All subjects were intravenously injected with 18F-FDG (dose range 185–295 megabecquerels) and hydrated with 500 mL of saline (0.9 % sodium chloride). PET/CT acquisition, using a General Electric VCT PET/CT scanner, started 30±5 min after 18F-FDG injection and lasted 10 min for all participants. 18F-FDG-PET studies were performed within one week of the PSG recording. The reconstruction parameters were as follows: ordered subset expectation maximization, 4 subsets and 14 iterations; matrix 256×256; full width at half maximum (FWHM): 5 mm [28 (link)].
Vct pet ct scanner
Manufactured by GE Healthcare
Sourced in United States
The VCT PET/CT scanner is a medical imaging device that combines positron emission tomography (PET) and computed tomography (CT) technology. It is designed to acquire high-quality images for diagnostic and treatment planning purposes.
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4 protocols using vct pet ct scanner
1
PET/CT Imaging of Metabolic Activity
2
PET/CT Imaging Protocol for 18F-FDG Quantification
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The PET/CT protocol study was conducted at the Nuclear Medicine facility of the University Hospital of Rome “Tor Vergata”. All subjects, the day before performing PSG, were intravenously injected with 18F-FDG (dose range 185–295 megabequerels) and hydrated with 500 mL of saline (0.9% sodium chloride). PET/CT acquisition, using a General Electric VCT PET/CT scanner, started 30 ± 5 min after 18F-FDG injection and lasted 10 min for all participants. The reconstruction parameters were as follows: ordered subset expectation maximisation, 4 subsets and 14 iterations; matrix 256 × 256; full width at half maximum (FWHM): 5 mm [52 (link)].
3
PET/CT Imaging for Alzheimer's Diagnosis
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The study was conducted at the Nuclear Medicine Unit of Policlinico Tor Vergata in Rome (General Electric VCT PET/CT scanner). Among the enrolled patients, 46 had undergone FDG-PET at our site (CSFAβ42 + /amyR − , n = 9; CSFAβ42 + /amyR + , n = 17; A + T + , n = 20).
Moreover, 24 subjects (male, 10; female, 14; mean age, 66.56 ± 10.44 years) undergoing FDG-PET/CT for other reasons were enrolled as part of a control group (CG) upon showing no signs of hypometabolism suggestive of neurodegenerative disorders nor other cerebral abnormalities, as assessed by visual reads (A.C). All subjects had an MRI performed within 14 ± 4 days before PET/CT examination, showing absence of brain alterations. An experienced neurologist (A.M.) evaluated all participants to assess the absence of clinical signs of cognitive decline. Patient and control selection strategies for the sub-study are summarized in Additional file2 .
Moreover, 24 subjects (male, 10; female, 14; mean age, 66.56 ± 10.44 years) undergoing FDG-PET/CT for other reasons were enrolled as part of a control group (CG) upon showing no signs of hypometabolism suggestive of neurodegenerative disorders nor other cerebral abnormalities, as assessed by visual reads (A.C). All subjects had an MRI performed within 14 ± 4 days before PET/CT examination, showing absence of brain alterations. An experienced neurologist (A.M.) evaluated all participants to assess the absence of clinical signs of cognitive decline. Patient and control selection strategies for the sub-study are summarized in Additional file
4
PET/CT Imaging Protocol for Alzheimer's Disease
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All acquisitions were performed at the Nuclear Medicine Unit of the University Hospital “Policlinico Tor Vergata” in Rome with a General Electric VCT PET/CT scanner (GE Medical Systems, Tennessee, USA). All subjects fasted for at least 5 h before i.v. injection of FDG, and serum glucose levels were in range according to European Association of Nuclear Medicine guidelines [36 (link)]. Patients were injected intravenously with 18F-FDG (dose range 185–295 MBq) and then hydrated with 500 mL of saline (0.9% sodium chloride), according to a previous similar report of our group in this field [37 (link)]. The scan started 30 min after the injection and lasted ten minutes. Acquisition and reconstruction parameters are reported elsewhere and were followed according to the report cited above. [37 (link)].
Upon imaging evaluation, all 27 subjects showed typical findings of brain cortical hypometabolism in key regions compatible with a diagnosis of classical AD (parietal and posterior cingulate cortices, precuneus, or a combination of the above) [38 (link)].
Upon imaging evaluation, all 27 subjects showed typical findings of brain cortical hypometabolism in key regions compatible with a diagnosis of classical AD (parietal and posterior cingulate cortices, precuneus, or a combination of the above) [38 (link)].
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