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Anti il 17

Manufactured by BioXCell
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Anti-IL-17 is a laboratory reagent that binds and neutralizes the cytokine interleukin-17 (IL-17). It can be used in research applications to study the role of IL-17 in various biological processes.

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Lab products found in correlation

Venlafaxine, by Merck Group (1 mentions) Anti ifn γ, by BioXCell (1 mentions) Anti tnf α, by BioXCell (1 mentions)

2 protocols using anti il 17

1

Venlafaxine and Anti-IL-17 Treatment in Mice

Cited 2 times
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Venlafaxine (Sigma Aldrich, St. Louis, MO, USA), a serotonin-norepinephrine reuptake inhibitor (SNRI) anti-depressant, was dissolved in normal saline [40 (link)]. Anti-IL-17 (Bioxcell, Lebanon, NH, USA) and IgG (Bioxcell) was dissolved in purified dilution buffer [59 (link)]. The mice were treated with Venlafaxine (10 mg/kg, once a day for 3 weeks, i.p) or Anti-IL-17 (100 μg/mouse, once every 3 days for 2 weeks, i.p). The same volume of saline or IgG was administrated to mice in the vehicle group, respectively.
Kim J., Suh Y.H, & Chang K.A. (2021). Interleukin-17 induced by cumulative mild stress promoted depression-like behaviors in young adult mice. Molecular Brain, 14, 11.
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2

Adoptive Transfer of Diabetogenic T Cells

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In vitro differentiated effector CD4+ T cells or ex vivo cells isolated from diabetic NOD mice were transferred into NOD, NOD.Scid, or NOD.Ifng−/− recipients by i.v. injection in doses of 0.2-1×106 as indicated for the individual experiments. In some experiments, anti-IL-17, anti-TNFα, or anti-IFNγ (BioXcell or purified in house), were injected i.p. at indicated doses and times. Control mouse and rat IgG were purchased from Jackson ImmunoResearch Laboratories Inc. Diabetes incidence was monitored by blood glucose test using Bayer’s Contour meters and strips (Bayer); two consecutive readings of higher than 300mg/dl were considered indicative of diabetes.
Li C.R., Mueller E.E, & Bradley L.M. (2014). Islet antigen-specific Th17 cells can induce TNFα-dependent autoimmune diabetes. Journal of immunology (Baltimore, Md. : 1950), 192(4), 1425-1432.
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