Accu chek aviva nano

Manufactured by Roche

Sourced in Switzerland, Germany

The Accu-Chek Aviva Nano is a blood glucose monitoring system designed for people with diabetes. It is a compact and portable device that allows users to quickly and easily measure their blood glucose levels. The Accu-Chek Aviva Nano uses a small blood sample to provide accurate results, enabling users to monitor their diabetes management effectively.

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Lab products found in correlation

W266523, by Merck Group (2 mentions) Streptozotocin (stz), by Merck Group (1 mentions) Ifn β, by BD (1 mentions) S0130, by Merck Group (1 mentions) Thermalert th 5, by Physitemp (1 mentions) Ultrasensitive mouse insulin elisa, by Mercodia (1 mentions) Echomri 4 in 1 1000, by Echo Medical Systems (1 mentions) Polar team system, by Polar Electro (1 mentions)

17 protocols using accu chek aviva nano

Metabolic Profiling in Aging Rats

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Body composition as well as oral glucose tolerance tests (oGTT) and insulin tolerance tests (ITT) were assessed in all rats after 0, 6, 12, 16, and 18 months of treatment (corresponding to 4, 10, 16, 20, and 22 months of age, respectively). Body composition was assessed in conscious rats using a whole-body composition analyzer (EchoMRI 4-in-1/1000, Echo Medical Systems LLC, Houston, TX, USA) throughout the study. For oGTT, rats were fasted overnight (16 h). Baseline blood glucose levels were assessed using a glucometer (AccuChek Aviva Nano, Roche Diagnostics) from a saphenous venipuncture. Rats were then administered a glucose solution (0.5 g D-glucose per mL water) by oral gavage at a dose of 2 g/kg body weight. Blood glucose concentrations were assessed at 15, 30, 60, 90, and 120 min post-glucose administration by saphenous venipuncture.
For the ITT, rats underwent a fast of 4 h prior to blood sampling. Baseline glucose levels were assessed from a saphenous venipuncture using an AccuChek Aviva Nano blood glucometer (Roche Diagnostics). Rats then received an intraperitoneal injection of insulin (1.0 IU/kg body weight for males or 0.75 IU/kg body weight for females). Blood glucose concentrations were then assessed at 15, 30, 60, 90, and 120 min post-insulin injection by saphenous venipuncture.

Noble R.M., Jahandideh F., Armstrong E.A., Bourque S.L, & Yager J.Y. (2022). Broccoli Sprouts Promote Sex-Dependent Cardiometabolic Health and Longevity in Long-Evans Rats. International Journal of Environmental Research and Public Health, 19(20), 13468.

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Streptozotocin-Induced Diabetic Mouse Model

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Body weights (BW) and blood sugar levels (BS) of mice were measured using ACCU-CHEK Aviva Nano (Roche) after a fasting for 4 hours. To induce the diabetic state, streptozotocin (STZ; Sigma-Aldrich) dissolved in 0.05 M citrate buffer (pH 4.5) was injected intraperitoneally once into mice (150 mg/kg). Control mice received an injection of equal amount of citrate buffer. At day 4 after the injection of STZ, BW and BS were measured again after a fasting for 4 hours. Mice were considered to be the state of diabetes when BS exceeded 300 mg/dl. All the experiments were performed with mice at day 7 after injection of STZ when angiogenic hyperpermeable vessels had not yet appeared33 (link).

Arima M., Cui D., Kimura T., Sonoda K.H., Ishibashi T., Matsuda S, & Ikeda E. (2016). Basigin can be a therapeutic target to restore the retinal vascular barrier function in the mouse model of diabetic retinopathy. Scientific Reports, 6, 38445.

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Serological Biomarker Evaluation

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Respective protein levels in mouse sera and culture media were evaluated using ELISA kits for OPG (R&D Systems), IFN-β (BD PharMingen), or insulin (Morinaga Institute of Biological Science). Blood glucose levels were measured using Accu-Chek Aviva Nano (Roche Diagnostics). Enzymatic activities of aspartate aminotransferase (AST), alanine aminotransferase (ALT), and lipase were measured using Fuji Dry-chem 3500i (Fuji film).

