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P jnk thr183 tyr185 rabbit mab

Manufactured by Cell Signaling Technology
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P-JNK (Thr183/Tyr185) rabbit mAb is a laboratory antibody product that specifically recognizes the phosphorylated form of c-Jun N-terminal kinase (JNK) at threonine 183 and tyrosine 185. This antibody can be used to detect and quantify the levels of activated JNK in various biological samples.

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Lab products found in correlation

Trizol, by Thermo Fisher Scientific (2 mentions) Gapdh mouse mab, by Cell Signaling Technology (1 mentions) Sp600125, by Cell Signaling Technology (1 mentions) C jun rabbit mab, by Cell Signaling Technology (1 mentions) Icrt14, by MedChemExpress (1 mentions) β actin rabbit mab, by Cell Signaling Technology (1 mentions) Mouse control igg, by ABclonal (1 mentions) Alexa fluor 488 conjugated goat anti rabbit igg h l, by Thermo Fisher Scientific (1 mentions) Lamina c mouse mab, by Santa Cruz Biotechnology (1 mentions) β tubulin rabbit pab, by ABclonal (1 mentions) Rabbit control igg, by ABclonal (1 mentions) Goat anti rabbit igg, by Cell Signaling Technology (1 mentions) Caspase 3, by Cell Signaling Technology (1 mentions) P perk, by Cell Signaling Technology (1 mentions) Antibodies against cytochrome c, by Abcam (1 mentions) Antibodies against bip, by Cell Signaling Technology (1 mentions) P ire1, by Cell Signaling Technology (1 mentions) P erk, by Cell Signaling Technology (1 mentions) Gapdh, by Abcam (1 mentions) In situ cell death detection kit, by Roche (1 mentions)

2 protocols using p jnk thr183 tyr185 rabbit mab

1

Investigating JNK/c-Jun Signaling Pathway

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The following chemical reagents were used in this study: iCRT14 (MedChemExpress, cat# HY16665), sp600125 (Cell Signaling Technology, cat#8177). The following antibodies were used in this study: phospho(p)-c-Jun (Ser73) rabbit monoclonal antibody (Cell Signaling Technology, cat# 3270), c-Jun rabbit mAb (Cell Signaling Technology, cat# 9165), p-JNK (Thr183/Tyr185) rabbit mAb (Cell Signaling Technology, cat# 9251), JNK rabbit polyclonal antibody (pAb) (Cell Signaling Technology, cat# 9252). p-β-catenin (Ser552) rabbit mAb (Cell Signaling Technology, cat# 9566). β-catenin (Ser552) rabbit mAb (Abcam, cat# ab32572), mouse control IgG (ABclonal, cat# AC011), rabbit control IgG (ABclonal, cat# AC005), laminA/C mouse mAb (Santa Cruz Biotechnology, cat# sc-376248), β-Tubulin rabbit pAb (Abclonal, cat# AC015), GAPDH mouse mAb (Cell Signaling Technology, cat# 2118), β-actin rabbit mAb (Cell Signaling Technology, cat# 4970), Alexa Fluor 488®-conjugated goat anti-rabbit IgG (H + L) (Invitrogen, cat# A-11008), HRP- (horseradish peroxidase-) conjugated goat anti-mouse IgG (Cell Signaling Technology, cat# 7076), and goat anti-rabbit IgG (Cell Signaling Technology, cat# 7074). Goat anti-BoHV-1 serum was purchased from VMDR Inc (cat# 20PAB-IBR).
Chang L., Yuan W, & Zhu L. (2020). β-cantenin is potentially involved in the regulation of c-Jun signaling following bovine herpesvirus 1 infection. Veterinary Microbiology, 248, 108804.
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2

Molecular Mechanisms of ER Stress Response

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Antibodies against Bip, p-PERK, PERK, p-IRE1, IRE1, ATF6, Chop and Caspase-3 were obtained from Cell Signaling Technology, Inc. Antibodies against cytochrome c, cytochrome c oxidase IV (COX IV) and GAPDH were purchased from Abcam. Phosphorylated (p)-Bcl-2 (Ser70) rabbit mAb, p-JNK (Thr183/Tyr185) rabbit mAb and JNK rabbit mAb were obtained from Cell Signaling Technology, Inc. LC3 rabbit mAb was purchased from Santa Cruz Biotechnology, Inc. The MDA Assay kit (TBA method) and Hydrogen Peroxide Assay kit were obtained from Nanjing Jiancheng Bioengineering Institute. An In Situ Cell Death Detection kit was obtained from Roche Applied Science. Dexamethasone (DEX) was purchased from MilliporeSigma. Tat-scramble (LPS VFG DVG APS RLP EVS LSP PRR RQR RKK RG-NH2) and Tat-SabKIM1 (GFE SLS VPS PLD LSG PRV VAP PRR RQR RKK RG-NH2) were purchased from NeoPeptide. TRIzol was purchased from Thermo Fisher Scientific, Inc.
Li C., Ma D., Chen Y., Liu W., Jin F, & Bo L. (2022). Selective inhibition of JNK located on mitochondria protects against mitochondrial dysfunction and cell death caused by endoplasmic reticulum stress in mice with LPS-induced ALI/ARDS. International Journal of Molecular Medicine, 49(6), 85.
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