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N6 cyclopentyladenosine

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N6-cyclopentyladenosine is a chemical compound used as a laboratory reagent. It functions as a selective agonist for the adenosine A1 receptor.

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Lab products found in correlation

Adenosine, by Merck Group (1 mentions) Mrs1754, by Bio-Techne (1 mentions) Cgs21680, by Bio-Techne (1 mentions) 2 chloroadenosine, by Merck Group (1 mentions) Sch442416, by Bio-Techne (1 mentions) 5 iodotubercidin, by Bio-Techne (1 mentions) 8 cyclopentyl 1 3 dipropylxanthine, by Bio-Techne (1 mentions) N methyl β d ribofuranuronamide, by Bio-Techne (1 mentions) Erythro 9 2 hydroxy 3 nonyl adenine ehna, by Merck Group (1 mentions) Enkephalin, by Bio-Techne (1 mentions) Picrotoxin ptx, by Merck Group (1 mentions) Rs baclofen, by Bio-Techne (1 mentions) Histamine dihydrochloride, by Bio-Techne (1 mentions) Win55 212, by Bio-Techne (1 mentions) Ng nitro l arginine methyl ester l name, by Merck Group (1 mentions) 1h 1 2 4 oxadiazole 4 3 a quinoxalin 1 one, by Merck Group (1 mentions) Adenosine deaminase ada, by Roche (1 mentions)

3 protocols using n6 cyclopentyladenosine

1

Adenosine Receptor Pharmacology Protocols

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Adenosine, 2-chloroAdenosine (stable Adenosine analogue), and erythro-9-(2-hydroxy-3-nonyl) adenine (EHNA; increases endogenous Adenosine by inhibiting Adenosine deaminase and thus reducing the metabolism of Adenosine to inosine) were purchased from Sigma-Aldrich (St. Louis, MO). N6-cyclopentylAdenosine (CPA; selective A1 receptor agonist), CGS21680 (selective A2A receptor agonist), 8-cyclopentyl-1,3-dipropylxanthine (DPCPX; selective A1 receptor antagonist), 5-iodotubercidin (IDO; increases endogenous Adenosine by inhibiting Adenosine kinase and thus reducing the metabolism of Adenosine to 5’-AMP), 5’-N-ethylcarboxamidoAdenosine (NECA; non-selective Adenosine receptor agonist), 5’-N-methylcarboxamidoAdenosine (MECA; non-selective Adenosine receptor agonist), 1-deoxy-1-[6-[[(3-iodophenyl)methyl]amino]-9H-purin-9-yl]-N-methyl-β-D-ribofuranuronamide (IB-MECA; selective A3 receptor agonist), SCH442416 (selective A2A receptor antagonist), MRS1754 (selective A2B receptor antagonist), VUF5574 (selective A3 receptor antagonist) were purchased from Tocris (Minneapolis, MN). 3H-thymidine (specific activity, 11.8 Ci/mmol) was purchased from PerkinElmer NEN (Walthan, MA). All other reagents were of tissue culture or best grade available.
Dubey R.K., Fingerle J., Gillespie D.G., Mi Z., Rosselli M., Imthurn B, & Jackson E.K. (2015). Adenosine Attenuates Human Coronary Artery Smooth Muscle Cell Proliferation By Inhibiting Multiple Signaling Pathways that Converge on Cyclin D. Hypertension, 66(6), 1207-1219.
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2

Pharmacological Modulation of Neuroreceptors

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(RS)-Baclofen (BAC), sumatriptan succinate, [D-Ala2, NMe-Phe4, Gly-ol5]-enkephalin (DAMGO), (-)–U50488, WIN 55-212, (1R, 4R, 5S, 6R)-4-Amino-2-oxabicyclo [3.1.0] hexane-4,6-dicarboxylic acid (LY379268), histamine dihydrochloride (HA), N6-Cyclopentyladenosine (CPA), all purchased from Tocris Bioscience. Picrotoxin (PTX) purchased from Sigma Aldrich. Benzyloxy-Cyclopentyladenosine (BnOCPA) was provided by the Hamm Lab.
Manz K.M., Zepeda J.C., Zurawski Z., Hamm H.E, & Grueter B.A. (2023). SNAP25 differentially contributes to Gi/o-coupled receptor function at glutamatergic synapses in the nucleus accumbens. Frontiers in Cellular Neuroscience, 17, 1165261.
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3

Adenosine and PKG Signaling Modulation

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N6-cyclopentyladenosine (CPA) was purchased from Tocris, (9S,10R,12R)-2,3,9,10,11,12-Hexahydro-10-methoxy-2,9-dimet-hyl-1-oxo-9,12-epoxy-1H-diindolo[1,2,3-fg:3′,2′,1′-kl]pyrrolo[3,4-i][1,6]benzodiazocine-10-carboxylic acid methylester (KT5823) was from Santa Cruz Biotecnology, 1H-[1,2,4]oxadiazole[4,3-a]quinoxalin-1-one (ODQ) and NG-nitro-L-arginine methylester (L-NAME) were from Sigma and Adenosine deaminase (ADA) was from Roche. All other reagents used were from analytical grade.
Pinto I., Serpa A., Sebastião A.M, & Cascalheira J.F. (2016). The Role of cGMP on Adenosine A1 Receptor-mediated Inhibition of Synaptic Transmission at the Hippocampus. Frontiers in Pharmacology, 7, 103.
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