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S961 peptide

Manufactured by Novo Nordisk
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Sourced in Denmark

The S961 peptide is a laboratory research tool used in scientific investigations. It is a synthetic peptide with a specific amino acid sequence. The core function of the S961 peptide is to serve as a research reagent for experimental purposes. No further details on its intended use or applications can be provided in an unbiased and factual manner.

Automatically generated - may contain errors

Lab products found in correlation

Puromycin solution, by Merck Group (2 mentions) Geneticin g418 sulfate solution, by Thermo Fisher Scientific (2 mentions)

2 protocols using s961 peptide

1

Reagent Acquisition for IGF-IR and InsR Signaling

Cited 1 time
Check if the same lab product or an alternative is used in the 5 most similar protocols
IGF-I, IGF-II and insulin were purchased from Gemini and Eli Lily, respectively. Puromycin solution was purchased from Sigma-Aldrich. Geneticin (G418 sulfate) solution was purchased from Life Technologies. Humanized anti-IGF1R monoclonal antibody HuEM164 was generously provided by Immunogen Inc. Anti-InsR monoclonal antibody, alpha subunit clone 83-7 was purchased from EMD Millipore. S961 peptide was generously provided by Novo Nordisk, Denmark (27 (link)).
Chan J.Y., LaPara K, & Yee D. (2016). Disruption of insulin receptor function inhibits proliferation in endocrine resistant breast cancer cells. Oncogene, 35(32), 4235-4243.
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2

Reagent Acquisition for IGF-IR and InsR Signaling

Cited 1 time
Check if the same lab product or an alternative is used in the 5 most similar protocols
IGF-I, IGF-II and insulin were purchased from Gemini and Eli Lily, respectively. Puromycin solution was purchased from Sigma-Aldrich. Geneticin (G418 sulfate) solution was purchased from Life Technologies. Humanized anti-IGF1R monoclonal antibody HuEM164 was generously provided by Immunogen Inc. Anti-InsR monoclonal antibody, alpha subunit clone 83-7 was purchased from EMD Millipore. S961 peptide was generously provided by Novo Nordisk, Denmark (27 (link)).
Chan J.Y., LaPara K, & Yee D. (2016). Disruption of insulin receptor function inhibits proliferation in endocrine resistant breast cancer cells. Oncogene, 35(32), 4235-4243.
+ Open protocol
+ Expand

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