panel including albumin, total bilirubin, aspartate aminotransferase (AST), alanine aminotransferase (ALT), viral serology including hepatitis B surface antigen (HBsAg) and antibody (anti-HBs) and HCV antibody (anti-HCV) using Lumi Quick diagnostics rapid test kits, and alpha-fetoprotein (AFP). Plasma samples were also collected for methylomic analysis. Patients who were confirmed HIV seropositive, also had CD4+ T cell count (flow cytometry (Partec GmbH, Munster, Germany), and HIV RNA Roche Ampliprep TaqMan (Roche Diagnostics Germany; lower limit of detection (LLD) of 20 cp/ml) testing performed and were referred immediately to the HIV care and treatment clinic at their respective institutions for further treatment and care. Confirmatory HBV and HCV viral load testing on stored specimens was performed on all HBsAg and anti-HCV positive participants using the Cepheid GeneXpert® system. Participants received the usual care and treatment for HCC, HIV, viral hepatitis and other health conditions, according to Nigerian national and international guidelines [16 , 17 (link)]. Sorafenib is offered to eligible patients with advanced disease and Child Pugh Turcotte (CPT) A where available. However, none of the study participants in the study received sorafenib or any other interventional therapies due to lack of access and/or high cost of the medication.
Inclusion criteria were: a) ≥18yrs; b) confirmed to have HCC diagnosed by ultrasound and computed tomography (CT) scan. Exclusion criteria were: a) other known primary or secondary malignancy either currently or within the past 5 years. HCC was diagnosed based on radiologic criteria using triple phase CT according to the AASLD 2018 guidelines [15 (link)], which was offered to all patients who had a ≥1cm nodule on ultrasound and/or risk factors for HCC (i.e. HIV, HBV, HCV infection or cirrhosis of any etiology), and/or other symptoms and signs suggestive of HCC. CT findings of arterial phase hyperenhancement and washout during the portal venous phase were considered diagnostic. A liver biopsy was not required for diagnosis, and was not performed in any of the study subjects.
All study subjects confirmed with HCC by CT were provided with information about the study and asked to sign an informed consent. At their enrollment visit, participants underwent a physical exam by the study physician and laboratory testing for HIV (ELISA and Western Blot); complete blood count (CBC), comprehensive chemistry panel including albumin, total bilirubin, aspartate aminotransferase (AST), alanine aminotransferase (ALT), viral serology including hepatitis B surface antigen (HBsAg) and antibody (anti-HBs) and HCV antibody (anti-HCV) using Lumi Quick diagnostics rapid test kits, and alpha-fetoprotein (AFP). Plasma samples were also collected for methylomic analysis. Patients who were confirmed HIV seropositive, also had CD4+ T cell count (flow cytometry (Partec GmbH, Munster, Germany), and HIV RNA Roche Ampliprep TaqMan (Roche Diagnostics Germany; lower limit of detection (LLD) of 20 cp/ml) testing performed and were referred immediately to the HIV care and treatment clinic at their respective institutions for further treatment and care. Confirmatory HBV and HCV viral load testing on stored specimens was performed on all HBsAg and anti-HCV positive participants using the Cepheid GeneXpert® system. Participants received the usual care and treatment for HCC, HIV, viral hepatitis and other health conditions, according to Nigerian national and international guidelines [16 , 17 (link)]. Sorafenib is offered to eligible patients with advanced disease and Child Pugh Turcotte (CPT) A where available. However, none of the study participants in the study received sorafenib or any other interventional therapies due to lack of access and/or high cost of the medication.
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Adult
Albumins
alpha-Fetoproteins
Antibodies
Arteries
Aspartate Transaminase
Bilirubin
Biological Markers
Biopsy
CD4+ Cell Counts
Child
Comprehensive Metabolic Panel
D-Alanine Transaminase
Diagnosis
Enzyme-Linked Immunosorbent Assay
Flow Cytometry
Hepatitis B Surface Antigens
Hepatitis C Antibodies
Hepatitis Viruses
Inpatient
Liver
Liver Cirrhosis
Malignant Neoplasms
Patients
Physical Examination
Physicians
Plasma
Radionuclide Imaging
Reagent Kits, Diagnostic
Sorafenib
Ultrasonics
Veins, Portal
Western Blotting
X-Ray Computed Tomography
Group C subjects received baseline analytical measurements (comprehensive metabolic panel, complete blood count, lipid panel, lactate, β-hydroxybutyrate, β-ketopentanoate and β-hydroxypentanoate) and physical examination. They then observed an overnight fast. C7 was calculated at a daily dose of 45% of daily caloric intake. The following morning, two doses were administered at approximately 6 AM (before breakfast) and 10 AM (before lunch), each containing ¼ of the total daily dose. Before lunch, at 12–1 PM, blood was collected for determination of ketone bodies as described31 (link), approximately 21 h after the baseline analysis. Figure 2 illustrates this experimental sequence.![]()
Stimulation of C5 ketogenesis by C7. Plasma levels of C5 ketones before and after two C7 doses. Top and middle panels illustrate C5 ketone values before and after an overnight fasting period followed by two C7 doses. The baseline (pre-C7) values were obtained at 2–2:30 PM. Two C7 doses (each ¼ of the MTD) were administered approximately at 6 AM and 10 AM. The only morning meal was breakfast approximately at 8 AM. Each color symbol and time identifies one individual.
