Finometer

Manufactured by Finapres Medical Systems

Sourced in Netherlands, United States

The Finometer is a non-invasive cardiovascular monitoring device produced by Finapres Medical Systems. It continuously measures beat-to-beat blood pressure and related cardiovascular parameters using the volume-clamp method.

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Lab products found in correlation

82 protocols using finometer

Hemodynamic and Skin Blood Flow Monitoring

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We measured the hemodynamic parameters using Finometer ® (Finapres Medical Systems, FMS, Arnhem, Netherlands), including heart rate (HR), SBP, diastolic blood pressure (DBP), stroke volume (SV), cardiac index (CI), and total peripheral vascular resistance (TPR).[ 21 ] One-minute average of the hemodynamic parameters was collected from the continuous recording of Finometer ® . Skin blood flow was measured in the other hand by a laser Doppler flowmeter (DRT4 Instrument, Moor Instruments, Devon, UK).

Chu Y.H., Tai Y.H., Yeh C.C., Tsou M.Y., Lee H.S., Ho S.T., Li M.H., Lin T.C, & Lu C.C. (2020). Glucose reduces the osmopressor response in connection with the tyrosine phosphorylation of focal adhesion kinase in red blood cells. The Chinese journal of physiology, 63(3).

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Measuring Cardiovascular Responses in Supine

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Heart rate (HR) was monitored with a 3‐lead ECG. Mean arterial pressure (MAP) was measured by placing a finger pressure cuff around the middle phalanx of the middle finger on the nonexperimental arm, which remained at heart level (supine position) throughout the measurement (Finometer; Finapres Medical Systems BV, Amsterdam, The Netherlands). Resting arterial blood pressure was measured over the brachial artery following 30 min of supine rest and just prior to each exercise trial (Cardiocap 5; Datex Ohmeda, Louisville, CO), and resting Finometer MAP was corrected for differences between the two readings (Kirby et al. 2005).

Richards J.C., Racine M.L., Hearon CM J.r., Kunkel M., Luckasen G.J., Larson D.G., Allen J.D, & Dinenno F.A. (2018). Acute ingestion of dietary nitrate increases muscle blood flow via local vasodilation during handgrip exercise in young adults. Physiological Reports, 6(2), e13572.

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Multimodal Cardiovascular Monitoring Protocol

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Instrumentation for each experiment was identical. Heart rate was continuously obtained from an electrocardiogram (HP Patient Monitor, Agilent, Santa Clara, CA, USA) interfaced with a cardiotachometer (CWE, Ardmore, PA, USA). Continuous beat-by-beat MAP was recorded from a finger using the Penaz method [Finometer; Finapres Medical Systems, Amsterdam, The Netherlands (experiment 1) and the CNAP, Biopac Monitor 500, Bruck an der Mur Austria (experiment 2)] and was corrected according to intermittent blood pressure measurements obtained by auscultation of the brachial artery via electrosphygmomanometry (SunTech, Raleigh, NC, USA). Blood flow in the middle cerebral artery (CBFV) was continuously measured using transcranial Doppler ultrasonography. A 2 MHz Doppler probe (Multi-flow; DWL Elektronische Systeme, Singen, Germany) was adjusted over the temporal window of the right middle cerebral artery until an optimal signal was identified. The probe was then fixed and held in place using a headband strap to prevent subtle movement of the Doppler probe. An index of cerebrovascular conductance (CVCI) was calculated from the ratio of CBFV to MAP. The was measured continuously using a capnograph (VitalCap Capnograph Monitor; Oridion, Needham, MA, USA).

Brothers R.M., Lucas R.A., Zhu Y.S., Crandall C.G, & Zhang R. (2014). Cerebral vasomotor reactivity: steady-state versus transient changes in carbon dioxide tension. Experimental Physiology, 99(11), 1499-1510.

