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Portapres

Manufactured by Finapres Medical Systems
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Portapres is a noninvasive, wearable device designed for continuous blood pressure monitoring. It is capable of measuring beat-to-beat blood pressure and related cardiovascular parameters. The Portapres device uses the volume-clamp method to obtain these measurements.

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Lab products found in correlation

Carescape v100, by GE Healthcare (2 mentions) Labchart software, by ADInstruments (2 mentions) Ecg100c module, by Biopac (2 mentions) Biosen c line glucose and lactate analyser, by EKF Diagnostics (2 mentions) Mp150 system with acqknowledge acquisition and analysis software, by Biopac (2 mentions) Ecg100c, by Biopac (1 mentions) Mp150, by Biopac (1 mentions) Ced 1401 mark 2, by Cambridge Electronic Design (1 mentions) Ced 1902, by Cambridge Electronic Design (1 mentions) Pl3508 powerlab 8 53, by ADInstruments (1 mentions) Intellivue mp50, by Philips (1 mentions) Dwl doppler box, by Compumedics (1 mentions) Bio amp, by ADInstruments (1 mentions) Portapres device, by Finapres Medical Systems (1 mentions) Labview software platform, by National Instruments (1 mentions) Ni daq card, by National Instruments (1 mentions) Labview, by National Instruments (1 mentions) Matlab, by National Instruments (1 mentions) Capnomac ultima, by GE Healthcare (1 mentions) Datex as 3, by GE Healthcare (1 mentions)

Spelling variants of this product

Portapres device (5 citations) Portapres Model-2 (4 citations) Portapres system (2 citations)

26 protocols using portapres

1

Cardiovascular Dynamics in Achalasia Patients

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All subjects provided written informed consent, their medical history was screened and underwent a physical examination. The Ethical Committee of the Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán approved the protocol for the physiological monitoring of the achalasia patients.
SBP and HR data were recorded simultaneously with a Portapress® device of Finapres Medical Systems, The Netherlands [71 (link)]. The Portapress® quantifies the blood pressure waveform at the finger with a precision of 1 mmHg and a time resolution of 1 ms, which allows us to derive other hemodynamic parameters. Here only the IBI and the SBP are analyzed, because the diastolic blood pressure (DBP) has an analogous time series as SBP as seen in S1 Fig. IBI is measured in units of seconds and SBP as mmHg. The detailed methodology for time series analysis is given in the S1 Appendix.
Student’s t test was used to compare the different study groups of control subjects and achalasia patients. A value of p<0.05 was considered statistically significant in the hypothesis test of different means of the moments (standard deviation, skewness and kurtosis), HMP α, LF/HF, frequency radius rf, and resonance parameter β.
Rivera A.L., Estañol B., Macias-Gallardo J.J., Delgado-Garcia G., Fossion R., Frank A, & Torres-Villalobos G.M. (2021). Cardiovascular dysautonomia in Achalasia Patients: Blood pressure and heart rate variability alterations. PLoS ONE, 16(3), e0248106.
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2

Non-invasive Cardiovascular Measurements

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Arterial pressure was measured non-invasively using a volume-clamp method (Portapress; Finapres Medical Systems, Enschede, the Netherlands). A cuff was attached to the right index finger and adjusted so that it would be maintained at heart level. HR was calculated using a type II ECG (ECG 100C; BIOPAC Systems, Santa Barbara, CA, USA). We measured the tympanic membrane temperature as the core temperature non-invasively using an earphone-type infrared tympanic thermometer (CE Thermo; Nipro, Osaka, Japan) Kiya et al. (17 (link)). The probe of the thermometer was inserted into the ear canal for measurement.
Arterial pressure was recorded with ECG by outputting the pressure pulse wave as an analog signal from the Portapress. The data of all experiments were stored on a hard disk at a sampling rate of 1,000 Hz through an analog-to- digital converter (MP-150; BIOPAC Systems).
Kubota S., Endo Y., Kubota M., Miyazaki H, & Shigemasa T. (2022). The Pressor Response to the Drinking of Cold Water and Cold Carbonated Water in Healthy Younger and Older Adults. Frontiers in Neurology, 12, 788954.
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3

Multimodal Gait Analysis with Telemetry

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Multichannel electromyogram (EMGs), electrocardiogram (ECG), and foot-contact signals were simultaneously recorded with a telemetry system (MEM-7000, Nihon Kohden, Tokyo, Japan). A foot sensor (LS-023, Tokyo Sensor, Tokyo, Japan) was attached under the heel. Heart rate (HR) was obtained from the R-waves of the ECG. EMG activity of bilateral tibialis anterior (TA), medial gastrocnemius (MG), and vastus lateralis (VL) muscles was recorded with a bandpass filter between 20 Hz and 2 kHz. Mean arterial blood pressure (MAP) was noninvasively measured with a PortaPress (Finapres Medical Systems BV, Arnhem, the Netherlands), whose cuff was attached to the left middle or index finger. Because of measurement artifact caused by finger and/or hand movement during over-ground walking, stable recordings of MAP were obtained in seven subjects only.
Matsukawa K., Asahara R., Ishii K., Kunishi M., Yamashita Y., Hashiguchi Y., Liang N, & Smith S.A. (2020). Increased prefrontal oxygenation prior to and at the onset of over-ground locomotion in humans. Journal of applied physiology (Bethesda, Md. : 1985), 129(5).
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4

