Ac1903

Manufactured by Merck Group

Sourced in Australia, United States

AC1903 is a laboratory equipment product manufactured by Merck Group. It is a high-precision analytical instrument designed for conducting various scientific analyses and experiments. The core function of AC1903 is to provide accurate measurements and data for research and testing purposes.

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Lab products found in correlation

3 protocols using ac1903

Chemical Screening for Cell Viability

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Dimethylsulfoxide (DMSO), Minimum Essential Medium Eagle (EMEM, M0643), sodium bicarbonate (NaHCO3), noble agar (A5431), resazurin sodium salt (199303), Vincristine (V0400000), Lomustine (L5918), YM-58483 (Y4895), Synta66 (SML1949), GSK1016790A (G0798), HC-067047 (SML0143), AC1903 (SML2244), ML218 (SML0385), Mibefradil dihydrochloride hydrate (M5441), NNC55-0396 hydrate (N0287), Yoda1 (SML1558), Verapamil hydrochloride (V4629) and Nifedipine (N7634) were purchased from Sigma-Aldrich (Ryde, NSW, Australia). IA65 was a kind gift from Professor William Denny and Dr. Ralph Stevenson, and was synthesised as previously described [28 (link)]. CellTiter-Fluor™ Cell Viability (G6081) was obtained from Promega (Madison, WA, USA).

Sedeeq M., Maklad A., Gueven N, & Azimi I. (2020). Development of a High-throughput Agar Colony Formation Assay to Identify Drug Candidates against Medulloblastoma. Pharmaceuticals, 13(11), 368.

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Gadolinium-based Contrast Agent Characterization

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GDD and Gd-DTPA were purchased from Selleck. Magnevist and Gadavist were purchased from Bayer Healthcare Pharmaceutical (Whippany, NJ, USA). GdCl₃, histamine, AC1903, and other salts used in the experimental solutions were purchased from Sigma-Aldrich (St. Louis, MO, USA).

Zhang W., Wang M., Lv W., White F.A., Chen X, & Obukhov A.G. (2023). Long-Term Treatment with Gadopentetic Acid or Gadodiamide Increases TRPC5 Expression and Decreases Adriamycin Nuclear Accumulation in Breast Cancer Cells. Cells, 12(9), 1304.

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TRPC5 Inhibition Ameliorates Proteinuria

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DOX-dependent proteinuria was induced as described above. TRPC5 inhibitors, 0.3 mg/kg (low dose) and 1 mg/kg (high dose) of ML204 or 0.75 mg/kg (low dose) and 1.5 mg/kg (high dose) of AC1903 were dissolved in dimethylsulfoxide (DMSO) and injected into mice 48 and 76 hrs after DOX-chow addition to establish the effect of TRPC5 inhibition on ACR. ML204 and AC1903 were obtained from Sigma-Aldrich. Control mice received only the vehicle.

Polat O.K., Isaeva E., Sudhini Y.R., Knott B., Zhu K., Noben M., Kumar V.S., Endlich N., Mangos S., Reddy T.V., Samelko B., Wei C., Altintas M.M., Dryer S.E., Sever S., Staruschenko A, & Reiser J. (2023). The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5. Kidney international, 103(6), 1056-1062.

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