Scanning was conducted on a GE TwinSpeed 1.5T scanner equipped with an 8-channel head coil. Functional spiral echo in/out pulse sequence scanning parameters consisted of: TR = 2000ms, slice thickness = 4.5mm, gap = 0 mm, flip angle = 84 degrees, FOV = 22.4 mm, matrix size = 64×64, slices = 23, axial orientation, with a resulting voxel size of [3.5, 3.5, 4.5] mm, using interleaved bottom-to-top acquisition. Structural spoiled gradient (SPGR) scan parameters consisted of: TR = 19s, TE = 5ms, slice thickness = 1mm, gap = 0, flip = 20, FOV = 25.6, sagittal orientation, matrix size=256×256×180, voxel size = [1, 1, 1]. Stimuli were projected onto a screen and deflected by a mirror for viewing, and responses were made with a scanner compatible track ball with two buttons. Stimulus presentation and behavioral data collection were administered with a desktop PC and Matlab software with the Psychophysics toolbox (Pelli, 1997 (link)).
Twinspeed 1.5t scanner
Manufactured by GE Healthcare
The TwinSpeed 1.5T scanner is a magnetic resonance imaging (MRI) system manufactured by GE Healthcare. It operates at a magnetic field strength of 1.5 Tesla, which is a common field strength used in clinical MRI applications. The TwinSpeed 1.5T scanner is designed to acquire high-quality imaging data for diagnostic purposes.
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Lab products found in correlation
2 protocols using twinspeed 1.5t scanner
1
Functional and Structural Brain Imaging Protocol
2
Demyelinating Lesion Assessment via MRI
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MRI scans were performed on a GE Twinspeed 1.5 T scanner. The sequences included 6-mm thickness axial T1WI (TR = 1980-2280ms, TE = 23-27ms, Matrix = 384×224), T2WI (TR = 3700-4800ms, TE = 99-106ms, Matrix = 384×224), 5-mm thickness sagittal T2 FLAIR (TR = 8267-8269ms, TE = 127-128ms, Matrix = 320×192), 3-mm thickness coronal T2 STIR (TR = 6000-6200ms, TE = 45ms, Matrix = 320×192), contrast-enhanced imaging via gadolinium (Gd) infusion (0.1 mmol/kg), and 8HRBRAIN. Both an experienced radiologist and a neurologist reviewed all MRI archives using a DICOM viewer program (eFilm Workstation™ 1.8.3). The lesion numbers, locations and sizes were recorded. A third reader (either a radiologist or a neurologist) made the final decision if the first two readers did not reach a consensus. We considered high white matter signals on the axial T2WI and sagittal T2FLAIR sequences to represent demyelinating lesions. The lesion size was not limited.
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