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P aminophenylmurcuric acetate apma

Manufactured by Merck Group
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P-aminophenylmurcuric acetate (APMA) is a laboratory reagent used in biochemical and molecular biology applications. It functions as a catalyst, enhancing the reactivity of certain biomolecules. The core purpose of APMA is to facilitate specific chemical transformations and reactions in experimental settings, without interpretation of its intended use.

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Lab products found in correlation

Pf573228, by Merck Group (2 mentions) Dimethyl sulfoxoide, by Merck Group (2 mentions) Sb 3ct, by MedChemExpress (2 mentions) Pf 562 271, by Merck Group (2 mentions)

2 protocols using p aminophenylmurcuric acetate apma

1

Investigating Dasatinib and MMP Inhibitors

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Dasatinib was from LC labs (Woburn, MA), SB-3CT was purchased from MedChem Express (Monmouth Junction, NJ). The human IL-1β neutralizing antibody was from R&D bio-techne. PF-562,271, PF-573,228, and p-aminophenylmurcuric acetate (APMA), for MMP activation, was purchased from Sigma (St. Louis, MO). All drugs were dissolved in dimethyl sulfoxoide (Sigma).
Kessler B.E., Mishall K.M., Kellett M.D., Clark E.G., Pugazhenthi U., Pozdeyev N., Kim J., Tan A.C, & Schweppe R.E. (2018). Resistance to Src inhibition alters the BRAF-mutant tumor secretome to promote an invasive phenotype and therapeutic escape through a FAK>p130Cas>c-Jun signaling axis. Oncogene, 38(14), 2565-2579.
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2

Inhibiting MMP-Mediated IL-1β Signaling

Check if the same lab product or an alternative is used in the 5 most similar protocols
Dasatinib was from LC labs (Woburn, MA), SB-3CT was purchased from MedChem Express (Monmouth Junction, NJ). The human IL-1β neutralizing antibody was from R&D bio-techne. PF-562,271, PF-573,228, and p-aminophenylmurcuric acetate (APMA), for MMP activation, was purchased from Sigma (St. Louis, MO). All drugs were dissolved in dimethyl sulfoxoide (Sigma).
Kessler B.E., Mishall K.M., Kellett M.D., Clark E.G., Pugazhenthi U., Pozdeyev N., Kim J., Tan A.C, & Schweppe R.E. (2018). Resistance to Src inhibition alters the BRAF-mutant tumor secretome to promote an invasive phenotype and therapeutic escape through a FAK>p130Cas>c-Jun signaling axis. Oncogene, 38(14), 2565-2579.
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