Neurod1 cre

Manufactured by Jackson ImmunoResearch

Neurod1-Cre is a Cre recombinase mouse line that expresses Cre under the control of the Neurod1 promoter. Neurod1 is a transcription factor involved in the differentiation and maturation of neurons.

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3 protocols using neurod1 cre

Mouse Genetic Tools for Neuroscience

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All animal procedures performed at the University of California San Diego were conducted with approval by the Institutional Animal Care and Use Committee. All animal procedures at the University of Melbourne were conducted according to the National Health and Medical Research Council of Australia guidelines and were approved by the University of Melbourne Animal Experimentation Ethics Committee. Neurod1-Cre (Jackson Labs, 028364), Rosa26-LSL-tdTomato (Jackson Labs, 007914), Piezo2-IRES-cre (Jackson Labs, 027719), Rosa26-LSL-CHRM3* (Jackson Labs, 026220), Rosa26-LSL-DTR (Jackson Labs, 007900), Piezo^fl/fl (Jackson Labs, 027720), Vilin-cre (Jackson labs, 021504) and C57BL/6 mice were purchased from Jackson Laboratories. Animals were housed in groups (2–5 mice/cage) in a specific pathogen-free facility provided with environmental enrichment (shelter, nesting material, etc.) and had normal immune status.

Song Y., Fothergill L.J., Lee K.S., Liu B.Y., Koo A., Perelis M., Diwakarla S., Callaghan B., Huang J., Wykosky J., Furness J.B, & Yeo G.W. (2023). Stratification of enterochromaffin cells by single-cell expression analysis. bioRxiv.

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Genetically Modified Mouse Strains for Colitis Research

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C57BL/6J (Jax strain #000664), ROSA26^iDTR (C57BL/6-Gt [ROSA]26Sor tm1[HBEGF]Awai/J, Jax strain #007900), Villin^Cre (B6.Cg-Tg[Vil1-cre]997Gum/J, Jax strain #004586), and NeuroD1^cre (Tg[Neurod1-cre]1Able/J, Jax strain #028364) mice were obtained from the Jackson Laboratory. Neurod1-loxP⁶⁸ (link) was re-derived at the Johns Hopkins transgenic core facility, using sperm provided as a kind gift from Dr Sandra Goebbels (Max Planck Institute of Experimental Medicine, Goettingen, Germany). Rosa26^LSLNeurod1 was a kind gift from Gabriela Pavlinkova, PhD (Czechia).⁶⁹ All mouse strains had free access to water and standard rodent chow (Teklad Global 18% protein rodent diets; Envigo). Studies took place exclusively in male mice to ensure consistency of colitis induced, given that a more robust and consistent colitis develops in males as opposed to females.⁶¹

Raouf Z., Steinway S.N., Scheese D., Lopez C.M., Duess J.W., Tsuboi K., Sampah M., Klerk D., El Baassiri M., Moore H., Tragesser C., Prindle T J.r., Wang S., Wang M., Jang H.S., Fulton W.B., Sodhi C.P, & Hackam D.J. (2024). Colitis-Induced Small Intestinal Hypomotility Is Dependent on Enteroendocrine Cell Loss in Mice. Cellular and Molecular Gastroenterology and Hepatology, 18(1), 53-70.

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Transgenic Mouse Models in Neuroscience

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Animals were maintained in an AAALAC-approved animal facility and all procedures were performed in accordance with IACUC guidelines (protocol number 17013). Pcp2/L7-Cre, Neurod1-Cre and Atoh1-Cre mice were obtained from The Jackson Laboratory (catalog numbers: 004146, 028364 and 011104). Memo1 mutant mice were generated using ES-cells from the European Conditional Mouse Mutagenesis (EUCOMM) repository as described previously (Van Otterloo et al., 2016 (link)). Mouse studies were allocated by genotype, and were not blinded.

Jereb S., Hwang H.W., Van Otterloo E., Govek E.E., Fak J.J., Yuan Y., Hatten M.E, & Darnell R.B. (2018). Differential 3’ processing of specific transcripts expands regulatory and protein diversity across neuronal cell types. eLife, 7, e34042.

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