MRI imaging was performed using two different systems; the 1.5T Ingenia (Philips, Eindhoven, The Netherlands) with anterior and posterior array and the 3T Verio (Siemens, Erlangen, Germany) with the 32-channel body coil. The same machine was used to acquire serial measurements on any individual subject. The following sequences were obtained before administration of contrast: coronal T1-weighted, coronal and axial Proton density fat saturation. Pre-contrast images of coronal and sagittal oblique T1-weighted fat saturation (T1FS) were also obtained. Approximately two minutes after the injection of Gadolinium (0.1 mmol/kg), post-contrast images on coronal and sagittal oblique T1FS sequences were then obtained. The subtraction images were generated by subtracting the initial pre-contrast T1FS images from the corresponding post-contrast T1FS. A slice thickness of 3 mm was employed.
Ingenia 1.5t
Manufactured by Philips
Sourced in Netherlands
The Ingenia 1.5T is a magnetic resonance imaging (MRI) system manufactured by Philips. It operates at a magnetic field strength of 1.5 Tesla and is designed for a wide range of clinical applications.
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Lab products found in correlation
Ingenia 3t, by Philips (4 mentions)
Prohance, by Bracco (3 mentions)
Multihance, by Bracco (3 mentions)
Achieva 3t, by Philips (2 mentions)
Magnetom avanto fit 1.5 t, by Siemens (2 mentions)
Verio 3t, by Siemens (1 mentions)
Viewforum, by Philips (1 mentions)
Intellispace portal, by Philips (1 mentions)
Achieva, by Philips (1 mentions)
Dotarem, by Guerbet (1 mentions)
Panorama hfo, by Philips (1 mentions)
Magnetom vida, by Siemens (1 mentions)
Magnetom espree, by Siemens (1 mentions)
Achieva dstream 3t, by Philips (1 mentions)
3t prisma, by Siemens (1 mentions)
3t discovery mr750, by GE Healthcare (1 mentions)
Brilliance ict 256, by Philips (1 mentions)
Ingenia 3.0t, by Philips (1 mentions)
Glucagon g novo, by Eisai (1 mentions)
Gadovist, by Bayer (1 mentions)
37 protocols using ingenia 1.5t
1
Contrast-enhanced MRI Protocol for Imaging
2
Cardiac MRI Imaging Protocol
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Standard CMR was performed supine in a 1.5T Achieva™, 1.5T Ingenia™ (1.5T) or 3T Ingenia™ (3T) whole-body scanner (Philips Healthcare, Best, The Netherlands), with a commercial cardiac phased-array receiver coil as previously described.18) (link)19) (link) Cine long axis 2-, 3- and 4-chamber views, as well as short axis cine images were obtained using a breath-hold, segmented-k-space balanced steady-state free precession sequence (bSSFP) employing retrospective electrocardiogram or pulse oximetric gating. Data were analyzed using commercially available workstations (Viewforum™ and IntelliSpace™ Portal, ISP™; Philips Healthcare) and a certified software (cmr42 Version 5.6.6, Circle Cardiovascular Imaging Inc., Calgary, Canada) as semi-automatic software for volumetric analysis. LV volumes and ejection fraction was acquired in short axis stacks. Details of acquisition and post-processing are available in the Supplementary Data 1 .
3
Comprehensive Brain MRI Analysis Protocol
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All patients included underwent one or multiple 1.5T MRI scans (Philips Ingenia 1.5T). Imaging techniques such as ROI’s (region-of-interest), SBM (surface-based morphometry) and VBM (voxel-based morphometry) were applied to specifically analyse the brain areas of interest.
Anomalies evaluated in brain MRI were morphological changes in term of shape and increase or reduction in volume of the specific brain area of interest and focal or diffuse abnormal signal intensities of brain tissue. White matter abnormalities were classified as multiple punctate or plaque-like confluent hypersignals on T2 and FLAIR sequences. These anomalies were considered at the level of corpus callosum, periventricular white matter, ventricular space, cerebellum, subarachnoid space and thalamus. Normal variants or minor abnormalities, such as posterior fossa cysts, abnormal hippocampal shape or minor cerebellar atrophy, were not considered as abnormalities and were classified as normal MRI.
Anomalies evaluated in brain MRI were morphological changes in term of shape and increase or reduction in volume of the specific brain area of interest and focal or diffuse abnormal signal intensities of brain tissue. White matter abnormalities were classified as multiple punctate or plaque-like confluent hypersignals on T2 and FLAIR sequences. These anomalies were considered at the level of corpus callosum, periventricular white matter, ventricular space, cerebellum, subarachnoid space and thalamus. Normal variants or minor abnormalities, such as posterior fossa cysts, abnormal hippocampal shape or minor cerebellar atrophy, were not considered as abnormalities and were classified as normal MRI.