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Multimodal Cardiovascular and Cerebrovascular Monitoring

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Instrumentation for each aim was identical. Heart rate was continuously obtained from an electrocardiogram (HP Patient Monitor, Agilent, Santa Clara, CA) interfaced with a cardiotachometer (CWE, Ardmore, PA). Continuous beat-by-beat mean arterial blood pressure (MAP) was recorded from a finger using the Penaz method (Finometer, Finapres Medical Systems, Amsterdam, Netherlands (aim #1) and the CNAP, Biopac Monitor 500, Austria (aim #2)) and was corrected according to intermittent blood pressure measurements obtained by auscultation of the brachial artery via electrosphygmomanometry (SunTech, Raleigh, NC). Blood flow in the middle cerebral artery (CBFV) was continuously measured using transcranial Doppler ultrasonography. A 2-MHz Doppler probe (Multi-flow, DWL Elektronische Systeme, Singen, Germany) was adjusted over the temporal window of the right middle cerebral artery until an optimal signal was identified. The probe was then fixed and held in place using a headband strap to prevent subtle movement of the Doppler probe. An index of cerebrovascular conductance (CVCI) was calculated from the ratio of CBFV to MAP. PETCO₂ was continuously measured using a capnograph (VitalCap Capnograph Monitor, Oridion, Needham, MA).

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Sympathetic Nerve Activity and Cardiovascular Monitoring

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Continuous measurements of muscle sympathetic nerve activity (MSNA), blood pressure, and heart rate were made throughout the protocol. Sympathetic nerve recordings were obtained in the right peroneal nerve by microneurography (Hagbarth and Vallbo, 1968 (link)), using methods from our laboratory described previously in detail (Kimmerly and Shoemaker, 2003 (link); Kimmerly et al., 2004 (link); Badrov et al., 2015 (link); Usselman et al., 2015 (link)). Beat-to-beat blood pressure was measured using finger photoplethysmography (Finometer; Finapres Medical Systems, Amsterdam, The Netherlands). Blood pressure values obtained from the Finometer were calibrated to the average of three baseline blood pressure measurements assessed using manual sphygmomanometry. Heart rate was measured throughout using a standard three-lead electrocardiogram. All data were collected and analyzed offline using PowerLab/16SP with LabChart 6 (ADInstruments, Colorado Springs, Colorado, USA). Typical measurements from one subject are shown in Figure 1.

Zamir M., Badrov M.B., Olver T.D, & Shoemaker J.K. (2017). Cardiac Baroreflex Variability and Resetting during Sustained Mild Effort. Frontiers in Physiology, 8, 246.

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Renal Nerve Ablation Cardiovascular Assessment

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The study protocol at 12 months after renal nerve ablation was exactly the same as 6 months after renal nerve ablation.[5 (link)] During testing, patients remained in the supine position. An ECG was continuously recorded (Niccomo, Medis GmbH, Germany). Noninvasive finger blood pressure recording was used (Finometer, Finapres Medical Systems, NL) and adjusted against brachial oscillometric blood pressure measurements (Dinamap, Critikon, USA).

Tank J., Heusser K., Brinkmann J., Schmidt B.M., Menne J., Bauersachs J., Haller H., Diedrich A, & Jordan J. (2015). Spike rate of multi-unit muscle sympathetic nerve fibers following catheter-based renal nerve ablation. Journal of the American Society of Hypertension : JASH, 9(10), 794-801.

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Noninvasive Hemodynamic Monitoring

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In both studies, mean arterial pressure (MAP) was measured noninvasively using photoelectric plethysmography with a Finometer (Finapres Medical Systems BV, Arnhem, Netherlands), which was calibrated with upper cuff and height adjustment. Furthermore, HR and stroke volume, hence cardiac output (CO), were determined from the blood pressure waveform using the Modelflow software program (BeatScope 1.1, Finapres Medical Systems BV, Amhem, Netherlands), which takes into account the participant's sex, age, height, and weight.

Oue A, & Sadamoto T. (2018). Compliance in the deep and superficial conduit veins of the nonexercising arm is unaffected by short‐term exercise. Physiological Reports, 6(11), e13724.

