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Anti h4k20me1

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Anti-H4K20me1 is a laboratory product that can be used to detect the presence of mono-methylated histone H4 at lysine 20 (H4K20me1) in biological samples. This antibody can be utilized in various analytical techniques, such as Western blotting and chromatin immunoprecipitation (ChIP), to study the epigenetic regulation of gene expression.

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Lab products found in correlation

Anti ash2l, by Fortis Life Sciences (2 mentions) Anti rbbp5, by Fortis Life Sciences (1 mentions) Anti ring1b, by Cell Signaling Technology (1 mentions) Anti flag m2, by Merck Group (1 mentions) Anti ezh2, by BD (1 mentions) Anti h2ak119ub, by Cell Signaling Technology (1 mentions)

2 protocols using anti h4k20me1

1

Epigenetic Markers Immunostaining Protocol

Cited 1 time
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Immuno-FISH was performed as described [28 (link),67 (link)]. Antibodies used in this study are the following: anti-H3K27me3 (#0323, MABI [1:1000 dilution] and #9733, Cell Signaling [1:1000]), anti-uH2A (#8240, Cell Signaling [1:1000]), anti-H4K20me1 (#39727, Active motif [1:5000]), anti-RBBP5 (A300-109A, Bethyl Laboratories [1:400]), anti-ASH2L (A300-107A, Bethyl Laboratories [1:400]), anti-EZH2 (#612666, BD [1:200]), anti-RING1B (#5694, Cell Signaling [1:50]) and anti-FLAG-M2 (F1804, SIGMA [1:4000]).
Yue M., Ogawa A., Yamada N., Charles Richard J.L., Barski A, & Ogawa Y. (2017). Xist RNA repeat E is essential for ASH2L recruitment to the inactive X and regulates histone modifications and escape gene expression. PLoS Genetics, 13(7), e1006890.
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2

Immuno-FISH Protocol for Histone Modifications

Cited 1 time
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Immuno-FISH was performed as described (21 (link),35 (link)). Antibodies used in this study are: anti-H3K27me3 (MABI #0323, 1:1000 dilution and Cell Signaling #9733, 1:1000), anti-H2AK119Ub (Cell Signaling #8240, 1:1000), anti-H4K20me1 (Active motif #39727, 1:5000), anti-ASH2L (Bethyl Laboratories A300-107A, 1:400).
Yue M, & Ogawa Y. (2017). CRISPR/Cas9-mediated modulation of splicing efficiency reveals short splicing isoform of Xist RNA is sufficient to induce X-chromosome inactivation. Nucleic Acids Research, 46(5), e26.
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