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S monovette clotting activator serum

Manufactured by Sarstedt
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Sourced in Germany

The S-Monovette® Clotting Activator/Serum is a laboratory collection system designed for the collection of serum blood samples. It contains a clotting activator to facilitate the clotting process, enabling the separation of serum from the blood sample.

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Lab products found in correlation

S monovette k3 edta, by Sarstedt (2 mentions)

Close spelling variants of this product

Clotting Activator/Serum S-Monovette® Serum Clotting activator Serum monovettes S-Monovette Monovettes

2 protocols using s monovette clotting activator serum

1

Venous Blood Collection and Preservation

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Following an overnight fast, venous blood (5 mL) was collected into tubes containing ethylenediaminetetraacetic acid (S-Monovette® EDTA-K3, Sarstedt, Germany) and into tubes with clot activator (S-Monovette® Clotting Activator/Serum, Sarstedt, Germany). In accordance with the manufacturer’s instructions, blood samples were protected from light, centrifuged (2000× g for 10 min at +4 °C), and then plasma and serum were immediately separated. Blood was frozen at a temperature of −80 °C until assayed, for no longer than 6 months.
Heropolitanska-Pliszka E., Berk K., Maciejczyk M., Sawicka-Powierza J., Bernatowska E., Wolska-Kusnierz B., Pac M., Dabrowska-Leonik N., Piatosa B., Lewandowicz-Uszynska A., Karpinska J., Zalewska A, & Mikoluc B. (2020). Systemic Redox Imbalance in Patients with Chronic Granulomatous Disease. Journal of Clinical Medicine, 9(5), 1397.
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2

Plasma and Serum Sample Collection

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Following an overnight fast, venous blood (2 × 10 mL) was collected into tubes containing ethylenediaminetetraacetic acid (S-Monovette® EDTA-K3, Sarstedt, Germany) and into tubes with a clot activator (S-Monovette® Clotting Activator/Serum, Sarstedt, Germany). According to the manufacturer's instructions, blood samples were protected from light, centrifuged (2000 × g for 10 min at +4°C) and then plasma and serum were immediately separated. Blood was frozen at the temperature of −80°C until assayed.
Maciejczyk M., Heropolitanska-Pliszka E., Pietrucha B., Sawicka-Powierza J., Bernatowska E., Wolska-Kusnierz B., Pac M., Car H., Zalewska A, & Mikoluc B. (2019). Antioxidant Defense, Redox Homeostasis, and Oxidative Damage in Children With Ataxia Telangiectasia and Nijmegen Breakage Syndrome. Frontiers in Immunology, 10, 2322.
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