Graded Exercise Test on Alticycle

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Participants were instrumented and then rested for at least 2 min while pre-exercise measurements of oxygen uptake (VO2), carbon dioxide production, VE, HR, SBP, and DBP were collected. Participants undertook a 5 min self-paced warm-up and a modified graded exercise test on the Alticycle. This modified graded exercise test began at an intensity of 30% of sea-level Wattmax, with 10% increases every 3 min up to 80% of sea-level Wattmax, followed by 10% increases every minute until volitional fatigue. Expired respiratory gases were analysed breath-by-breath using a Cosmed K4b2 (Metabolic Company, Rome, Italy) portable metabolic system alongside continuous measurements of HR (via three-lead ECG), pulse oximetry (oxygen saturation (SpO2), Datex Ohmeda 3900, GE Healthcare, USA), and beat-to-beat measurements of SBP and DBP by photoplethysmography (Portapres, Finapres Medical Systems BV, Netherlands). The exercise-induced change in SBP and DBP is reported, calculated as the difference between pre-exercise and value obtained at Wattmax.
Lucas S.J., Malein W.L., Thomas O.D., Ashdown K.M., Rue C.A., Joyce K.E., Newman C., Cadigan P., Johnson B., Myers S.D., Myers F.A., Wright A.D., Delamere J., Imray C.H., Bradwell A.R, & Edsell M. (2021). Effect of losartan on performance and physiological responses to exercise at high altitude (5035 m). BMJ Open Sport — Exercise Medicine, 7(1), e000982.
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5

Multimodal Physiological Monitoring in Neurological Research

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Heart rate (beats per minute (bpm)) was derived from a 3-lead electrocardiogram using disposable adhesive Ag/AgCl electrodes (H207PT, Kendall-LTP, MA, USA) and amplified 1000x (CED 1902, Cambridge Electronic Design, Cambridge, UK). Systolic/diastolic blood pressure (sBP/dBP) was recorded continuously, non-invasively by a Portapres (Finapres Medical Systems, Amsterdam, Netherlands) portable blood pressure monitor. The finger pressure and electrocardiogram were digitised at 4kHz with 16-bit resolution (CED 1401 Mark II, Cambridge Electronic Design, Cambridge, UK) using Spike II software (version 5.0, Cambridge Electronic Design). Local field potentials (LFPs) were simultaneously recorded with bipolar configuration from the adjacent four circumferential 1.5mm contacts of each deep brain macroelectrode. Signals were filtered at 0.5-500Hz and amplified (10000x) using isolated CED 1902 amplifiers and digitised using CED 1401 Mark II at a rate of 2.5kHz (Cambridge Electronic Design). LFPs were displayed online and saved onto hard disk using Spike II and subsequently analysed off-line.
Gillies M.J., Huang Y., Hyam J.A., Aziz T.Z, & Green A.L. (2018). Direct neurophysiological evidence for a role of the human anterior cingulate cortex in central command. Autonomic neuroscience : basic & clinical, 216, 51-58.
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6

Evaluation of Cardiac Autonomic Function

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Cardiovascular autonomic reflex tests were performed as previously described (10 (link)) by following a consensus statement (11 (link)). All patients were euglycemic at the time of autonomic function testing. Those with two or more abnormal cardiovagal tests were regarded as having definite cardiac autonomic neuropathy and were excluded. Spontaneous cardiovagal baroreceptor sensitivity (BRS) was obtained by using a Portapres (Finapres Medical Systems, Amsterdam, the Netherlands). BRS analysis was based on the sequence method and performed with the use of dedicated software (Nevrokard version 5.1.3; Nevrokard Kiauta, Izola, Slovenia) (12 (link)). All participants had a normal 12-lead ECG at baseline.
Chow E., Bernjak A., Walkinshaw E., Lubina-Solomon A., Freeman J., Macdonald I.A., Sheridan P.J, & Heller S.R. (2017). Cardiac Autonomic Regulation and Repolarization During Acute Experimental Hypoglycemia in Type 2 Diabetes. Diabetes, 66(5), 1322-1333.
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7