Kuroda Y., Maruyama K., Fujii H., Sugawara I., Ko S.B., Yasuda H., Matsui H, & Matsuo K. (2016). Osteoprotegerin Regulates Pancreatic β-Cell Homeostasis upon Microbial Invasion. PLoS ONE, 11(1), e0146544.

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Induction of Mild Diabetes in Rats

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Mild diabetes was induced according to previous protocols [17 (link)], using a single high IV dose of streptozotocin (125 mg/kg) preceded by IV dosing of nicotinamide (67 mg/kg). One group of animals (all in cohort A) was induced twice, at age 16 and 30 weeks (HFD-D_A, n = 6), due to waning effect of the first induction, the other group (all in cohort B) was induced once at age 40 weeks (HFD-D_B, n = 6). For monitoring of glucose (GLU) and fructosamine (FRA) levels, plasma samples were evaluated every 4–6 weeks. Weekly blood GLU was assessed from ear capillary blood, using a hand-held device (Accu-Chek^® Aviva Nano, Roche Denmark, Hvidovre, DK) (data not shown).

Ludvigsen T.P., Kirk R.K., Christoffersen B.Ø., Pedersen H.D., Martinussen T., Kildegaard J., Heegaard P.M., Lykkesfeldt J, & Olsen L.H. (2015). Göttingen minipig model of diet-induced atherosclerosis: influence of mild streptozotocin-induced diabetes on lesion severity and markers of inflammation evaluated in obese, obese and diabetic, and lean control animals. Journal of Translational Medicine, 13, 312.

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Oral Glucose Tolerance Test in Rats

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At approximately 20 weeks of age, offspring were fasted overnight, and baseline glucose levels were assessed from a saphenous venipuncture using an AccuChek Aviva Nano blood glucometer (Roche Diagnostics). Rats were then administered a glucose solution (0.5 g D-glucose per mL water) by oral gavage at a dose of 2 g/kg body weight. Blood glucose concentrations were then assessed at 15, 30, 60, 90, and 120 minutes by saphenous venipuncture following glucose administration.

Jahandideh F., Bourque S.L., Armstrong E.A., Cherak S.J., Panahi S., Macala K.F., Davidge S.T, & Yager J.Y. (2020). Late-pregnancy uterine artery ligation increases susceptibility to postnatal Western diet-induced fat accumulation in adult female offspring. Scientific Reports, 10, 6926.

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Exendin-4 Impact on Glucose Tolerance

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After 13 days of treatment with exendin-4, an OGTT was performed after an overnight fast where the animals were given free access to water. Healthy, untreated mice (same strain, gender and age as study animals) were included as normal controls to provide a baseline. The test was performed 30 min after exendin-4 was administered or not. Each mouse was gavaged with 1.5 g/kg D-glucose (Fresenius Kabi, Oslo, Norway). Blood glucose was measured at 0, 15, 30, 60 and 120 min after glucose administration using a glucometer (Accu-Chek Aviva Nano, Roche Diagnostics, Indiana, USA). The area under the curve (AUC) of the glucose levels was calculated for each mouse.

Sahraoui A., Winzell M.S., Gorman T., Smith D.M., Skrtic S., Hoeyem M., Abadpour S., Johansson L., Korsgren O., Foss A, & Scholz H. (2015). The Effects of Exendin-4 Treatment on Graft Failure: An Animal Study Using a Novel Re-Vascularized Minimal Human Islet Transplant Model. PLoS ONE, 10(3), e0121204.

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Dextrose-Induced Glycemic Response in Rats

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On PD 71, following 12 h of fasting, rats were given an injection of 0.75 g/mL dextrose solution (an intraperitoneal dose 1.75 g/kg) as previously described (MacKay et al., 2014 (link)). Blood glucose was measured by applying a drop of blood (via tail venipuncture) onto a test strip and then taking a reading with an over the counter glucose measuring sticks and blood glucose meter (Accu-Chek Aviva Nano, Roche Diagnostics, Mannheim, Germany). Glucose levels were assessed immediately before the injection (baseline) and 15, 30, 60 and 120 min post-injection.

Ali E.F., MacKay J.C., Graitson S., James J.S., Cayer C., Audet M.C., Kent P., Abizaid A, & Merali Z. (2018). Palatable Food Dampens the Long-Term Behavioral and Endocrine Effects of Juvenile Stressor Exposure but May Also Provoke Metabolic Syndrome in Rats. Frontiers in Behavioral Neuroscience, 12, 216.

