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Ssp 25 cells

Manufactured by RIKEN BioResource Center
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Sourced in Japan

The SSP-25 cells are a type of cell line developed and maintained at the RIKEN BioResource Center. They are derived from mouse embryonic stem cells and are designed for use in various research applications. The core function of the SSP-25 cells is to serve as a model system for the study of embryonic development, stem cell biology, and other related areas of research.

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Lab products found in correlation

Huh 28 cells, by RIKEN BioResource Center (2 mentions) Rpmi 1640, by Thermo Fisher Scientific (1 mentions) Fetal bovine serum (fbs), by Thermo Fisher Scientific (1 mentions) Lovastatin, by Merck Group (1 mentions) Gefitinib, by Selleck Chemicals (1 mentions) P s solution, by Thermo Fisher Scientific (1 mentions)

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SSP-25 (5 citations)

2 protocols using ssp 25 cells

1

Culturing Cholangiocarcinoma Cell Lines

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Human cholangiocarcinoma SSP‐25 cells and HuH‐28 cells were purchased from RIKEN Bioresource Center. SSP‐25 cells were grown in RPMI‐1640 medium supplemented with 10% FBS, and HuH‐28 cells were grown in Eagle's minimum essential medium supplemented with 10% FBS. The cells were maintained at 37°C in a humidified atmosphere containing 5% CO2.
Miyata T., Yamashita Y., Yoshizumi T., Shiraishi M., Ohta M., Eguchi S., Aishima S., Fujioka H, & Baba H. (2019). CXCL12 expression in intrahepatic cholangiocarcinoma is associated with metastasis and poor prognosis. Cancer Science, 110(10), 3197-3203.
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2

Cell Lines and Reagents for Cholangiocarcinoma

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The human cholangiocarcinoma SSP-25 cells and HuH-28 cells were purchased from RIKEN Bioresource Center (Ibaraki, Japan) and preserved for the study in RPMI-1640 or minimum essential medium content with 10% FBS and P/S solution (Invitrogen, Carlsbad, CA) in 5% CO2 incubator at 37°C. Gefitinib was purchased from Selleckchem, and lovastatin was purchased from Sigma.
Yang S.H., Lin H.Y., Chang V.H., Chen C.C., Liu Y.R., Wang J., Zhang K., Jiang X, & Yen Y. (2015). Lovastatin overcomes gefitinib resistance through TNF-α signaling in human cholangiocarcinomas with different LKB1 statuses in vitro and in vivo. Oncotarget, 6(27), 23857-23873.
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