Xe 1200

At baseline, fasting whole blood samples were obtained from a vein in the antecubital fossa and were collected in precooled ethylenediaminetetraacetic acid tubes from each patient. The ABO blood groups were determined by standard procedures using agglutination techniques as we previously indicated.^{8 (link)} The concentrations of low-density lipoprotein cholesterol (LDL-C) and high-density lipoprotein cholesterol (HDL-C) were analyzed by selective solubilization method (LDL-C or HDL-C kit, Kyowa Medex, Tokyo). All of the lipid profiles were determined by automatic biochemistry analyzer (Hitachi 7150, Tokyo, Japan). The high sensitivity-C reactive protein (hs-CRP) concentrations were determined by immunoturbidimetry (Beckmann Assay, Bera, CA). The erythrocyte sedimentation rate (ESR) was analyzed by Westergren method. The fibrinogen levels were measured by the Stago auto analyzer using Clauss method (Diagnostic Stago, Taverny, France). White blood cell counts were determined by the automated hematology analyzer (Sysmex XE-1200, Kobe, Japan).

Mesenteric ischemia was diagnosed by a detailed physical examination, laboratory tests, and abdominal ultrasonography. Ultrasonography is valuable for proximal arterial flow assessment; however, the evaluation of distal arterial segment flow is sometimes complicated. Furthermore, in obese patients, in situations with excessive intra-abdominal gas and densely calcified arteries, ultrasonography is not convenient. All patients underwent computerized tomography angiography to confirm diagnosis. Patients were followed up at general surgery ward. All patients left ventricle ejection, and heart valves were evaluated with echocardiography (Vivid 9, GE VingMedUltrasound, Horten, Norway). Plasma total protein (TP) levels and HCT values at the beginning of hospitalization with the diagnosis of acute mesenteric ischemia were obtained from the hospital virtual laboratory data archive and recorded in the data form. The erythrocyte count, hemoglobin, HCT, and white blood cell count were measured using the automated hematology analyzer XE-1200 (Sysmex, Kobe, Japan). Biochemical measurements were performed with a molecular analyzer (Roche Diagnostics, Manheim, Germany).

Peripheral blood samples were collected via the antecubital vein after a 12-hour overnight fast in appropriate tubes. Dry tubes for biochemical tests and tubes with ethylenediaminetetraacetic acid (EDTA) for the hematological test were used. Erythrocyte count, hemoglobin, hematocrit, and white blood cell (WBC) count were measured using an automated hematology analyzer XE-1200 (Sysmex, Kobe, Japan). The biochemical measurements were determined by using a molecular analyzer (Roche Diagnostics, Manheim, Germany).

Peripheral venous blood samples were obtained from a large antecubital vein and collected at baseline in dry tubes for biochemical tests and pre-cooled EDTA tubes for the hematological test. Complete blood counts were measured using an automated hematology analyzer XE-1200 (Sysmex, Kobe, Japan). Baseline SII level was calculated using the following equation: