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Accu check compact

Manufactured by Roche
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Sourced in Spain, United States, Germany, India, Switzerland

The Accu-Chek Compact is a blood glucose monitoring system designed for personal use. It is a compact, handheld device used to measure the level of glucose in a small drop of blood. The device provides accurate and reliable blood glucose readings to help individuals with diabetes manage their condition.

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Lab products found in correlation

Elisa kits for insulin, by Cayman Chemical (2 mentions) Tnf α, by Thermo Fisher Scientific (2 mentions) Pilocarpine, by Merck Group (1 mentions) A1c now multitest system, by Bayer (1 mentions) Glucose solution, by Merck Group (1 mentions)

Close spelling variants of this product

Accu-Check (349 citations) Accu-Check Compact Plus (11 citations) Accu-check compact glucometer (2 citations)

8 protocols using accu check compact

1

Glucose and Insulin Tolerance Tests

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For intraperitoneal glucose tolerance tests (ipGTT), animals were fasted for 6 h [26] (link), and blood samples were obtained from the tail vein. Animals were then injected intraperitoneally with 1.5 g/kg body weight of glucose, and blood samples were taken at the indicated intervals.
For intraperitoneal insulin tolerance tests (ipITT), fed animals were used. Animals were injected intraperitoneally with 0.75 IU/kg body weight of soluble insulin, and blood samples were obtained from the tail vein. Blood glucose was measured in each sample using an Accu-check compact glucometer (Roche, Madrid, Spain). Levels of glycemia after insulin injection are expressed as % of glycemia compared to basal glycemia levels in fed state.
ipGTT and ipITT were made at 17 and 28 weeks of age.
García-Arevalo M., Alonso-Magdalena P., Rebelo Dos Santos J., Quesada I., Carneiro E.M, & Nadal A. (2014). Exposure to Bisphenol-A during Pregnancy Partially Mimics the Effects of a High-Fat Diet Altering Glucose Homeostasis and Gene Expression in Adult Male Mice. PLoS ONE, 9(6), e100214.
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2

Pilocarpine-induced Salivation in Mice

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Mice were anesthetized by inhalation of isoflurane (2%), and blood glucose levels were determined using an Accu-Check Compact glucometer (Roche, Indianapolis, IN, USA). Whole saliva was collected for 7 min from the oral cavity, starting at 90 s after intraperitoneal injection of pilocarpine (100 μg/mouse; Sigma-Aldrich, St. Louis, MO, USA). Saliva flow rates were expressed as μL saliva secreted per g body weight per min (μL/g/min).
Hwang S.H., Woo J.S., Moon J., Yang S., Park J.S., Lee J., Choi J., Lee K.H., Kwok S.K., Park S.H, & Cho M.L. (2021). IL-17 and CCR9+α4β7– Th17 Cells Promote Salivary Gland Inflammation, Dysfunction, and Cell Death in Sjögren’s Syndrome. Frontiers in Immunology, 12, 721453.
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3

Rat Models of Retinal Neurodegeneration

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In this study, we used 6-8-week-old Wistar and Lewis rats (Janvier Laboratory, Le Genest-St-Isle, France), and 18-month-old Goto-Kakizaki (GK) rats (a Wistar strain of nonobese type 2 diabetic rats) (Taconic Europe, Denmark). In GK and Wistar rats, non-fasting blood glucose was measured at the tail vein with Accutrend GC and Accu-check compact equipment (Roche) and HbA1c was measured with A1C NOW+ multitest system (Bayer, Germany). We defined a plasma glucose level > 250 mg/dL (14 mmol/L) as diabetic. In contrast to the control Wistar rats, male GK rats develop hyperglycaemia at approximately 14 weeks of age. Control age-matched Wistar rats (WS) were normoglycaemic. The strain is raised in our animal facility and the males only enter the experiments: group formation depended on the number of available males of the needed age. Animal models. Three complementary rat models of retinal neurodegeneration were used:
Berdugo M., Delaunay K., Naud M.C., Guegan J., Moulin A., Savoldelli M., Picard E., Radet L., Jonet L., Djerada Z., Gozalo C., Daruich A., Beltrand J., Jeanny J.C., Kermorvant-Duchemin E., Crisanti P., Polak M, & Behar-Cohen F. (2021). The antidiabetic drug glibenclamide exerts direct retinal neuroprotection. Translational research : the journal of laboratory and clinical medicine, 229.
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4

Oral Glucose Tolerance Test in Rats

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After 8-week treatment, all the rats received oral administration of glucose solution (3 g/kg, Sigma, St. Louis, MO, USA) after 12-hour fasting. Then the level of blood glucose was measured with an Accu-Check Compact glucometer (Roche) via sampling from the tail at 0, 0.5, 1, and 2 hours respectively after glucose intake.
Huang Q., Meng L., Li H., Xiong N., Zeng L., Wang G., Zhang P., Zhao H, & Liu D. (2022). Huoxue Jiangtang Decoction Alleviates Type 2 Diabetes Mellitus by Regulating the Oral Microbiota and Food Preferences. Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy, 15, 3739-3751.
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5

Streptozotocin-Induced Diabetes in Mice

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Diabetes was induced (starting at experimental day 0) in adult (16 week old) male C57BL/6 mice by daily intraperitoneal injections of streptozotocin (50 mg/kg body weight in 0.1 M citrate buffer, pH 4.5) for 5 consecutive days. Controls were given citrate buffer alone. Alendronate was delivered (beginning on day 0) by weekly subcutaneous injections in control (n=8) and diabetic (n=8) mice at a concentration of 2 mg/kg/week, a dose consistent with past mouse studies [Nijenhuis et al., 2008 (link)]. Mice were maintained on a 12-hour light, 12-hour dark cycle at 23°C, given standard lab chow and water ad libitum. Diabetes was confirmed 12 days after initial injection using an Accu-Check compact glucometer (Roche Diagnostics Corporation, Indianapolis, IN) with a drop of blood from the saphenous vein. Mice were euthanized at either 7 or 40 days after the start of diabetes induction. At this time general parameters were measured, including blood glucose levels and total body, tibialis anterior and subcutaneous femoral fat pad mass. Animal procedures were approved by the Michigan State University Institutional Animal Care and Use Committee.
Coe L.M., Tekalur S.A., Shu Y., Baumann M.J, & McCabe L.R. (2015). Bisphosphonate treatment of type I diabetic mice prevents early bone loss but accentuates suppression of bone formation. Journal of cellular physiology, 230(8), 1944-1953.
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6

Comprehensive Metabolic Profiling in Rats

Cited 1 time
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Rats were weighed, anesthetized with isoflurane, and femoral arterial catheters implanted for blood collection and determination of blood glucose (Accu-Check Compact, Roche Diagnostics, Indianapolis, IN) and lipid profile (Cardio-Check PA analyzer, QAS, Orlando, FL). Additional blood was collected in tubes containing EDTA and plasma obtained by centrifugation. Plasma levels of specific proteins were determined using commercial ELISA kits for insulin (Cayman Chemical, Ann Arbor, MI), tumor necrosis factor-α (TNFα) (Thermo Scientific, Waltham, MA), and oxidized low-density lipoprotein (oxLDL) (Mercodia, Winston Salem, NC). Circulating levels of amino acids were quantified by ion exchange chromatography (Molecular Genetics Laboratory at Baylor College of Medicine, Houston, TX). Global arginine bioavailability, which is a more sensitive indicator of disturbances in arginine metabolism than levels of individual amino acids, was calculated as the ratio of plasma arginine to the sum of plasma ornithine plus citrulline.13 (link),14 (link) Following blood collection, animals were heparinized (1000U/kg, iv) and the thoracic aorta and gracilis anticus muscles removed.
Johnson F.K., Peyton K.J., Liu X.M., Azam M.A., Shebib A.R., Johnson R.A, & Durante W. (2014). ARGINASE PROMOTES ENDOTHELIAL DYSFUNCTION AND HYPERTENSION IN OBESE RATS. Obesity (Silver Spring, Md.), 23(2), 383-390.
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7

Comprehensive Metabolic Profiling in Rats

Cited 1 time
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Rats were weighed, anesthetized with isoflurane, and femoral arterial catheters implanted for blood collection and determination of blood glucose (Accu-Check Compact, Roche Diagnostics, Indianapolis, IN) and lipid profile (Cardio-Check PA analyzer, QAS, Orlando, FL). Additional blood was collected in tubes containing EDTA and plasma obtained by centrifugation. Plasma levels of specific proteins were determined using commercial ELISA kits for insulin (Cayman Chemical, Ann Arbor, MI), tumor necrosis factor-α (TNFα) (Thermo Scientific, Waltham, MA), and oxidized low-density lipoprotein (oxLDL) (Mercodia, Winston Salem, NC). Circulating levels of amino acids were quantified by ion exchange chromatography (Molecular Genetics Laboratory at Baylor College of Medicine, Houston, TX). Global arginine bioavailability, which is a more sensitive indicator of disturbances in arginine metabolism than levels of individual amino acids, was calculated as the ratio of plasma arginine to the sum of plasma ornithine plus citrulline.13 (link),14 (link) Following blood collection, animals were heparinized (1000U/kg, iv) and the thoracic aorta and gracilis anticus muscles removed.
Johnson F.K., Peyton K.J., Liu X.M., Azam M.A., Shebib A.R., Johnson R.A, & Durante W. (2014). ARGINASE PROMOTES ENDOTHELIAL DYSFUNCTION AND HYPERTENSION IN OBESE RATS. Obesity (Silver Spring, Md.), 23(2), 383-390.
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8

Glucose Tolerance Test in Mouse Models

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At 8th week, a glucose tolerance test (GTT) was performed in C (n = 8), HFD (n = 24), and HFHSu (n = 11) mice. After 6 h of fasting, mice received an intraperitoneal injection of glucose (1 g/kg) and blood glucose levels were determined from an incision in the tail using Accu‐Check Compact glucometer (Roche Diagnostics, Basel, Switzerland) at 0, 15, 30, 45, 60, 90, and 120 min after injection.
Chaar L.J., Coelho A., Silva N.M., Festuccia W.L, & Antunes V.R. (2016). High‐fat diet‐induced hypertension and autonomic imbalance are associated with an upregulation of CART in the dorsomedial hypothalamus of mice. Physiological Reports, 4(11), e12811.
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