4
Breast MRI Protocol for Dynamic Contrast Enhancement
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All the patients had breast MRI on the same MR device (Ingenia 1.5 T, Philips medical imaging, Best, the Netherlands). The rationale is detailed in Additional file 1 : Appendix 1. The DCE sequences were performed before, then 1, 2, 3, 4, and 5 min after intra-venous injection of gadolinium (DOTAREM: 0.2 cc/kg, Guerbet, Aulnay sous Bois, France). Native reconstructions were performed for each acquisition time (DCEn 0, DCEn 1, DCEn 2, DCEn 3, DCEn 4, and DCEn 5). Subtractions were also made between the post-contrast and pre-contrast acquisitions, and the pre-contrast acquisition was used as a mask for each time-point (DCEs 1, DCEs 2, DCEs 3, DCEs 4, and DCEs 5). The native T1-weighted, T2-weighted, DCEn, and DCEs sequences were used for the segmentations of the lesions and analysis performed by the radiologists.
5
Dynamic Contrast-Enhanced Brain MRI Protocol
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Brain MRI examinations were performed using 1T (n = 8), 1.5T (n = 52), and 3T (n = 24) MRI systems (Panorama HFO, Ingenia 1.5T, Ingenia 3T, Achieva: Philips Healthcare, Eindhoven; MAGNETOM Espree, MAGNETOM Vida: Siemens, Erlangen) with a 32-channel head coil in the supine position.
The MRI acquisition protocols are summarized in Table 1. DSC-MRI was only performed using Ingenia 1.5T, Ingenia 3T, or MAGNETOM Vida (3T). For DSC-MRI, an intravenous bolus of 20 mL of gadobenate dimeglumine (Multihance, Bracco Diagnostics, Singen, Germany) or gadoteridol (ProHance, Bracco Diagnostics, Inc., Princeton, NJ) was administered using a power injector via a peripheral arm vein at a flow rate of 5.0 mL/s, followed by a 20 mL saline flush. An additional 5 mL of contrast was administered 5 minutes before the dynamic perfusion scan. The parameters of fast field echo T2*-weighted imaging were as follows: dynamic measurements, 40; temporal resolution, 1.5 s; and total acquisition time, 1 min and 4.5 s. Steroids were given in five and four patients with PA and MB, respectively, within three months before the date of MRI examinations.
The MRI acquisition protocols are summarized in Table 1. DSC-MRI was only performed using Ingenia 1.5T, Ingenia 3T, or MAGNETOM Vida (3T). For DSC-MRI, an intravenous bolus of 20 mL of gadobenate dimeglumine (Multihance, Bracco Diagnostics, Singen, Germany) or gadoteridol (ProHance, Bracco Diagnostics, Inc., Princeton, NJ) was administered using a power injector via a peripheral arm vein at a flow rate of 5.0 mL/s, followed by a 20 mL saline flush. An additional 5 mL of contrast was administered 5 minutes before the dynamic perfusion scan. The parameters of fast field echo T2*-weighted imaging were as follows: dynamic measurements, 40; temporal resolution, 1.5 s; and total acquisition time, 1 min and 4.5 s. Steroids were given in five and four patients with PA and MB, respectively, within three months before the date of MRI examinations.
6
Interictal fMRI Scanning of TMS Patients
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The patients underwent fMRI scanning not earlier than a week before and not later than a week after the 5-day TMS course on a Philips Ingenia 1.5T magnetic resonance imaging scanner in the interictal period (at least 24 h after the last attack). The scanning was performed in the evening (from 5 p.m. to 8 p.m.). Patients did not eat or drink coffee at least 3 h before the scan. The protocols–T1-weighted (301 axial sections, planar resolution of 1 × 1 mm; repetition time/echo time 8.0/3.7ms; flip angle = 8) and EPI (echo-planar imaging scan) (35 axial sections; planar resolution of 3.03 × 3.03 mm; section depth of 4.0 mm; repetition time/echo time 3,000/50 ms; flip angle = 90)–were obtained for each patient with preceding instructions: “Remain lying and relaxed, with closed eyes, but do not sleep.”
7
Comprehensive MRI Evaluation of New Device
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Multiple scanners were used in this study in order to evaluate the reproducibility (same participant, different scanner) and repeatability (same participant, same scanner) of the new device. These scanners were Siemens Prisma 3T, Siemens Avanto fit 1.5T, GE Discovery MR750 3T, GE Optima MR450w 1.5T, Philips Achieva dStream 3T and Philips Ingenia 1.5T. Fat-saturated T1-weighted gradient recalled echo (GRE) images without contrast-agent were used for volumetric analysis. cT1, PDFF and T2* maps were acquired as previously described using multislice shortened MOLLI and IDEAL sequences [31 , 32 ] with extensive validation. Imaging data as DICOM files are transferred using a secure online portal for analysis by an operator. Summary results are then reviewed and returned to the referring clinician as a report.
