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Cd19 hib19 and igm mhm 88

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CD19 (HIB19) is a monoclonal antibody that specifically binds to the CD19 antigen. IgM (MHM-88) is a monoclonal antibody that specifically binds to the IgM antibody. These products are intended for use in research applications.

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Lab products found in correlation

Pd 1 j43, by Thermo Fisher Scientific (1 mentions) Pna fl 1071, by Vector Laboratories (1 mentions) Fas jo2, by BD (1 mentions) Cd44 im7, by BioLegend (1 mentions) Cd27 o323, by Thermo Fisher Scientific (1 mentions) Cd38 hit2, by BD (1 mentions) Igd 11 26c 2a, by BD (1 mentions)

Spelling variants of this product

CD19 (HIB19; (23 citations) MHM-88 (7 citations) IgM (MHM-88, (6 citations)

3 protocols using cd19 hib19 and igm mhm 88

1

Comprehensive Antibody Panel for Immunophenotyping

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Anti–mouse antibodies used in this study included: B220 (RA3-6B2), CD19 (1D3), CD4 (RM4-5), CXCR5 (2G8), and Fas (Jo2) from BD; CD62L (MEL-14), CD11c (N418), Gr-1 (RB6-8C5), Ly5.1 (A20), Ly5.2 (104), CD11b (M1/70), GL7 (GL-7), and PD-1 (J43), from eBioscience; goat anti–mouse IgM−, IgG−, IgG2c− horseradish peroxidase–conjugated, unlabeled, or isotype from SouthernBiotech; CD19 (6D5), CD24 (M1/69), CD23 (B3B4), CD21 (7E9), CD44 (IM7), CD4 (RM4-4), Ly5.2 (104) from BioLegend; PNA (Fl-1071) from Vector Laboratories. Anti–human antibodies used in this study include CD38 (HIT2) and CD27 (L128) from BD; CD19 (HIB19) and IgM (MHM-88) from BioLegend; and IL-6 (MQ2-12A5) from eBioscience.
Arkatkar T., Du S.W., Jacobs H.M., Dam E.M., Hou B., Buckner J.H., Rawlings D.J, & Jackson S.W. (2017). B cell–derived IL-6 initiates spontaneous germinal center formation during systemic autoimmunity. The Journal of Experimental Medicine, 214(11), 3207-3217.
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2

Murine and Human B-cell Antibody Analysis

Cited 1 time
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Anti-murine Abs used in these studies include: B220 (RA3-6B2), CD24 (M1/69), CD21 (7E9), CD23 (B3B4), CD4 (RM4-4) and CD44 (IM7) from Biolegend; IgMa (DS-1) and CD40 (3/23) from BD; Ly 5.1 (A20), Ly 5.2 (104), Gr1 (RB6-8C5), CD11b (M1/70), CD3 (17A2), BAFFR (eBio7H22-E16), CD93 (AA4.1), CD62L (MEL-14) and CD40L (MR1) from eBiosciences; IgM (1B4B1) and IgD (11 (link)-26 (link)) from Southern Biotech; recombinant human insulin conjugated to biotin from Fitzgerald Industries; Streptavadin (S-868) from Life Technologies. Anti-human Abs used include: CD19 (HIB19) and IgM (MHM-88) from Biolegend; CD27 (O323) from eBiosciences; CD10 (HI10a), CD24 (ML5), CD38 (HIT2), IgD (IA6-2), and BAFFR (IIC1) from BD; anti–human FITC-conjugated 9G4 antibody (18 (link)).
Metzler G., Dai X., Thouvenel C.D., Khim S., Habib T., Buckner J.H, & Rawlings D.J. (2017). The autoimmune risk variant PTPN22 C1858T alters B cell tolerance at discrete checkpoints and differentially shapes the naïve repertoire. Journal of immunology (Baltimore, Md. : 1950), 199(7), 2249-2260.
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3

Antibodies for Murine and Human Immune Cell Analysis

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Anti–murine antibodies used in this study include AA4.1, CD24 (M1/69), CD21 (7G6), B220 (RA3-6B2), and IgD (11-26C.2A) from BD; BP1 (FG35.4) and CD23 (B3B4) from Invitrogen; IgM (1B4B1), κ (187.1), λ (JC5-1), and SA-HRP conjugated from SouthernBiotech; CD19 (ID3) and IgMa (DS-1) from BioLegend; and Cy5 anti–rabbit polyclonal IgG from Jackson ImmunoResearch Laboratories, Inc. Alexa Fluor 647 anti-M167 (28–6-20) rat IgG2a was provided by S. Porcelli (Albert Einstein College of Medicine, Bronx, NY). Anti–human antibodies used in this study include CD19 (HIB19) and IgM (MHM-88) from BioLegend; CD10 (HI10a), CD24 (ML5), and IgD (IAG-2) from BD; CD27 (323) from eBioscience; and CD38 (HIT2) from BD. Anti–human rat monoclonal, FITC-conjugated 9G4 antibody was provided by I. Sanz.
Kolhatkar N.S., Brahmandam A., Thouvenel C.D., Becker-Herman S., Jacobs H.M., Schwartz M.A., Allenspach E.J., Khim S., Panigrahi A.K., Luning Prak E.T., Thrasher A.J., Notarangelo L.D., Candotti F., Torgerson T.R., Sanz I, & Rawlings D.J. (2015). Altered BCR and TLR signals promote enhanced positive selection of autoreactive transitional B cells in Wiskott-Aldrich syndrome. The Journal of Experimental Medicine, 212(10), 1663-1677.
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