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Pe anti human cd95 mab

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The PE anti-human CD95 monoclonal antibody is a fluorescently labeled immune cell marker used for the identification and analysis of CD95-positive cells in flow cytometry applications.

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Lab products found in correlation

Pe anti human dr5 ab, by Thermo Fisher Scientific (2 mentions) Facscalibur flow cytometer, by BD (1 mentions) Facscalibur, by BD (1 mentions)

Spelling variants of this product

CD95-PE (14 citations) Anti-CD95 (10 citations) Anti-CD95-PE (8 citations) Anti-human mAbs (2 citations)

2 protocols using pe anti human cd95 mab

1

Surface Receptor Expression in Glioblastoma

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Surface levels of TRAIL-R2/DR5 (PE anti-human DR5 Ab, eBioscience #12-9908), TRAIL-R1/DR4 (PE anti-human DR4, eBioscience #12-6644); FAS/CD95 (PE anti-human CD95 mAb, BD Pharmingen #555674) , TNF-R1 (PE anti-human TNFR1 mAb, R&D #FAB225P) and TNF-R2 (PE Rat anti-human CD120b #552418, BD Pharmingen) on human glioblastoma cell lines were determined by staining with the corresponding Abs and subsequent flow cytometry. A FACS Calibur flow cytometer (Becton Dickinson, Mountain View, CA, USA) and the CellQuest program were used to perform flow cytometric analysis. All experiments were independently repeated 3–5 times.
Ivanov V.N., Wu J, & Hei T.K. (2017). Regulation of human glioblastoma cell death by combined treatment of cannabidiol, γ-radiation and small molecule inhibitors of cell signaling pathways. Oncotarget, 8(43), 74068-74095.
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2

Flow Cytometric Analysis of Death Receptors

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Surface levels of TRAIL-R2/DR5 (PE anti-human DR5 Ab, eBioscience #12-9908), FAS/CD95 (PE anti-human CD95 mAb, BD Pharmingen #555674), PD-L1 (CD274) (PE anti-human mAb eBioscience #12-5983) on human GBM cell lines were determined by staining with the corresponding Abs and subsequent flow cytometry. A FACS Calibur flow cytometer (Becton Dickinson, Mountain View, CA, USA) and the CellQuest program were used to perform flow cytometric analysis. All experiments were independently repeated 3-5 times.
Ivanov V.N., Wu J., Wang T.J, & Hei T.K. (2019). Inhibition of ATM kinase upregulates levels of cell death induced by cannabidiol and γ-irradiation in human glioblastoma cells. Oncotarget, 10(8), 825-846.
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