8
Imaging-Based Diagnosis and Evaluation of HCC
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The diagnosis of HCC was done with contrast-enhanced imaging (multidetector row CT or magnetic resonance imaging (MRI)). Commercially available clinical 3.0 Tesla MR imaging system (Ingenia 3.0 T; Philips Healthcare, Best, Netherlands) or a 1.5 Tesla MR imaging system (Ingenia 1.5 T; Philips Healthcare, Best, Netherlands) or CT imaging systems (Philips Brilliance 64 or Philips Brilliance 256 iCT, both Philips Healthcare, Best, the Netherlands) were used. HCC was diagnosed only if typical imaging features were detected [11 ]. Follow-up imaging was also performed by contrast-enhanced imaging (CT or MRI). Response to treatment was determined by contrast-enhanced imaging at three months (Fig 1b ). Response to local ablative therapy was defined as lack of contrast enhancement in the lesion of interest as sign of vital tumor mass. Non-response was defined as detection of vital tumor mass. Additionally, response was also classified according to RECIST criteria [12 (link)]. Response was defined as RECIST complete remission (CR), while no response was defined as RECIST stable disease (SD) or progressive disease (PD). Follow up imaging was then performed 90 days after local ablative therapy and further recorded until the end of study, death or occurrence of Non-TL.
9
Cardiac Fibrosis and Epicardial Fat Imaging
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All images were obtained using a Philips Medical System Ingenia 1.5 T clinical scanner. For fibrosis evaluation, LGE-MRI was acquired following the methods previously described (21 (link)). Briefly, scans were performed 15–25 min after contrast injection, using a 3D inversion-recovery, respiration-navigated, ECG-gated, gradient echo pulse sequence. Acquisition parameters included transverse imaging volume with a voxel size of 1.25 x 1.25 x 2.5 mm (reconstructed to 0.625 x 0.625 x 1.25 mm).
To assess EAT, a 3D respiration-navigated, ECG-gated Dixon sequence was obtained with the following parameters: 1.5 mm slice thickness, repetition time (TR) = 5.4 ms, echo time 1 (TE1) / echo time 2 (TE2) = 1.8/4.0 ms, flip angle (α) = 15°, voxel size = 1.5 × 1.5 × 3.0 mm3 (reconstructed to 1.0 × 1.0 × 1.5 mm3), parallel imaging factor (SENSE) = 1.5 in both phase encoding directions and water fat shift = 0.16 pixel. Arrhythmia rejection was applied, the T2 preparation duration was 50 ms and the acquisition window was 100 to 156 ms.
To assess EAT, a 3D respiration-navigated, ECG-gated Dixon sequence was obtained with the following parameters: 1.5 mm slice thickness, repetition time (TR) = 5.4 ms, echo time 1 (TE1) / echo time 2 (TE2) = 1.8/4.0 ms, flip angle (α) = 15°, voxel size = 1.5 × 1.5 × 3.0 mm3 (reconstructed to 1.0 × 1.0 × 1.5 mm3), parallel imaging factor (SENSE) = 1.5 in both phase encoding directions and water fat shift = 0.16 pixel. Arrhythmia rejection was applied, the T2 preparation duration was 50 ms and the acquisition window was 100 to 156 ms.
10
Prostate MRI Imaging Protocol
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All examinations were performed on a 3T or 1.5T MR scanner (Achieva 3T and Ingenia 1.5T; Philips Healthcare, Eindhoven, The Netherlands) using a 32-channel phased-array coil. To prevent artifacts from bowel peristalsis, 1 mg of intramuscular glucagon (Glucagon G Novo; Eisai, Tokyo, Japan) was administered immediately before the MRI examination. We used the following hospital prostate imaging protocols: axial and coronal T2-weighted imaging (T2WI) (TR/TE = 3500–4000 ms/70–100 ms; section thickness/intersection gap = 3 mm/0 mm; FOV = 160 × 160 mm2; matrix = 512 × 260, zero-filled interpolation [ZIP] = 1024), DWI (TR/TE = 4000–6500/55–74 ms; section thickness/intersection gap = 3 mm/0 mm; FOV = 240 × 240 mm2; matrix = 256 × 256; diffusion sensitization gradients oriented along three orthogonal directions at five b-values [0, 500, 1000, 1500, and 2000 s/mm2]), and gadolinium-enhanced dynamic MRI (enhanced T1-weighted high-resolution isotropic volume excitation) (TR/TE = 3.8/1.9 ms; flip angle = 15°; section thickness = 3.0 mm [ZIP, 1.5 mm]; FOV = 240 × 240 mm2; matrix = 240 × 194 [ZIP, 512]). In the dynamic studies, images were sequentially obtained at baseline (unenhanced) and at 25, 60, and 180 s after a bolus injection of 0.1 mmol/kg of gadodiamide hydrate (Omniscan; Daiichi Sankyo, Tokyo, Japan) or gadobutrol (Gadovist; Bayer Pharmaceuticals, Osaka, Japan).
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