17l 5 probe

Manufactured by Siemens

Sourced in Germany

The 17L-5 probe is a laboratory equipment manufactured by Siemens. It is a specialized device designed for use in various scientific and research applications. The core function of the 17L-5 probe is to provide precise measurement and data collection capabilities, without any interpretation or extrapolation on its intended use.

Automatically generated - may contain errors

Lab products found in correlation

Sequoia 512 system, by Siemens (5 mentions)

5 protocols using 17l 5 probe

Echocardiographic Assessment of Cardiac Function

Check if the same lab product or an alternative is used in the 5 most similar protocols

Cardiac dimensions and function were evaluated by echocardiography with a Sequoia 512 system and a 17L-5 probe (Siemens, Germany). After mice were anaesthetized with 1.5% inhalational isoflurane, two-dimensional parasternal short-axis images of the left ventricle were obtained at the level of the papillary muscles. M-mode echocardiography was performed to measure the LVEDd, LVESd, and LVFS.

Wang Y., Chen Y., Yan Y., Li X., Chen G., He N., Shen S., Chen G., Zhang C., Liao W., Liao Y, & Bin J. (2016). Loss of CEACAM1, a Tumor-Associated Factor, Attenuates Post-infarction Cardiac Remodeling by Inhibiting Apoptosis. Scientific Reports, 6, 21972.

+ Open protocol

+ Expand

Cardiac Function Assessment via Echocardiography

Check if the same lab product or an alternative is used in the 5 most similar protocols

Both cardiac function and cardiac remodeling were evaluated by echocardiography using a Sequoia 512 system with a 17L-5 probe (Siemens, Germany). Mice were anesthetized by inhalation of 2% isoflurane and M-mode images were obtained with guidance by 2D parasternal short-axis images of the left ventricle at the level of the papillary muscles. LVEF, LVESd and LVEDd were measured from the M-mode images.

Hao H., Ma S., Zheng C., Wang Q., Lin H., Chen Z., Xie J., Chen L., Chen K., Wang Y., Huang X., Cao S., Liao W., Bin J, & Liao Y. (2021). Excessive fibroblast growth factor 23 promotes renal fibrosis in mice with type 2 cardiorenal syndrome. Aging (Albany NY), 13(2), 2982-3009.

+ Open protocol

+ Expand

Echocardiography in Murine Models

Cited 1 time

Check if the same lab product or an alternative is used in the 5 most similar protocols

Non-invasive transthoracic echocardiography was performed in mice using a Sequoia 512 system with a 17L-5 probe (Siemens, Germany). [30 (link)]

Hao H., Li X., Li Q., Lin H., Chen Z., Xie J., Xuan W., Liao W., Bin J., Huang X., Kitakaze M, & Liao Y. (2016). FGF23 promotes myocardial fibrosis in mice through activation of β-catenin. Oncotarget, 7(40), 64649-64664.

+ Open protocol

+ Expand

Echocardiographic Evaluation of Murine Cardiac Function

Check if the same lab product or an alternative is used in the 5 most similar protocols

Transthoracic echocardiography was performed in mice 4 weeks after surgery using a Sequoia 512 system with a 17L-5 probe (Siemens, Germany). Mice were anesthetized with inhalational isoflurane at a concentration of 1.5%. Under M-mode, we measured the posterior wall thickness (LVPWd), end-diastolic diameter (LVEDd), and end-systolic diameter (LVESd). LV fractional shortening (FS) was calculated as follows: LVFS (%) = (LVEDd—LVESd) / LVEDd × 100.

Lin H., Shen L., Zhang X., Xie J., Hao H., Zhang Y., Chen Z., Yamamoto H., Liao W., Bin J., Cao S., Huang X, & Liao Y. (2016). HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload. PLoS ONE, 11(6), e0158514.

+ Open protocol

+ Expand

Cardiac Dimensions and Function Evaluation

Check if the same lab product or an alternative is used in the 5 most similar protocols

Cardiac dimensions and function were evaluated by echocardiography with the Sequoia 512 system and a 17 L-5 probe (Siemens, Berlin, Germany) before infarction (pre-MI), and at 1, 14, 30, and 60 days post-MI (also see details in the Supplementary material online).

Li X., He X., Wang H., Li M., Huang S., Chen G., Jing Y., Wang S., Chen Y., Liao W., Liao Y, & Bin J. (2018). Loss of AZIN2 splice variant facilitates endogenous cardiac regeneration. Cardiovascular Research, 114(12), 1642-1655.

+ Open protocol

+ Expand

About PubCompare

Our mission is to provide scientists with the largest repository of trustworthy protocols and intelligent analytical tools, thereby offering them extensive information to design robust protocols aimed at minimizing the risk of failures.

We believe that the most crucial aspect is to grant scientists access to a wide range of reliable sources and new useful tools that surpass human capabilities.

However, we trust in allowing scientists to determine how to construct their own protocols based on this information, as they are the experts in their field.

Ready to get started?

Revolutionizing how scientists
search and build protocols!