Lucifer yellow dilithium salt

Manufactured by Merck Group

Sourced in United States

Lucifer yellow dilithium salt is a fluorescent dye used as a labeling agent in various biological applications. It exhibits excitation and emission wavelengths suitable for fluorescence microscopy and flow cytometry. The compound is soluble in aqueous solutions and has a high quantum yield, making it a useful tool for researchers studying cellular processes and structures.

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Lab products found in correlation

Streptomycin, by Thermo Fisher Scientific (2 mentions) Adenosine triphosphate (atp), by Merck Group (2 mentions) Ethylene diamine tetraacetic acid (edta), by Merck Group (2 mentions) Bapta am, by Thermo Fisher Scientific (2 mentions) Yo pro 1, by Thermo Fisher Scientific (2 mentions) Yoyo 1, by Thermo Fisher Scientific (2 mentions) Phrodo red e coli bioparticles conjugate, by Thermo Fisher Scientific (2 mentions) Tannic acid, by Merck Group (2 mentions) Nigericin, by Merck Group (2 mentions) Bzatp, by Merck Group (2 mentions) 4 4 diisothiocyano 2 2 stilbenedisulfonic acid dids, by Merck Group (2 mentions) Bx430, by Bio-Techne (2 mentions) 10panx inhibitory peptide, by Bio-Techne (2 mentions) A804598, by Bio-Techne (2 mentions) Fluo 4 am, by Thermo Fisher Scientific (1 mentions) Ethidium bromide, by Merck Group (1 mentions) Probenecid, by Merck Group (1 mentions) A438079, by Bio-Techne (1 mentions) Rpmi 1640, by Thermo Fisher Scientific (1 mentions) Penicillin, by Thermo Fisher Scientific (1 mentions)

Close spelling variants of this product

Lucifer Yellow (172 citations) Lucifer Yellow CH dilithium salt (19 citations) Lucifer Yellow CH dilithium salt (LY, (2 citations)

2 protocols using lucifer yellow dilithium salt

Detailed Cytotoxicity Assays Protocol

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Dulbecco’s modified Eagle’s medium (DMEM), RPMI 1640, fetal bovine serum (FBS), penicillin, and streptomycin were purchased from Gibco by Life Technologies (Waltham, MA, USA). ATP, BzATP, EDTA, ethidium bromide, lipopolysaccharides from E. coli O55:B5 (LPS; L2880), dimethyl sulfoxide (DMSO), Lucifer yellow dilithium salt, 5(6)-carboxyfluorescein, carbenoxolone, probenecid, tannic acid, 4,4′-diisothiocyano-2,2′-stilbenedisulfonic acid (DIDS), and nigericin were purchased from Millipore-Sigma (St. Louis, MO, USA). BAPTA-AM, YO-PRO-1, YOYO-1, Fluo-4-AM, pHrodo Red E. coli BioParticles Conjugate, and pHrodo Green AM were purchased from Invitrogen/ThermoFisher (Carlsbad, CA, USA). A438079, A804598, BX430, and ¹⁰Panx inhibitory peptide were purchased from Tocris (Minneapolis, MN, USA). Complete growth differentiated media with serum (E37089-01-S), extracellular matrix-coated T25 or T75 culture flasks (E37089-01-T25,T75), and Xeno-free cell dissociation media (M37001-02CM) were obtained from Celprogen (Torrance, CA, USA).

Janks L., Sharma C.V, & Egan T.M. (2018). A central role for P2X7 receptors in human microglia. Journal of Neuroinflammation, 15, 325.

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Investigating Purinergic Signaling Mechanisms

Cited 1 time

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RPMI 1640, penicillin and streptomycin were purchased from Gibco by Life Technologies (Waltham, MA, USA). ATP, BzATP, EDTA, lipopolysaccharides from E. coli O55:B5 (LPS; L2880), dimethyl sulfoxide (DMSO), Na gluconate, K gluconate, Lucifer yellow dilithium salt, 5 (6 (link ))-carboxyfluorescein, cabenoxolone, tannic acid, 4,4’-Diisothiocyano-2,2’-stilbenedisulfonic acid (DIDS), CaCCinh-A01 (A01), 5-Nitro-2-(3-phenylpropylamino)benzoic acid (NPPB), nigericin, ionomycin, MCC950, and Melittin (MEL) were purchased from Millipore Sigma (St. Louis, MO, USA). MQAE, BAPTA-AM, YO-PRO-1, YOYO-1, pHrodo Red E. coli BioParticles Conjugate, and 35 mm Nunclon surface dishes were purchased from Invitrogen/ThermoFisher (Carlsbad, CA, USA). A804598, BX430, ¹⁰Panx inhibitory peptide, and Y-27632 were purchased from Tocris (Minneapolis, MN, USA). N-(p-amylcinnamoyl) Anthranilic Acid (ACA), Bromoenol lactone (BEL), SB-203580 (SB), PP2, IAA-94, and Ac-YVAD-CMK (YVAD) were purchased from Cayman Chemical (Ann Arbor, Michigan, USA).

Janks L., Sprague R.S, & Egan T.M. (2018). ATP-gated P2X7 receptors require chloride channels to promote inflammation in human macrophages. Journal of immunology (Baltimore, Md. : 1950), 202(3), 883-898.

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