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Induction of Diabetes in Mice

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Diabetes was induced using a protocol of multiple low-dose STZ (S0130, Sigma-Aldrich) injections. Female mice were injected intraperitoneally with STZ on five consecutive days at a concentration of 50 mg/kg of body weight. STZ was prepared fresh in 0.1 M sodium citrate buffer (pH 4.5) to a final concentration of 7.5 mg/ml (Tesch and Allen, 2007 (link)). Blood glucose levels were monitored 10 days after the last STZ injection using a commercial glucometer (Accu-Chek Aviva Nano, Roche Diagnostics). Mice with non-fasting blood glucose above 250 mg/dl (Pavlinkova et al., 2008 (link)) were considerer diabetic mice. Diabetic and age-matched control female mice were mated with Daam1^+/gt male mice.

López-Escobar B., Cano D.A., Rojas A., de Felipe B., Palma F., Sánchez-Alcázar J.A., Henderson D, & Ybot-González P. (2014). The effect of maternal diabetes on the Wnt-PCP pathway during embryogenesis as reflected in the developing mouse eye. Disease Models & Mechanisms, 8(2), 157-168.

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Quantifying Brown Adipose Tissue Activity

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[¹⁸F]-FDG PET imaging was performed at the Unidad de Imagen Molecular, Centro de Investigaciones Médico-Sanitarias (CIMES), Spain.⁶⁷ (link) Animals (n = 6; 34.33 ± 3.2 g) fasted overnight were anesthetized by inhalation of a mixture of isoflurane/oxygen (5% for induction and 2% for maintenance) and placed prone on a PET scanner bed 1 h after intraperitoneal radiotracer administration (10.36 ± 2.22 MBq) to perform a static acquisition of 30 min. Images were subsequently reconstructed using an iterative 3D row action maximum likelihood algorithm (3D RAMLA). Corrections for dead time decay and random coincidences were applied. Images were reconstructed on a 128 × 128 × 120 matrix, where the voxel size equals 1 × 1 × 1 mm. PET images were normalized using the whole brain average uptake with PMOD software (3.3 PMOD Technologies, Zurich, Switzerland). The inguinal AT region of interest (ROI) was manually drawn.
Animals were kept at 25°C to maintain BAT activity as low as possible and to ensure that differences in thermogenic activity were only due to treatment. The blood glucose concentration (88.83 ± 6.88 mg/dL) was determined before tracer injection using a glucose-level meter with test strips (Accu-Chek Aviva Nano; Roche, Mannheim, Germany).

Lhamyani S., Gentile A.M., Giráldez-Pérez R.M., Feijóo-Cuaresma M., Romero-Zerbo S.Y., Clemente-Postigo M., Zayed H., Olivera W.O., Bermúdez-Silva F.J., Salas J., Gómez C.L., Hmadcha A., Hajji N., Olveira G., Tinahones F.J, & El Bekay R. (2021). miR-21 mimic blocks obesity in mice: A novel therapeutic option. Molecular Therapy. Nucleic Acids, 26, 401-416.

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Blood Glucose and Insulin Analysis

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Analysis of blood samples collected during the MD and sucrose loading studies were assigned to the Okayama Medical Association, Medical Center (Okayama, Japan). Blood glucose was measured as plasma glucose concentration using the glucose oxidase (GOD) method. Serum insulin was measured by enzyme immunoassay. In the glucose loading study, a lancing device (Safe-T-Pro Uno, Roche Diagnostics, Basel, Switzerland) was used for the self-sampling of capillary blood, and blood glucose concentration was self-measured with a meter (ACCU-CHEK Aviva Nano, Roche Diagnostics, Basel, Switzerland). The conductor attended in the self-measurement to check the condition of subjects, to confirm of the processes, to record measured values.

Sadakiyo T., Ishida Y., Inoue S.I., Taniguchi Y., Sakurai T., Takagaki R., Kurose M., Mori T., Yasuda-Yamashita A., Mitsuzumi H., Kubota M., Watanabe H, & Fukuda S. (2017). Attenuation of postprandial blood glucose in humans consuming isomaltodextrin: carbohydrate loading studies. Food & Nutrition Research, 61(1), 1325306.

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