Antibodies to ve cadherin

Manufactured by Abcam

Abcam's antibodies to VE-cadherin are specific for the vascular endothelial cadherin protein. VE-cadherin is a cell-cell adhesion molecule that plays a crucial role in the regulation of endothelial cell junctions and vascular permeability.

Automatically generated - may contain errors

Lab products found in correlation

Diethylaminoethyl deae dextran, by Merck Group (2 mentions) Lipopolysaccharide lps from e coli, by Merck Group (1 mentions) Rela p65, by Santa Cruz Biotechnology (1 mentions) Human alpha thrombin, by Enzyme Research (1 mentions) 4 phenylbutyrate 4 pba, by Merck Group (1 mentions) β actin, by Santa Cruz Biotechnology (1 mentions) Icam 1, by Santa Cruz Biotechnology (1 mentions) Plasmid maxi kit, by Qiagen (1 mentions) Anti gapdh antibody sc 32233, by Santa Cruz Biotechnology (1 mentions) Human α thrombin, by Enzyme Research (1 mentions) β actin sc 47778, by Santa Cruz Biotechnology (1 mentions) Anti phospho ser3 cofilin1 antibody 3311s, by Cell Signaling Technology (1 mentions)

Close spelling variants of this product

VE-cadherin (100 citations)

2 protocols using antibodies to ve cadherin

Modulation of Endothelial Cell Stress Pathways

Cited 1 time

Check if the same lab product or an alternative is used in the 5 most similar protocols

Human alpha thrombin was obtained from Enzyme Research Laboratories (South Bend, IN). Lipopolysaccharide (LPS) from E. coli, diethylaminoethyl (DEAE)-dextran, and 4-phenylbutyrate (4-PBA) were purchased from Sigma-Aldrich Chemical (St. Louis, MO). Polyclonal antibodies to VCAM-1, ICAM-1, IκBα, RelA/p65, and β-actin were from Santa Cruz Biotechnology (CA). Antibodies to VE-cadherin were purchased from Abcam (Cambridge, MA) and BD Biosciences (San Jose, CA). BiP/GRP78 polyclonal antibody was obtained from Cell Signaling Technology (Beverly, MA). Expression vector encoding Wild type BiP/GRP78 and dominant negative BiP/GRP78 were from Addgene. SubAB, SubA and the non-active derivative SubA_A272B were purified as previously described^{28 (link),29 (link),46 (link)}. All other materials were from VWR Scientific Products Corporation (Gaithersburg, MD) and Fisher Scientific (Pittsburgh, PA).

Leonard A., Grose V., Paton A.W., Paton J.C., Yule D.I., Rahman A, & Fazal F. (2019). Selective Inactivation of Intracellular BiP/GRP78 Attenuates Endothelial Inflammation and Permeability in Acute Lung Injury. Scientific Reports, 9, 2096.

+ Open protocol

+ Expand

Antibody Characterization for Cell Signaling

Check if the same lab product or an alternative is used in the 5 most similar protocols

Human α-thrombin was purchased from Enzyme Research Laboratories (South Bend, IN). Diethylaminoethyl (DEAE)-dextran was obtained from Sigma-Aldrich Chemical Company (St. Louis, MO). A rabbit polyclonal anti-Beclin1 antibody (3738S) was purchased from Cell Signaling Technology (Beverly, MA). Polyclonal antibodies to VCAM-1 (SC-8304), RelA/p65 (SC-8008), IκBα (SC-371), and β-actin (SC-47778) were from Santa Cruz Biotechnology (Santa Cruz, CA). An anti-phospho-(Ser⁵³⁶)-RelA/p65 (3033S) was from Cell Signaling Technology (Beverly, MA). Antibodies to VE-cadherin were obtained from Abcam (AB33168, Cambridge, MA) and BD Biosciences (BD555661, San Jose, CA). A rabbit polyclonal anti-Cofilin1 (clone D3F9, 5175S) antibody and a rabbit polyclonal anti-phospho-(Ser³)-Cofilin1 antibody (3311S) were obtained from Cell Signaling Technology (Beverly, MA). An anti-GAPDH antibody (SC-32233) and an anti-Lamin B antibody (SC-6216) were obtained from Santa Cruz Biotechnology (Santa Cruz, CA). Plasmid maxi kit was from QIAGEN Inc. (Valencia, CA). All other materials were purchased from Thermo Fisher Scientific (Waltham, MA).

Leonard A., Millar M.W., Slavin S.A., Bijli K.M., Santos D.A., Dean D.A., Fazal F, & Rahman A. (2019). Critical Role of Autophagy Regulator Beclin1 in Endothelial Cell Inflammation and Barrier Disruption. Cellular signalling, 61, 120-129.

+ Open protocol

+ Expand

About PubCompare

Our mission is to provide scientists with the largest repository of trustworthy protocols and intelligent analytical tools, thereby offering them extensive information to design robust protocols aimed at minimizing the risk of failures.

We believe that the most crucial aspect is to grant scientists access to a wide range of reliable sources and new useful tools that surpass human capabilities.

However, we trust in allowing scientists to determine how to construct their own protocols based on this information, as they are the experts in their field.

Ready to get started?

Revolutionizing how scientists
search and build protocols!