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Efm192b

Manufactured by Leibniz Institute DSMZ
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Sourced in Germany

The EFM192B is a compact, high-performance field emission scanning electron microscope (FE-SEM) designed for versatile imaging and analysis applications. It provides high-resolution imaging capabilities with an accelerating voltage range of 0.5 to 30 kV. The instrument features a stable electron beam and advanced optics, delivering consistent and reliable performance.

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Lab products found in correlation

Skbr3, by American Type Culture Collection (2 mentions) Bt474, by American Type Culture Collection (2 mentions) Jimt1, by Leibniz Institute DSMZ (2 mentions) Hcc1419, by American Type Culture Collection (2 mentions) Efm 192a, by Leibniz Institute DSMZ (2 mentions) Uacc 893, by American Type Culture Collection (2 mentions) Hcc1954, by American Type Culture Collection (2 mentions) Mda mb 453, by American Type Culture Collection (2 mentions) Mda mb 361, by American Type Culture Collection (2 mentions) Zr 75 30, by American Type Culture Collection (2 mentions) Au565, by American Type Culture Collection (2 mentions) Efm192c, by Leibniz Institute DSMZ (2 mentions) Hcc202, by American Type Culture Collection (2 mentions) Hcc2218, by American Type Culture Collection (2 mentions) Hcc1569, by American Type Culture Collection (2 mentions)

2 protocols using efm192b

1

Comprehensive Panel of Breast Cancer Cell Lines

Check if the same lab product or an alternative is used in the 5 most similar protocols
Breast cancer cell lines derived from human female tumors were used in this study. The cell lines AU565, SKBR3, HCC1954, HCC-1569, HCC-202, HCC-2218, HCC-1419, MDA-MB-361, ZR-75-30, BT474, UACC893, and MDA-MB-453 were obtained from American Type Culture Collection (ATCC), Manassas, VA. HCC-3153 was obtained from UT-Southwestern, SUM190PT and SUM225CWN were provided by Steve Ethier at UCSF, and 21NT1, 21PT1, and 21MT1 were provided by Ruth Sager and Kornelia Polyak at the Dana-Farber Institute of Harvard, Cambridge MA. JIMT1, EFM192A, EFM192B, and EFM192C were obtained from DSMZ, Braunschweig Germany. Each cell line was genotyped to ensure accurate identity, and regularly screened for mycoplasma infection. Cell lines were maintained in their respective medium and serum concentration as recommended by originator specifications at 37°C in 5% CO2 in a humidified incubator and cultured according to ATCC recommendations.
Watson S.S., Dane M., Chin K., Tatarova Z., Liu M., Liby T., Thompson W., Smith R., Nederlof M., Bucher E., Kilburn D., Whitman M., Sudar D., Mills G.B., Heiser L.M., Jonas O., Gray J.W, & Korkola J.E. (2018). Microenvironment-Mediated Mechanisms of Resistance to HER2 Inhibitors Differ between HER2+ Breast Cancer Subtypes. Cell systems, 6(3), 329-342.e6.
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2

Comprehensive Panel of Breast Cancer Cell Lines

Check if the same lab product or an alternative is used in the 5 most similar protocols
Breast cancer cell lines derived from human female tumors were used in this study. The cell lines AU565, SKBR3, HCC1954, HCC-1569, HCC-202, HCC-2218, HCC-1419, MDA-MB-361, ZR-75-30, BT474, UACC893, and MDA-MB-453 were obtained from American Type Culture Collection (ATCC), Manassas, VA. HCC-3153 was obtained from UT-Southwestern, SUM190PT and SUM225CWN were provided by Steve Ethier at UCSF, and 21NT1, 21PT1, and 21MT1 were provided by Ruth Sager and Kornelia Polyak at the Dana-Farber Institute of Harvard, Cambridge MA. JIMT1, EFM192A, EFM192B, and EFM192C were obtained from DSMZ, Braunschweig Germany. Each cell line was genotyped to ensure accurate identity, and regularly screened for mycoplasma infection. Cell lines were maintained in their respective medium and serum concentration as recommended by originator specifications at 37°C in 5% CO2 in a humidified incubator and cultured according to ATCC recommendations.
Watson S.S., Dane M., Chin K., Tatarova Z., Liu M., Liby T., Thompson W., Smith R., Nederlof M., Bucher E., Kilburn D., Whitman M., Sudar D., Mills G.B., Heiser L.M., Jonas O., Gray J.W, & Korkola J.E. (2018). Microenvironment-Mediated Mechanisms of Resistance to HER2 Inhibitors Differ between HER2+ Breast Cancer Subtypes. Cell systems, 6(3), 329-342.e6.
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