The holographic haptic display (UHEV1, Ultrahaptics Ltd.) comprised a planar array of N ultrasound emitters. Such a display focuses sound in air by applying a phase delay to each transducer signal. Constructive interference yields localized regions of high differential pressure, sufficient to impart time-varying displacements to the skin (1 (link), 2 ). The differential acoustic pressure field may be described by p(x,t)=n1N14πrnanexp[j(krnωtϕn)] where x is a position within the Fresnel zone of the array, an is the amplitude of the acoustic pressure wave emitted from transducer n, k = ω/c is the wave number, f = ω/(2π) = 40 kHz is the ultrasound carrier frequency, c is the speed of sound, rn = ‖xxn‖ is the distance from x to the location xn of transducer n, and ϕn is the corresponding phase delay. Focusing at a location xf is achieved by matching the phase delays, ϕn, to the propagation time for a wavefront to reach the focus ϕn = ω‖xfxn‖/c, thus yielding constructive interference. The focal width, δ, satisfies a Rayleigh diffraction limit, with δ ⪆ c/(2f).
Ultrasound frequency oscillations cannot be directly felt via touch. In haptic holography, palpable low-frequency mechanical signals are produced via a nonlinear phenomenon known as acoustic radiation pressure. Neglecting viscosity, the Langevin acoustic radiation force FL imparted to an object (here, the skin) at a focus location is, to second order, given by FL=SdS(p2I+ρ0u1u1)n where p, ρ, and u are the fluid pressure, density, and velocity fields. The angular brackets denote time-averaged quantities, n is the surface normal, I is the unit tensor, and S is a surface region containing the focus location. Subscripts 0, 1, and 2 refer to successive terms in a perturbation expansion about a quiescent fluid configuration (3336 ). Applying the same expansion to the Navier-Stokes equation yields an expression for 〈p2〉 in terms of lower-order quantities p2=121ρ0c02p1212ρ0u12
Averaged over 1 cycle of oscillation, only the quadratic terms are nonzero. Together, they yield a nonvanishing, low-frequency force FL(x, t) that elicits vibrations in the skin (see Supplementary Text). The time evolution of these vibrations is governed by a driven elastic wave equation FL(x,t)={m2t2+μ2+[(K+μ/3)]}ξ(x,t)
Here, ξ(x, t) is tissue displacement, and m, μ, and K are the mass density, shear modulus, and bulk modulus, respectively. Plane wave solutions, ξ(x,t)=Ar^ej(kxωt) , describe oscillations along polarization directions r^ . Because of the low-frequency content of the acoustic radiation force and high propagation speed of compression waves, most acoustic energy is transferred to shear (transverse) wave components, k^r^=0 . For skin or soft tissues, shear wave speeds, c=μ/m , are frequency dependent and can range from 1 to 10 m/s at the tactile frequencies applicable to this study (22 (link), 23 ). Because of viscoelasticity, wave amplitudes are also attenuated in a frequency-dependent manner with increasing propagation distance (30 (link)).