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Tmp195

Manufactured by Cayman Chemical
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TMP195 is a laboratory instrument used for the measurement and analysis of temperature. It is a high-precision digital thermometer designed to provide accurate temperature readings. The device features a user-friendly interface and is suitable for a variety of laboratory applications.

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Lab products found in correlation

Cycloheximide (chx), by Merck Group (1 mentions) Pronase, by Merck Group (1 mentions) Mocetinostat, by Cayman Chemical (1 mentions) Ms 275, by Santa Cruz Biotechnology (1 mentions) Rgfp966, by Cayman Chemical (1 mentions) Brd6688, by Cayman Chemical (1 mentions) Trichostatin a, by Cayman Chemical (1 mentions) Pci 34051, by Cayman Chemical (1 mentions)

2 protocols using tmp195

1

Modulation of HDAC and NLRP3 Pathways

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Pan-HDAC inhibitor Trichostatin A (TSA), class IIa HDAC inhibitors (HDAC4,5,7 and 9) TMP195, Class I & IV HDAC inhibitor (HDAC1,2,3 and 11) Mocetinostat, HDAC6 and 8 inhibitor BRD73954, HDAC3 inhibitor RGFP966 and a HDAC1 and 2 inhibitor BRD6688 were purchased from Cayman Chemical, HDAC1 and 3 inhibitor MS-275, and HDAC1 inhibitor 4-(dimethylamino)-N-[6-(hydroxyamino)-6-oxohexyl]-benzamide (DHOB) were purchased from Santa Cruz Biotechnology, NLRP3 specific inhibitor CP-456773/MCC 950, caspase-1 inhibitor caspase-1 Inhibitor II and protein synthesis inhibitor, cycloheximide, and pronase were purchased from Sigma-Aldrich. The stocks of all the chemical inhibitors were prepared in DMSO following the instructions of the vendors and stored at -80°C before use.
Moreira J.D., Iakhiaev A., Vankayalapati R., Jung B.G, & Samten B. (2022). Histone deacetylase-2 controls IL-1β production through the regulation of NLRP3 expression and activation in tuberculosis infection. iScience, 25(8), 104799.
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2

Histone Deacetylase Inhibition in Cell Treatment

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Three cycles of 48-h treatment with PCI34051 (HDAC8 inhibitor, Cayman chemical, cat. no 10444) or TMP 195 (HDAC4 and HDAC7 inhibitor, Cayman chemical, cat. no 23242) were performed. 24 h after seeding, the cells were treated with one first cycle of inhibition. Chemotherapy treatment was performed together with the second cycle of inhibition.
Pantelaiou-Prokaki G., Mieczkowska I., Schmidt G.E., Fritzsche S., Prokakis E., Gallwas J, & Wegwitz F. (2022). HDAC8 suppresses the epithelial phenotype and promotes EMT in chemotherapy-treated basal-like breast cancer. Clinical Epigenetics, 14, 7.
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