Comprehensive Cardiovascular Hemodynamics Assessment

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Heart rate (HR) was determined from a lead II electrocardiogram configuration. Beat-by-beat systolic (SBP) and diastolic blood pressure (DBP) were measured via finger photoplethysmography (Portapres®; Finapres Medical Systems, Amsterdam, The Netherlands). The Portapres height correction unit accounted for deviations in the vertical distance between the heart and the pressure cuff. Intermittent brachial SBP and DBP were determined using an automated vital signs monitor (Carescape V100; General Electric Healthcare, Mississauga, ON, Canada) to calibrate the Portapres waveform (Figure 1). The electrocardiogram and Portapres waveforms were sampled at 1000 Hz and 200 Hz, respectively, using a PowerLab data acquisition system (PL3508 PowerLab 8/53; ADInstruments, Sydney, Australia). LabChart software (Version 8; ADInstruments) was used to view recorded signals in real-time and for offline analysis. At least 5 minutes of supine data were averaged to represent resting systemic hemodynamic outcomes. Mean arterial pressure (MAP) was calculated using the equation: ⅓SBP + ⅔DBP.
Shivgulam M.E., O’Brien M.W., Johns J.A., Petterson J.L., Wu Y., Frayne R.J, & Kimmerly D.S. (2022). Impact of habitual sedentary patterns on popliteal artery endothelial-dependent vasodilation in healthy adults. Vascular Medicine (London, England), 27(2), 120-126.
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8

Cardiovascular and Respiratory Measurements

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At each of the three time points, we monitored heart rate as electrocardiographic RR intervals (RRI), systolic and diastolic blood pressure (BPsys, BPdia), as well as respiratory frequency at supine rest. Measurements were performed between 9 a.m. and 2 p.m. after a 40-min resting period in a reclining armchair, in a quiet room with an ambient temperature of 24 °C and stable humidity [3 (link), 17 –19 (link)].
We recorded RRIs via a standard three-lead electrocardiogram, and used finger-pulse photoplethysmography (Portapres®, Finapres Medical Systems BV, Amsterdam, The Netherlands) to continuously measure beat-to beat Bpsys and Bpdia [17 ]. Respiratory frequency was monitored with a piezoelectric belt at the lower thorax (at the point of maximal respiratory excursion) [17 ].
The data were digitized and recorded on a custom-designed data acquisition and analysis system (SUEmpathy, SUESS-Medizin-Technik, Aue, Germany) and stored on a personal computer for off-line analysis [17 ]. From 5-min recordings without artifacts at rest, we extracted the most stationary 120-s epochs, then calculated mean values and standard deviations (SD) of all signals.
Wang R., Köhrmann M., Kollmar R., Koehn J., Schwab S., Kallmünzer B, & Hilz M.J. (2022). Cardiovascular medication seems to promote recovery of autonomic dysfunction after stroke. Journal of Neurology, 269(10), 5454-5465.
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9

Continuous Noninvasive Hemodynamic Monitoring

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Continuous non-invasive arterial blood pressure profiles were recorded at the medium finger of the left arm (Portapres, Finapres® Medical Systems, Enschede, The Netherlands). The left arm was positioned on a rigid support at the heart level. Beat-by-beat HR was recorded by electrocardiography (ECG100C module, BIOPAC® Systems Inc., Goleta, CA, USA). Applied workload and pedalling speed were continuously monitored by analogic outputs from the cycle ergometer which were digitalised (UIM100C module, BIOPAC® Systems Inc., Goleta, CA, USA) and used to provide a precise time alignment of the three repetitions of the two exercise transients. All signals were collected and sampled at 400 Hz (MP150 system with AcqKnowledge acquisition and analysis software, BIOPAC® Systems Inc., Goleta, CA, USA) and stored on a personal computer for subsequent analysis. [La] was measured by an enzymatic-amperometric method (Biosen C-Line Glucose and Lactate analyser, EKF Diagnostics, Cardiff, UK) on 20 μl capillary blood samples.
Taboni A., Fagoni N., Fontolliet T., Vinetti G, & Ferretti G. (2022). Dynamics of cardiovascular and baroreflex readjustments during a light-to-moderate exercise transient in humans. European Journal of Applied Physiology, 122(11), 2343-2354.
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10

Orthostatic Tolerance and Cognitive Testing

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All medicines that could affect orthostatic tolerance were discontinued !5 half-lives. Video EEG (Comet AS-40; GRASS systems, Warwick, RI) was synchronized with continuous heart rate and blood pressure (Portapres; Finapres Medical Systems, Amsterdam, The Netherlands) monitoring at baseline and during HUT testing. After 30 minutes of recumbency, patients were tilted upright (70 ), up to 45 minutes. Patients with syncope were lowered with onset of loss of consciousness. Patients who developed hypotension without syncope were encouraged to remain upright until symptoms became intolerable. They underwent simple cognitive testing during the hypotensive period (e.g., What is the name of this hospital? Where were you born?). Four patients with syncope were given sublingual nitroglycerin (0.3 mg) as part of their clinical protocol to provoke syncope. Medicine provocation was not given to any patient with hypotension only.
Patients were asked to report all symptoms immediately upon symptom onset. Clinical signs were recorded in real time and confirmed by video review. On recovery, several individuals recalled prodromal symptoms that were not reported in real time, so precise timing of symptom onset could not be established. Amnesia of prodromal symptoms did not occur in any patient. EEGs were interpreted by a pediatric neurologist trained in neurophysiology (M.P.I.).
Heyer G.L., Schmittauer C, & Islam M.P. (2016). The Clinical and Electroencephalographic Spectrum of Tilt-Induced Syncope and "Near Syncope" in Youth. Pediatric neurology, 62.
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