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γENaC is a laboratory equipment product manufactured by Thermo Fisher Scientific. It is a component of the epithelial sodium channel (ENaC) family, which plays a critical role in the regulation of sodium ion transport across cell membranes. The core function of γENaC is to facilitate the measurement and analysis of this sodium ion transport process in experimental settings.

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Lab products found in correlation

αenac, by Thermo Fisher Scientific (2 mentions) Agtrap, by Thermo Fisher Scientific (2 mentions) Nkcc2, by Thermo Fisher Scientific (2 mentions) βenac, by Thermo Fisher Scientific (2 mentions)

2 protocols using γenac

1

Quantitative RT-PCR Analysis of Kidney Genes

Cited 1 time
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Quantitative real-time reverse transcriptase–PCR was performed using designed TaqMan probes (Agtrap, Mm00507771_m1; AT1R, Mm01957722_s1; NHE3, Mm01352473_m1; NKCC2, Mm01275821_m1; NCC, Mm00490213_m1; αENaC, Mm00803386_m1; βENaC, Mm00441215_m1; γENaC, Mm00441228_m1; and AGT, Mm00599662_m1; Applied Biosystems, Carlsbad, CA), as described previously.8 (link) The mRNA levels were normalized to those of the 18S rRNA control.
Ohsawa M., Tamura K., Wakui H., Maeda A., Dejima T., Kanaoka T., Azushima K., Uneda K., Tsurumi-Ikeya Y., Kobayashi R., Matsuda M., Uchida S., Toya Y., Kobori H., Nishiyama A., Yamashita A., Ishikawa Y, & Umemura S. (2014). Deletion of the angiotensin II type 1 receptor–associated protein enhances renal sodium reabsorption and exacerbates angiotensin II–mediated hypertension. Kidney International, 86(3), 570-581.
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2

Real-Time RT-qPCR Analysis of Kidney Genes

Check if the same lab product or an alternative is used in the 5 most similar protocols
Quantitative real-time reverse transcriptase–PCR was performed using designed TaqMan probes (Agtrap, Mm00507771_m1; AT1R, Mm01957722_s1; NHE3, Mm01352473_m1; NKCC2, Mm01275821_m1; NCC, Mm00490213_m1; αENaC, Mm00803386_m1; βENaC, Mm00441215_m1; γENaC, Mm00441228_m1; and AGT, Mm00599662_m1; Applied Biosystems, Carlsbad, CA), as described previously.8 The mRNA levels were normalized to those of the 18S rRNA control.
Ohsawa M., Tamura K., Wakui H., Maeda A., Dejima T., Kanaoka T., Azushima K., Uneda K., Tsurumi-Ikeya Y., Kobayashi R., Matsuda M., Uchida S., Toya Y., Kobori H., Nishiyama A., Yamashita A., Ishikawa Y, & Umemura S. (2014). Deletion of the angiotensin II type 1 receptor–associated protein enhances renal sodium reabsorption and exacerbates angiotensin II–mediated hypertension. Kidney International, 86(3), 570-581.
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