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BLOOD
Complete Blood Count
Comprehensive Metabolic Panel
Hydroxybutyrates
Ketone Bodies
Ketones
Lactates
Lipids
Physical Examination
Plasma
All experiments measuring ASO activity in animals were approved by the Institutional Animal Care and Use Committee of University of California, Irvine. Six- to eight-week old, male Balbc/J mice were purchased from the Jackson Laboratory and acclimated to the university vivarium for at least 7 days prior to experimentation. Mice were housed under a 12 h light/dark cycle at 20–22°C in groups of 2–4. Cages contained 1/8′ corncob bedding (7092A, Envigo, Huntingdon, UK) enriched with ∼6 g of cotton fiber nestlets (Ancare, Corp., Bellmore, NY). Mice were fed the vivarium stock diet (chow, 2020x, Envigo). Access to food and water was ad libitum. For oral administration of SH-BC-893, polypropylene feeding tubes (20 g × 38 mm; Instech Laboratories Inc., Plymouth, PA) were used to dose 120 mg/kg of SH-BC-893 dissolved in H2O (stock = 24 mg/ml). To aid gavage by inducing salivation, feeding tubes were dipped into a 1 g/ml sucrose solution immediately prior to treatment. For subcutaneous administration, ASOs were dissolved in PBS at a concentration such that 10 ml/kg was administered using a 27 G needle. For blood chemistry analysis, blood was collected by decapitation from nine-week old male Balbc/J mice. Serum was separated from whole blood in a SST-MINI tube with clot activator gel (Greiner, cat# 450571VET). Serum samples were sent to IDEXX Bioanalytics for a comprehensive chemistry panel (cat# 6006).
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Administration, Oral
Animals
BLOOD
Blood Chemical Analysis
Clotrimazole
Comprehensive Metabolic Panel
Decapitation
Diet
Food
Hematologic Tests
Institutional Animal Care and Use Committees
Males
Mice, House
Needles
Polypropylenes
Serum
Sialorrhea
Suby's G solution
Sucrose
Tube Feeding
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Amplicor is a lab equipment product designed for nucleic acid amplification. It is a fully automated system used for the detection and quantification of target DNA or RNA sequences.
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The Piccolo General Chemistry 13 Panel discs are a diagnostic lab equipment product designed for comprehensive metabolic panel testing. The discs provide an automated and efficient way to analyze multiple analytes, including electrolytes, kidney and liver function markers, and other key chemical indicators, from a single sample. The product is intended to support clinical decision-making by providing accurate and reliable test results.
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The Piccolo general chemistry 13 panel is a laboratory equipment product offered by Abaxis. It is designed to perform a comprehensive panel of general chemistry tests. The device provides analytical results for 13 key biomarkers in a single sample. The core function of the Piccolo general chemistry 13 panel is to enable efficient and accurate testing across a range of common clinical analytes.
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The General Chemistry 13 panel is a comprehensive diagnostic tool designed to provide a broad analysis of an individual's overall health status. This panel includes the measurement of various biochemical markers such as electrolytes, liver enzymes, kidney function indicators, and other related parameters. The General Chemistry 13 panel is intended to assist healthcare professionals in assessing an individual's general health and detecting potential imbalances or abnormalities that may require further investigation or treatment.
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More about "Comprehensive Metabolic Panel"
The Comprehensive Metabolic Panel (CMP) is a widely used laboratory test that provides a comprehensive assessment of an individual's metabolic status.
This panel measures a variety of serum chemicals and electrolytes, including glucose, kidney and liver function markers, and other key metabolic indicators.
The CMP is often employed as a screening tool to evaluate overall health and detect potential imbalances or underlying medical conditions.
Clinicians may order a CMP as part of a routine checkup or to investigate specific symptoms or concerns.
The test results can offer valuable insights into a patient's metabolic profile and help guide further diagnostic evaluation and treatment planning.
Optimizing CMP research through techniques like PubCompare.ai can enhance the reproducibility and accuracy of these important clinical measurements.
Synonyms and related terms for the CMP include the General Chemistry 13 panel, which is a similar test that measures the same set of metabolic markers.
The Piccolo General Chemistry 13 Panel discs and the Piccolo Xpress Chemistry Analyzer are examples of laboratory equipment used to perform the CMP.
The Hemavet 950FS and IDEXX ProCyte DX analyzer are other instruments that can be used for related blood tests.
Abbreviations commonly used for the Comprehensive Metabolic Panel include CMP and GC13.
The BD Vacutainer is a type of blood collection tube often used for CMP testing.
By leveraging the insights from these related terms and technologies, researchers can optimize their CMP research and enhance the reliability and accuracy of their findings.
This panel measures a variety of serum chemicals and electrolytes, including glucose, kidney and liver function markers, and other key metabolic indicators.
The CMP is often employed as a screening tool to evaluate overall health and detect potential imbalances or underlying medical conditions.
Clinicians may order a CMP as part of a routine checkup or to investigate specific symptoms or concerns.
The test results can offer valuable insights into a patient's metabolic profile and help guide further diagnostic evaluation and treatment planning.
Optimizing CMP research through techniques like PubCompare.ai can enhance the reproducibility and accuracy of these important clinical measurements.
Synonyms and related terms for the CMP include the General Chemistry 13 panel, which is a similar test that measures the same set of metabolic markers.
The Piccolo General Chemistry 13 Panel discs and the Piccolo Xpress Chemistry Analyzer are examples of laboratory equipment used to perform the CMP.
The Hemavet 950FS and IDEXX ProCyte DX analyzer are other instruments that can be used for related blood tests.
Abbreviations commonly used for the Comprehensive Metabolic Panel include CMP and GC13.
The BD Vacutainer is a type of blood collection tube often used for CMP testing.
By leveraging the insights from these related terms and technologies, researchers can optimize their CMP research and enhance the reliability and accuracy of their findings.