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Phospho tbc1d1 thr590

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Phospho-TBC1D1 Thr590 is a primary antibody that recognizes TBC1D1 protein phosphorylated at threonine 590. TBC1D1 is a Rab-GTPase-activating protein involved in glucose and lipid metabolism.

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Lab products found in correlation

Total acetyl coa carboxylase acc, by Cell Signaling Technology (2 mentions) Antibodies against phospho as160 thr642, by Merck Group (2 mentions) Phospho acc ser79, by Merck Group (2 mentions) Phospho tbc1d1 ser237, by Merck Group (2 mentions) Hrp conjugated anti sheep igg, by Merck Group (2 mentions) Phospho akt thr308, by Cell Signaling Technology (2 mentions) Total akt, by Cell Signaling Technology (2 mentions) Ecl plus, by GE Healthcare (2 mentions) Ecl prime, by GE Healthcare (2 mentions) Anti glut4antibody, by Bio-Rad (2 mentions) Total as160, by Merck Group (1 mentions) Hrp conjugated anti rabbit igg, by Thermo Fisher Scientific (1 mentions) Antibodies against phospho akt ser473, by Cell Signaling Technology (1 mentions) Enhanced chemi luminescence (ecl), by GE Healthcare (1 mentions) Total erk1 2, by Cell Signaling Technology (1 mentions) Total p38 mapk, by Cell Signaling Technology (1 mentions) Total iκbα, by Cell Signaling Technology (1 mentions) Total jnk, by Cell Signaling Technology (1 mentions) Total tbc1d1, by Cell Signaling Technology (1 mentions) Horseradish peroxidase hrp conjugated anti rabbit igg, by Thermo Fisher Scientific (1 mentions)

Close spelling variants of this product

TBC1D1

2 protocols using phospho tbc1d1 thr590

1

Antibody-based Analysis of Insulin Signaling

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Antibodies against phospho‐Akt Ser473 (#9271), phospho‐Akt
Thr308 (#9275), phospho‐TBC1D1 Thr590 (#6927), total Akt
(#9272), and total acetyl CoA carboxylase (ACC, #3662) were purchased from Cell
Signaling Technology (Beverly, MA). Antibodies against phospho‐AS160 Thr642(#07‐802), phospho‐ACC Ser79 (#07‐303),
phospho‐TBC1D1 Ser237 (#07‐2268), and total AS160
(#07‐741) were from Millipore (Temecula, CA). Anti‐phospho‐AS160
Ser588(#3028P2) was from Symansis Limited (Timaru, New Zealand). Anti‐GLUT4
antibody (#4670‐1704) was from Bio‐Rad AbD Serotec (Oxford, UK).
HRP‐conjugated anti‐rabbit IgG was from Biosource International (Camarillo, CA).
HRP‐conjugated anti‐sheep IgG was from Millipore. Enhanced chemiluminescence reagents
(ECL, ECL Plus, and ECL Prime) were obtained from GE Healthcare Life Sciences (Uppsala, Sweden). All
other reagents were obtained from Sigma (St. Louis, MO).
Iwabe M., Kawamoto E., Koshinaka K, & Kawanaka K. (2014). Increased postexercise insulin sensitivity is accompanied by increased AS160 phosphorylation in slow‐twitch soleus muscle. Physiological Reports, 2(12), e12162.
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2

Signaling Pathway Protein Detection

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Antibodies against phospho‐Akt Ser473 (#9271), phospho‐Akt Thr308 (#9275), phospho‐TBC1D1 Thr590 (#6927), phospho‐JNK Thr183/Tyr185 (#9251), phospho‐p38 MAPK Thr180/Tyr182 (#9216), phospho‐ERK1/2 Thr202/Tyr204 (#9101), total Akt (#9272), total TBC1D1 (#4629S), total‐JNK (#9252), total‐p38 MAPK (#9212), total‐ERK1/2 (#9102), total IκBα (#9242), and total‐acetyl CoA carboxylase (ACC, #3662) were from Cell Signaling Technology (Beverly, MA). Antibodies against phospho‐AS160 Thr642 (#07‐802), phospho‐ACC Ser79 (#07‐303), phospho‐TBC1D1 Ser237 (#07‐2268), and total AS160 (#07‐741) were from Millipore (Temecula, CA). Anti‐phospho‐AS160 Ser588 (#3028P2) was from Symansis Limited (Timaru, New Zealand). Anti‐GLUT4 antibody (#4670‐1704) was from Bio‐Rad AbD Serotec (Oxford, UK). Anti‐SPT2 antibody (ab23696) was from Abcam (Cambridge, MA). Horseradish peroxidase (HRP)‐conjugated anti‐rabbit IgG was from Biosource International (Camarillo, CA). HRP‐conjugated anti‐sheep IgG was from Millipore. HRP‐conjugated anti‐mouse IgG was from Santa Cruz Biotechnology. Enhanced chemiluminescence reagents (ECL, ECL plus, and ECL Prime) were obtained from GE Healthcare Life Sciences (Uppsala, Sweden). All other reagents were obtained from Sigma‐Aldrich (St. Louis, MO).
Kawamoto E., Koshinaka K., Yoshimura T., Masuda H, & Kawanaka K. (2016). Immobilization rapidly induces muscle insulin resistance together with the activation of MAPKs (JNK and p38) and impairment of AS160 phosphorylation. Physiological Reports, 4(15), e12876.
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