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Trio 3.0 t system

Manufactured by Siemens
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The Trio 3.0 T system is a magnetic resonance imaging (MRI) device manufactured by Siemens. It is a 3.0 Tesla (T) MRI system designed for clinical and research applications. The Trio 3.0 T system provides high-field MRI imaging capabilities for various medical and scientific purposes.

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Lab products found in correlation

Biograph 64 pet ct system, by Siemens (1 mentions) Eight channel head coil, by Siemens (1 mentions)

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3.0-T Trio 3.0-T system Trio system TrioTM

5 protocols using trio 3.0 t system

1

Cerebral Infarction Measurement in Cynomolgus Monkeys

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All cynomolgus monkeys underwent cranial MRI scans with Siemens’ 3.0 T Trio system (Siemens, Germany) before and 2 weeks after the surgery to clarify the site and severity of cerebral infarction. T1-weighted three-dimensional magnetisation prepared rapid acquisition gradient echo (T1W1-3D-MPRAGE) sequences and T2-weighted sequences were scanned to assess brain parenchyma, and the specific scan parameters are detailed in our previous study.18 (link) Brain infarct volume was measured on T1W1-3D-MPRAGE images of each monkey 2 weeks after MCAO. In summary, infarct areas and the ipsilateral hemisphere (region of interest) were manually mapped and then measured using ITK-SNAP.21 (link) The infarct volume ratio was computed by the formula:(infarct volume/ipsilateral hemisphere volume) ×100% (online supplemental material 1).
Li P., Chen C., Huang B., Jiang Z., Wei J, & Zeng J. (2022). Altered excitability of motor neuron pathways after stroke: more than upper motor neuron impairments. Stroke and Vascular Neurology, 7(6), 518-526.
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2

Blinded Radiological Analysis of Brain MRI

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Brain MRI was performed using a 3.0-T Trio system (Siemens, Erlangen, Germany) and T2-weighted, T1-weighted, and fluid-attenuated inversion recovery (FLAIR) sequences. Working independently, two radiologists who were blinded to clinical data retrospectively analyzed images in order to determine whether findings were normal or abnormal. Abnormal findings were defined as T2/FLAIR signal hyperintensity in the cerebellar cortex, basal ganglia, thalamus, subcortical areas, and/or cerebral cortex, or leptomeningeal contrast enhancement (Figure 1) (6 (link)). A third neurologist was consulted in the event of disagreement.
Lei C., Chang X., Li H, & Zhong L. (2022). Abnormal Brain MRI Findings in Anti-N-Methyl-D-Aspartate Receptor Encephalitis and Correlation With Outcomes. Frontiers in Neurology, 13, 834929.
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3

Multimodal Brain Imaging Protocol

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MR images of the entire brain were acquired using GE-EPI (with an acceleration factor of 2) on a Siemens Trio 3.0 T system with the following parameters: time of repetition (TR)/time of echo (TE)/flip angle (FA) = 2000 ms/30 ms/90°, FOV= 220 mm × 220 mm, acquisition matrix= 80 × 80, 30 slices, slice thickness= 4 mm, and the number of repetitions= 234. T1-MPRAGE anatomical images were also acquired for anatomical underlay with the following parameters: slices= 160, slice thickness= 1mm, FOV= 256 mm × 256 mm × 160 mm, matrix size= 256 × 256 × 160, TE= 30 ms, TR= 2300 ms, and acquisition time= 321 s.
Karunanayaka P.R., Wilson D.A., Tobia M.J., Martinez B.E., Meadowcroft M.D., Eslinger P.J, & Yang Q.X. (2016). Default mode network deactivation during odor–visual association. Human Brain Mapping, 38(3), 1125-1139.
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4

Multimodal MRI and PET/CT Imaging Protocol

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The MRI was performed on a Siemens Trio 3.0T system (Siemens, Erlangen, Germany). The axial T2WI, coronal T2WI, sagittal T2WI, T1WI, and fat suppression T2WI images were acquired using the fast spin echo sequence. After the abnormal region was found, DCE-MRI was acquired using a FLASH 3D VIBE sequence, with TR=4.1ms, TE=1.5 ms, flip angle=10°, matrix=256×192, FOV (field of view) =250mm×250 mm, slice thickness=3mm. A total of 16 DCE time frames were taken covering the DCE time period of 120~168 s. The contrast agent, gadopentetate dimeglumine (Gd-DTPA), at a dose of 0.1 mmol/kg was injected after one pre-contrast frame was taken, with the speed of 2 ml/s.
PET/CT was performed using a Siemens Biograph 64 PET/CT system (Siemens, Erlangen, Germany) with 52-rings PET, and 64-row spiral CT. All patients were fasted before the examination, and blood glucose levels were in the range of 4.4–9.3 mmol/L. All patients underwent whole-body PET/CT conventional imaging 60 minutes after injection of 3.7–4.5 MBq/kg 18F-FDG. A 64-row spiral CT scan was performed with voltage of 140 kV, current of 100 mA, and a slice thickness of 3 mm. PET imaging was done using the 3D mode acquisition with iterative construction. The scan time was 8 minutes for the head scan, and 3 minutes per bed for the body scan from calvaria to the upper part of the femur.
Zhang J., Chen Y., Zhang Y., Zhang E., Yu H.J., Yuan H., Zhang Y., Su M.Y, & Lang N. (2019). Diagnosis of Spinal Lesions Using Perfusion Parameters Measured by DCE-MRI and Metabolism Parameters Measured by PET/CT. European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society, 29(5), 1061-1070.
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5

Functional Neuroimaging of Olfactory-Visual Processing

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MR images of the entire brain were acquired using GE EPI on a Siemens Trio 3.0 T system with an eight channel head coil. Functional MRI utilizing the blood oxygen level dependent (BOLD) signal was used to evaluate brain functional activities during odor+visual and visual‐only conditions. A T2∗ ‐weighted echo planar imaging sequence was used to acquire functional data with the following parameters: TR/TE/FA = 2,000 ms/30 ms/90°, FOV = 220 mm × 220 mm, acquisition matrix = 80 × 80, 30 slices, slice thickness = 4 mm, and the number of repetitions = 234. For volumetric analysis of the POC and hippocampus, T1‐weighted images with 1 mm isotropic resolution were acquired with MPRAGE method: TE = 2.98 ms, TR = 2300 ms, inversion time (IT) = 900 ms, FA = 9°, FOV = 256 mm × 256 mm × 160 mm, acquisition matrix = 256 × 256 × 160, acceleration factor = 2, and TA = 6 min 21 s.
Lu J., Yang Q.X., Zhang H., Eslinger P.J., Zhang X., Wu S., Zhang B., Zhu B, & Karunanayaka P.R. (2019). Disruptions of the olfactory and default mode networks in Alzheimer's disease. Brain and Behavior, 9(7), e01296.
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