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Tocrifluor t1117

Manufactured by Bio-Techne
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Sourced in United States

Tocrifluor T1117 is a fluorescent probe used for the detection of thiol-containing compounds. It is a membrane-permeable, zinc-chelating fluorophore that specifically reacts with free thiols, resulting in a fluorescence enhancement. Tocrifluor T1117 can be utilized for the quantification of thiol-containing analytes in various biological samples.

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Lab products found in correlation

O 1602, by Cayman Chemical (1 mentions) Cid16020046, by Bio-Techne (1 mentions) Forskolin, by Bio-Techne (1 mentions) Ici 118 551, by Merck Group (1 mentions) Salmeterol, by Merck Group (1 mentions) 14 22 amide, by Bio-Techne (1 mentions) O 1602, by Bio-Techne (1 mentions)

Close spelling variants of this product

Tocrifluor

2 protocols using tocrifluor t1117

1

Cannabinoid Receptor Agonist and Antagonist Evaluation

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Cocaine, nicotine and Δ9-THC was provided by the NIDA pharmacy (Baltimore, MD). O-1602 was supplied by Cayman Chemical (Cat#: 10006803). The selective GPR55 antagonist CID 16020046 (CID, Cat#: 4959) and Tocrifluor T1117 were purchased from Tocris Bioscience. For the more in detail, please see Supplmentary Information.
Xi Z.X., He Y., Shen H., Bi G.H., Zhang H.Y., Soler-Cedeno O., Alton H, & Yang Y. (2023). GPR55 is expressed in glutamate neurons and functionally modulates nicotine taking and seeking in rats and mice. Research Square.
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2

Synthesis and Use of Fenoterol Analogs

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(R,R′)-Fenoterol [(R,R′)-Fen], (R,S′)-4′-methoxy-1-naphthylfenoterol [(R,S′)-MNF], and (R,R′)-MNF were synthesized as described previously28 (link). The following compounds from Tocris Bioscience (Ellisville, MO, USA) were used for cell treatments: O-1602 (GPR55 agonist), Tocrifluor T1117 (T1117, fluorescent GPR55 agonist), myristoylated protein kinase inhibitor-(14–22)-amide (PKI), and forskolin (adenylyl cyclase activator). Salmeterol (selective β2-AR agonist) and ICI-118,551 (ICI, selective β2-AR inverse agonist) were purchased from Sigma-Aldrich (St-Louis, MO, USA).
Wnorowski A., Dudzik D., Bernier M., Wójcik J., Keijzers G., Diaz-Ruiz A., Mazur K., Zhang Y., Han H., Scheibye-Knudsen M., Jozwiak K., Barbas C, & Wainer I.W. (2022). Deprogramming metabolism in pancreatic cancer with a bi-functional GPR55 inhibitor and biased β2 adrenergic agonist. Scientific Reports, 12, 3618.
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