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Dl α tocopheryl acetate

Manufactured by Merck Group
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Sourced in United States

DL-α-tocopheryl acetate is a synthetic form of vitamin E. It is a colorless to pale yellow, viscous oily liquid. DL-α-tocopheryl acetate is used as a nutritional supplement and as an antioxidant in various applications.

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Lab products found in correlation

γ tocopherol, by Merck Group (1 mentions) Dl α tocopherol, by Merck Group (1 mentions) δ tocopherol, by Merck Group (1 mentions) Copper chloride, by Merck Group (1 mentions) Trientine hydrochloride, by Merck Group (1 mentions) D penicillamine, by Merck Group (1 mentions) Bathocuproinedisulfonic acid, by Merck Group (1 mentions) Hydrogen peroxide h2o2, by Merck Group (1 mentions) Diethyl ether, by Merck Group (1 mentions) Nitric acid hno3, by Merck Group (1 mentions) L selenomethionine, by Merck Group (1 mentions) Methylmercury, by Merck Group (1 mentions)

Spelling variants of this product

α-tocopheryl acetate (3 citations) Tocopheryl acetate (3 citations)

3 protocols using dl α tocopheryl acetate

1

Tocopherol Compounds Acquisition

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dl-α-tocopherol, dl-α-tocopheryl acetate, (+)-γ-tocopherol and (+)-δ-tocopherol were bought from Sigma Aldrich (Vienna, Austria). Hexanal-d12 (98.5 atom% D) was purchased from C/D/N Isotopes Inc. (Pointe-Claire, QC, Canada) and all other chemicals were ordered from Sigma-Aldrich (Vienna, Austria) or Carl Roth (Karlsruhe, Germany).
Zaunschirm M., Pignitter M., Kienesberger J., Hernler N., Riegger C., Eggersdorfer M, & Somoza V. (2018). Contribution of the Ratio of Tocopherol Homologs to the Oxidative Stability of Commercial Vegetable Oils. Molecules : A Journal of Synthetic Chemistry and Natural Product Chemistry, 23(1), 206.
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2

Modeling Wilson's Disease in Vitro

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To generate Wilson’s disease environment, copper chloride (Sigma-Aldrich) was supplemented to culture medium in various concentrations. D-penicillamine, trientine hydrochloride, bathocuproinedisulfonic acid, and DL-α-tocopheryl acetate (all from Sigma-Aldrich) were additionally treated to investigate recovery effect.
Kim D., Kim S.B., Ryu J.L., Hong H., Chang J.H., Yoo T.J., Jin X., Park H.J., Han C., Lee B.H., Choi J.H., Yoo H.W., Kim J.H, & Woo D.H. (2020). Human Embryonic Stem Cell-Derived Wilson’s Disease Model for Screening Drug Efficacy. Cells, 9(4), 872.
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3

Methylmercury Toxicity Mitigation

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Methylmercury (II) chloride, Cl ~13% (CH3HgCl) as a source of Hg, L-selenomethionine (97% pure), DL-α-tocopheryl acetate as a source of vitamin E were collected from Sigma-Aldrich, St. Louis, USA and L-ascorbyl-2-monophosphate (containing 35% ascorbic acid activity) as a source of vitamin C was purchased from Hoffman La Roche, Basel, Switzerland; diethyl ether, nitric acid (HNO3) and hydrogen peroxide (H2O2) were procured from Merck, Darmstadt, Germany.
Moniruzzaman M., Lee S., Park Y., Min T, & Bai S.C. (2021). Evaluation of dietary selenium, vitamin C and E as the multi-antioxidants on the methylmercury intoxicated mice based on mercury bioaccumulation, antioxidant enzyme activity, lipid peroxidation and mitochondrial oxidative stress. Chemosphere, 273.
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