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Comprehensive Physiological Monitoring Protocol

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HR was monitored via a three‐lead electrocardiogram. Arterial blood pressure was measured non‐invasively using finger photoplethysmography (Finometer, Finapres Medical Systems). The monitoring cuff was placed around the middle finger of the right hand, with the forearm and hand supported so that the cuff was at the vertical level of the heart. Core temperature (T_c) was assessed using a commercially available rectal probe (RET‐1, Physitemp Instruments) inserted 15 cm past the sphincter muscle and connected to a thermocouple meter (TC‐2000, Sable Systems). Mean skin temperature (T_sk) from four sites (standard weightings of chest, arm, thigh, and calf, (Ramanathan, 1964)) was obtained using a wireless monitoring system (iButton^®, Maxim Integrated). Analog signals of the electrocardiogram, blood pressure waveform, and T_c were sampled at 1,000 Hz using a data acquisition unit (Powerlab 16/30, ADInstruments) and analyzed using an off‐line data analysis software (LabChart 8, ADInstruments).

Watanabe K., Stöhr E.J., Akiyama K., Watanabe S, & González‐Alonso J. (2020). Dehydration reduces stroke volume and cardiac output during exercise because of impaired cardiac filling and venous return, not left ventricular function. Physiological Reports, 8(11), e14433.

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Continuous Arterial Pressure Monitoring

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Beat-to-beat systolic (SAP), diastolic (DAP), and mean (MAP) arterial pressures were measured continuously using a volume-clamp technique (Finometer, Finapres Medical Systems BV, Amsterdam, The Netherlands), with the pressure cuff placed around the middle phalanx of the left middle finger, and with the reference pressure transducer positioned at the level of the heart. The Finometer-derived values were verified intermittently by electro-sphygmomanometry (Omron, M6, Kyoto, Japan). HR was derived from the arterial pressure curves as the inverse of the interbeat interval.

Keramidas M.E., Kölegård R., Gäng P., Wilkins F., Elia A, & Eiken O. (2022). Acral skin vasoreactivity and thermosensitivity to hand cooling following 5 days of intermittent whole body cold exposure. American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, 323(1), R1-R15.

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Hemodynamic and Muscle Activity Monitoring

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An electrocardiogram (ECG) was monitored with a telemetry system (DynaScope DS‐3140; Fukuda Denshi, Tokyo, Japan). Arterial blood pressure (AP) was noninvasively and continuously measured with a Finometer^® (Finapres Medical Systems BV, Arnhem, the Netherlands), whose cuff was attached to the left middle finger. The AP waveform was sampled at a frequency of 200 Hz. The beat‐to‐beat values of systolic, diastolic, and MAP and heart rate (HR) were obtained throughout the experiments. Simultaneously, the beat‐to‐beat values of cardiac output (CO), stroke volume (SV), and total peripheral resistance (TPR) were calculated from the aortic pressure waveform using a Modelflow^® software (BeatScope 1.1; Finapres Medical Systems BV, Arnhem, the Netherlands). The reliability of the CO measurement using the Modelflow^® has been confirmed previously (van Lieshout et al. 2003 (link); Matsukawa et al. 2004 (link); Tam et al. 2004 (link)).
Electromyogram (EMG) activity of the VL muscle was bilaterally measured using a pair of silver‐bar electrodes attached on the central portion of the muscle belly (Bagnoli‐2 EMG System, Delsys, Boston, MA). The EMG signals were amplified (×10,000) and passed through a bandpass filter between 20 and 2000 Hz.

Ishii K., Matsukawa K., Liang N., Endo K., Idesako M., Hamada H., Kataoka T., Ueno K., Watanabe T, & Takahashi M. (2014). Differential contribution of ACh‐muscarinic and β‐adrenergic receptors to vasodilatation in noncontracting muscle during voluntary one‐legged exercise. Physiological Reports, 2(11), e12202.

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