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B6 ly5

Manufactured by Jackson ImmunoResearch
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B6 Ly5.1 is a mouse monoclonal antibody that recognizes the Ly-5.1 (CD45.1) allotype expressed on cells from certain mouse strains. It can be used to identify and distinguish cells expressing the Ly-5.1 allotype from those expressing the Ly-5.2 (CD45.2) allotype.

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Lab products found in correlation

C57bl 6, by Jackson ImmunoResearch (3 mentions) B6 rag1 mice, by Jackson ImmunoResearch (2 mentions) B6 il27ra mice, by Amgen (2 mentions) B6 rag1, by Jackson ImmunoResearch (1 mentions) C57bl 6 mice, by Jackson ImmunoResearch (1 mentions) Nsg mice, by Jackson ImmunoResearch (1 mentions)

Spelling variants of this product

Ly5.1 (B6.SJL-Ptprca Pepcb/BoyJ), (3 citations) Ly5.1 congenic B6 mice (2 citations) B6.Ly5.2 mice (2 citations) B6.Ly5.1 (H2b Ly5.1+) (2 citations) B6.SJL-Ptprca Pepcb/BoyJ (CD45.1/Ly5.1) (2 citations) B6-Ptprca Pepcb/BoyJ (Ly5.1) (2 citations)

5 protocols using b6 ly5

1

Mouse Strains for Immunology Research

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C57BL/6, B6 Ly5.1, B6 Thy1.1, B6 Rag1−/− mice were purchased from the Jackson Laboratory (Bar Harbor, ME). B6 Il27ra−/− mice were obtained from Amgen (Thousand Oaks, CA). B6 Foxp3GFP 41 (link) and B6 Lag3−/− mice were obtained from Drs. Yasmine Belkaid and Christophe Benoist, respectively. All animal procedures were conducted according to the guidelines of the Institutional Animal Care and Use Committee.
Do J.S., Visperas A., Sanogo Y.O., Bechtel J.J., Dvorina N., Kim S., Jang E., Stohlman S.A., Shen B., Fairchild R.L., Baldwin WM I.I.I., Vignali D.A, & Min B. (2015). An IL-27/Lag3 axis enhances Foxp3+ regulatory T cell suppressive function and therapeutic efficacy. Mucosal immunology, 9(1), 137-145.
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2

Mouse Strains for Immunology Research

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C57BL/6, B6 Ly5.1, B6 Thy1.1, B6 Rag1−/− mice were purchased from the Jackson Laboratory (Bar Harbor, ME). B6 Il27ra−/− mice were obtained from Amgen (Thousand Oaks, CA). B6 Foxp3GFP 41 (link) and B6 Lag3−/− mice were obtained from Drs. Yasmine Belkaid and Christophe Benoist, respectively. All animal procedures were conducted according to the guidelines of the Institutional Animal Care and Use Committee.
Do J.S., Visperas A., Sanogo Y.O., Bechtel J.J., Dvorina N., Kim S., Jang E., Stohlman S.A., Shen B., Fairchild R.L., Baldwin WM I.I.I., Vignali D.A, & Min B. (2015). An IL-27/Lag3 axis enhances Foxp3+ regulatory T cell suppressive function and therapeutic efficacy. Mucosal immunology, 9(1), 137-145.
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3

Mouse Strains for Immunological Studies

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C57BL/6, B6 Ly5.1, B6 Thy1.1, B6 IFNγR−/−, B6 Rag1−/− mice were purchased from the Jackson Laboratory (Bar Harbor, ME). IFNγR−/− Rag−/− mice were bred at the animal facility of the Lerner Research Institute. All animal procedures were conducted according to the guidelines of the Institutional Animal Care and Use Committee.
Do J.S., Asosingh K., Baldwin WM I.I.I, & Min B. (2014). IFNγR signaling in non-T cell targets regulates T cell-mediated intestinal inflammation through multiple mechanisms. Journal of immunology (Baltimore, Md. : 1950), 192(6), 2537-2541.
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4

Diverse Mouse Models Comparison

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C57BL/6 mice, B6.Ly5.1, B6.129P2(C)-Ccr7tm1Rfor/J (CCR7-deficient), and NSG mice were from Jackson Laboratories (Bar Harbor, ME) 1 . B6.CARMA1-deficient mice were gift from Dr. Marisa Alegre (University of Chicago). All mice were bred and/or maintained in a specific pathogen-free conditions (University of New Mexico) and conform to the principles outlined by the Animal Welfare Act and NIH guidelines, IACUC protocol 15-101294. All efforts were made to minimize suffering.
Oruganti S.R., Torres D.J., Krebsbach S., Asperti-Boursin F., Winters J., Matlawska-Wasowska K., Winter S.S., Halsey C, & Cannon J.L. (2016). CARMA1 is a novel regulator of T-ALL disease and leukemic cell migration to the CNS. Leukemia, 31(1), 255-258.
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5

Mouse Breeding and Care Protocol

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Breeding stock of B6Ly5.2 (H2b, Ly5.2) and B6Ly5.1 (H2b, Ly5.1, Jackson Laboratory, Bar Harbor, ME) and Balb/c (H2d, Ly5.2, Charles River Laboratories, Wilmington, MA) mice were bred in our colony in the Cincinnati Children’s Research Foundation. All experimental protocols were approved by the Institutional Animal Care and Use Committee and in compliance with the U.S. Department of Health Guide for the Care and Use of Laboratory Animals.
Alhajjat A.M., Strong B.S., Lee A.E., Turner L.E., Wadhwani R.K., Ortaldo J.R., Heusel J.W, & Shaaban A.F. (2015). Prenatal allospecific NK cell tolerance hinges on instructive allorecognition through the activating receptor during development. Journal of immunology (Baltimore, Md. : 1950), 195(4), 1506-1516.
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