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Sihdac1

Manufactured by GenePharma
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Sourced in China

SiHDAC1 is a laboratory equipment product manufactured by GenePharma. It is a tool used for the study of gene expression and regulation. The core function of SiHDAC1 is to facilitate the silencing or downregulation of target genes through the use of small interfering RNA (siRNA) technology.

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Lab products found in correlation

Sip300, by GenePharma (1 mentions) Si p53, by GenePharma (1 mentions) Mir 135b 5p, by GenePharma (1 mentions) Lipofectamine 2000 reagent, by Thermo Fisher Scientific (1 mentions) Si nc, by GenePharma (1 mentions) Mir nc, by GenePharma (1 mentions) Anti mir nc, by GenePharma (1 mentions)

2 protocols using sihdac1

1

siRNA Targeting Regulators of p53

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The siRNAs targeting Homo sapiens lnc‐Ip53 (GeneBank accession No. NR_037177.1), p53 (NM_000546.5), p300 (NM_001429.3), CBP (NM_004380.2), MOZ (NM_006766.4), and HDAC1 (NM_004964.2) transcripts are designated as silnc‐Ip53, sip53, sip300, siCBP, siMOZ, and siHDAC1, respectively, and were purchased from GenePharma (Shanghai, China). The negative control (NC) RNA duplex for siRNA is nonhomologous to any human genome sequences. The sequences of oligos are listed in Table S2, Supporting Information.
Zhang L., Yang J., Luo Y., Liu F., Yuan Y, & Zhuang S. (2020). A p53/lnc‐Ip53 Negative Feedback Loop Regulates Tumor Growth and Chemoresistance. Advanced Science, 7(21), 2001364.
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2

Modulating circRSF1 and miR-135b-5p in ox-LDL HUVECs

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CircRSF1 overexpression (circRSF1), HDAC1 inhibitor (si-HDAC1), miR-135b-5p mimics (miR-135b-5p), miR-135b-5p inhibitor (anti-miR-135-5p) and their negative control (NC; vector, si-NC, miR-NC and anti-miR-NC) were purchased from GenePharma (Shanghai, China). The vectors and oligonucleotides (50 nM) were transfected into ox-LDL induced HUVECs using the Lipofectamine 2000 reagent (Invitrogen, Carlsbad, CA, USA). All transfections experiments were performed in triplicates.
Zhang X., Lu J., Zhang Q., Luo Q, & Liu B. (2021). CircRNA RSF1 regulated ox-LDL induced vascular endothelial cells proliferation, apoptosis and inflammation through modulating miR-135b-5p/HDAC1 axis in atherosclerosis. Biological Research, 54, 11.
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